The characteristic of an unsteady answer of interferon therapy are.
Disappearance of markers of viral replication upon completion of course of therapy
Normalization of activity of ALaT during the course of therapy
An origin of relapse in next 6 months
Disappearance of icterus
*All the above
That characteristic of a partial answer of interferon therapy are all, except.
*Disappearance of markers of viral replication
Normalization of activity of ALaT is upon completion of course of therapy
Disappearance of icterus
Normalization the state of patient
Normalization of the size of liver
When is interferon therapy effective in the the patient.
Normalization of the state of patient
Normalization of activity of ALaT upon completion of course of therapy
Disappearance of icterus
Normalization of the size of liver
*The markers of viral replication, are determined upon completion of course of therapy
What laboratory work-up is needed for confirming the diagnosis of viral hepatitis.
Total analysis of blood
Determination of level of bilirubin
Determination of activity of aminotransferase
*Determination of markers of HV in IFA
All the above
What laboratory and instrumental examinations are needed for confirming the diagnosis of viral hepatitis.
Complete analysis of blood
Ultrasound of abdominal region
Determination of activity of aminotransferase
*Determination of antigen of viruses
Duodenal probing
What is incubation period for hepatitis B:
45 days
*180 days
360 days
90 days
25 days
All the hepatitis have parenteral route of transmission except:
*A
B
C
D
TTV
Chronic course is common for viral hepatitis except:
*A
B
C
D
B+C
All the following medicines are interferons except:
Intron
Roferon
Reaferon
Leukinferon
*Cycloferon
All the following medicines are hepatoprotective agents except:
Carsil
Silibor
Legalon
*Lomusol
Arginine
Phage symptom in case of yellow fever is:
Pain in right iliac area
Enanthema on a soft palate
*Replacement of tachicardia on expressed bradicardia
Hemorrhages in a conjunctiva
Yellow hands
Hemograme in the second period of yellow fever:
Leukocytosis
Normal global analysis of blood
*Leukopenia, neutropenia
Leukopenia, neutrophilosis
Leukocytosis, lymphomonocytosis
Whatever complication meets at the yellow fever:
*Liver insufficiency
Kidney insufficiency
Infectious-toxic shock
Myocarditis
Edema of lungs
Unlike leptospirosis in case of yellow fever is absent:
Hemorrhagic syndrome
Kidney insufficiency
Іntoxication syndrome
Міalglic syndrome
*Hepatic insufficiency
For confirmation of yellow fever diagnosis use:
Bacteriological analysis of blood
Bacteriological examination of urine
*Virological hemanalysis
Biochemical blood test
Global analysis of blood
In the initial period of hemorrhagic fever with a kidney syndrome a characteristic sign is:
High temperatures
Pains in gastrocnemius muscles and positive Pasternatsky symptom
*Pains in joints and positive Pasternatsky symptom
Hemorragic syndrome
Dyspepsia phenomena
An initial period at the hemorrhagic fever with a kidneys syndrome lasts:
Few hours
One day
*Three days
one week
Two weeks
When a violations of diuresis at patients with hemorrhagic fever with a kidneys syndrome appear:
In initial period
Don’t appear
During all periods of disease
*In climax period
In recovering period
General view of patient with the hemorrhagic fever with a kidneys syndrome:
Skinning covers
*Pallor of nasolabial triangle, hyperemia of neck and overhead half of trunk
Hyperemia of person, scleritis, conjunctivitis
Grayish color of person
Icteric color of skin
In a biochemical blood test of patients with the hemorrhagic fever with a kidneys syndrome not typically:
High level of urea
Decline of potassium level
*Bilirubinemia
Increasing of kreatinine
Increasing of nitrogen
For confirmation of diagnosis of hemorragic fever with a kidney syndrome use:
Bacteriological method
Virological method
*Reaction of immunofluorescence
Reaction of braking of hemagglutination
Research of blood drop under a microscope
For treatment of patients with the hemorrhagic fever with a kidney syndrome does not use:
Glucocorticoids
Anabolic steroid
Disintoxication facilities
*Dihydration facilities
Antihistaminics
For the initial period of the Congo hemorrhagic fever not characteristically:
Fever
Pains in joints and muscles
Severe pain of head
*Oliguria
Dizziness
At an objective review for the Congo hemorrhagic fever characteristically:
*Mucosal hyperemia of person
Pallor of person
Puffiness of person
Ochrodermia of person
Exanthema on face
The most characteristic symptom in the climax period of the Congo hemorrhagic fever is:
*Hemorrhagic syndrome
Hepatic insufficiency
Dyspepsia phenomena
Sharp kidney insufficiency
Мeningeal syndrome
In a case of Congo fever in a general blood analysis is not typical:
. Leukocytosis
*Leukopenia
Neutropenia
Thrombocytopenia
Increasing of ESR
What laboratory and instrumental examinations should used for flu diagnosis?
Complete analysis of blood
X-ray of organs of thoraxic cavity
Analysis sputum
*Determination of viruses by the method of immunofluorescence
Biochemical blood test
Virus causing hemorrhagic cystitis, diarrhea and conjunctivitis:
RSV
Rhinovirus
*Adenovirus
Rotavirus
Flu
What is conduct specific passive immunnoprophylaxis of flu?
Live antenuated vaccine
Inactive parenteral vaccine
*Immune protein
Remantadin
Antibiotics of wide spectrum of action|
Duration of isolation of patient with influenza complications?
4 days
7 days
*10 days
17 days
20 days
How is the urgent prophylaxis of scarlet fever conducted?
Vaccination
*Isolation of children, who had contact with a patient
Chemioprophylaxis
Disinfection
Non-admission of contact with carrier of B-streptococcus
What level is necessary to reduce the temperature of patient’s body with hyperthermia?
39 °C
*38 °C
37,5 °C
37 °C
38,5 °C
What made specific passive flu immunization?
Living intranasal vaccine
Parenteral inactivated vaccine
* Immune globulin
Remantadin
Antibiotic
What pathogen causes severe acute respiratory syndrome?
Bocavirus
Rheovirus
Metapneumovirus
Adenovirus
*Coronavirus
Which family owned influenza?
Pallidum
Legionella
Tohovirus
* Ortomixovirus
Rickettsiae
The intensity of intoxication caused by flu depends on?
Hemagglutinin
Neuraminidase
* Rybonucleoproteid
Membrane proteins
S-protein
What determs the immunosuppressive action of influenza virus?
*Hemagglutinin
S-protein
Neuraminidase
RNA polymerase
The membrane protein
What is antigenic shift of influenza virus?
*Antigenic changes of rybonucleoproteid
Recombination hemagglutinin and neuraminidase
Antigenic changes of the virus within serovars
Genetic recombination between different strains of influenza virus
Variability neuraminidase
What medium used for culturing influenza virus?
*Chicken embryos and cell culture
Gall broth
Meat pepton agar
Medium containing blood
Water-serum culture medium
Which of these protein of influenza virus is capable to hemolise red blood cells?
RNA polymerase
S-protein
Neuraminidase
Membrane protein
* Hemagglutinin
Specify contagious period in flu?
The end of the incubation period + the entire period of the disease
End of the incubation period
Only during the height
* The period of convalescence
The crisis period + 7 days normal body temperature
What is a gateway for the flu virus?
*Cylindrical mucosal epithelium of respiratory tract
Solitary follicles
Mucous of tonsils
Epithelial cells of the skin
Mucous membranes of the digestive tract
What is antigenic drift of influenza virus?
*Partial change in antigenic specificity of hemagglutinin
Recombination hemagglutinin and neuraminidase
Antigenic changes of the virus within serovars
Genetic recombination between different strains of influenza virus
Variability neuraminidase
What is the main preventive measure in case of contact with sick with the flu?
Vaccination
* Chemoprophylaxis
Vitaminoprophylaxis
Admission antipyretics
Admission immunostimulators
Which of these vaccines is the least reactivity?
Fullvirion
Split vaccines
* Subunit
Live
Inactivated
What is the most common form of adenovirus infection?
Tracheobronchitis
Laryngitis
Pneumonia
* Pharyngoconjunctive fever
Rhinitis
What is most often clinically manifested flu?
Nasopharyngitis
Bronchospasm
Rhinitis
Acute respiratory failure
* Laryngotracheitis
What is most evident parainfluenza infection?
Rhinitis
* Laryngitis
Tracheobronchitis
Lymphadenopathy
Bronchospasm
Which agents of acute respiratory infections contain DNA?
Respiratory virus syntytsialnyy
Influenza virus
Parainfluenza virus
* Adenovirus
Rhinovirus
Which of acute respiratory diseases transmitted not only through airborne, but the fecal-oral transmission mechanism?
Rhinoviral disease
Parainfluenza
* Adenoviral disease
Respiratory syntytsial disease
Flu
What is the duration of incubation period of adenoviral infection?
Several hours - 1 day
1-2 days
* 1-14 days
2-3 days
14-21 day
What is characteristic of adenoviral conjunctivitis unlike diphtheria eyes?
Conjunctiva of eyelids bright red tape snug, hard shot
Complete loss of vision due panoftalmit
* Conjunctiva of eyelids bright red, covered with films that are easily removed
Photophobia, pain during palpation
Conjunctiva with haemorrhages
Which of the following is characteristic of adenovirus infection?
Bronchiolitis
Bronchiolitis and pneumonia
Real croup, pneumonia
* Membranous conjunctivitis and pharyngitis
Bronchiolitiasis and false croup
Which family pathogen of parainfluenza belong to?
Pneumoviridae
* Paramyxoviridae
Ortomyxoviridae
Adenoviridae
Reoviridae
What is theduration of incubation period in parainfluenza?
Several hours - 2 days
7-9 days
9-14 days
* 7-2 days
14-21 day
What is most often damaged in parainfluenza?
*The mucous membrane of the larynx and trachea
Lower respiratory tract
Maxillary sinus
Conjunctiva
Nasal mucosa
What complication is typical for parainfluenza?
Real croup
* False croup
Sinusitis
Pneumonia
Pharyngitis
Which family pathogen of respiratory syncytial infection belong to?
Picornaviridae
Orthomyxoviridae
Adenoviridae
Reoviridae
* Paramyxoviridae
Which of acute respiratory disease is characterized by the development of pulmonary edema?
Adenovirus infection
* Flu
Respiratory syncytial infection
Parainfluenza
Psittacosis
What changes in general blood analisis is characteristic in rhinovirus infection?
*The number of leukocytes and ESR are not changed
Significant leukocytosis and increased ESR
Leukocytosis with neutrocytosis
Leukopenia with lymphocytosis
Leukocytosis, anemia, thrombocytopenia
What is the possible complication of respiratory syncytial infection?
Etmoiditis
Otitis
Pneumonia
Sinusitis
* All of the above
Which family agent of rhinovirus infection belong to?
Retroviridae
Paramyxoviridae
*Picornaviridae
Adenoviridae
Reoviridae
What is the duration of incubation period of rhinovirus infection?
Several hours-1 day
1-6 days
*7-10 days
10-14 days
14-20 days
Which family pathogen of metapnevmovirus infection owned to?
Retroviridae
* Paramyxoviridae
Picornaviridae
Adenoviridae
Reoviridae
What is the most effective drug in the treatment of metapneumovirus infection?
Ganacyclovir
Acyclovir
Tamiflu
Remantadin
* Ribavirin
What transfer factor is not typical for coronavirus infection?
Air
Water
* Semen
Blood
Urine
People of what ages is most sensitive for bocavirus infection?
*Children from neonatal period up to 1 year
Children from 1 to 5 years
Children from 6 to 10 years
Teens
Adults
For what viral infection convulsions may occur?
Adenovirus infection
Respiratory syntytsialna
Rynovirusna infection
Parainfluenza
* Flu
What are the symptoms of croup syndrome?
*Inspiratory dyspnea
Expiratory wheeze
Gruff "barking" cough
Spastic cough
Availability of films in the oropharynx
The usage of what drug is not appropriate in mild and moderate forms of flu?
Antiviral
Expectorant
* Antibiotics
Immunostimulators
Desensitizing
What is the most effective drug in the treatment of influenza?
Ganacyclovir
Acyclovir
* Ingavirin
Remantadin
Amizon
For what infection meningeal syndrome is most typical?
Adenovirus infection
Parainfluenza
Rhinovirus infection
* Flu
Respiratory syncytial disease
What infectious disease is characterized by the local reaction of the nasal mucosa with hyperemia, edema and significant secretion?
*Rhinovirus disease
Typhoid fever
Malaria
Varicella
Flu
What viruse more often cause nosocomial infection?
Adenoviruses
Parainfluenza virus
Rhinovirus
Influenza viruses
* Coronavirus
Methods of laboratory diagnosis of acute respiratory viral infections except:
Virological
Serum
* Blood culture
PCR
Flu
Specific methods of laboratory diagnosis of influenza:
Cultivation of the virus in chicken eggs or tissue cultures
Detection of virus antigens by immunofluorescence in nasal washings from
Detection of antibodies to the virus in paired sera of blood
Detection of virus antigens by fluorescent microscopy in smears from the nasal mucosa
* All of the above
What laboratory tests of influenza?
Immunofluorescence method of nasopharyngeal swabs
General blood
Bacteriological investigation of sputum
Biological test on laboratory animals
*All of the above
What are the indications for antibiotics therapy in flu?
Very severe course
Presence of complications
Some age groups (children, elderly people age)
The presence of foci of chronic bacterial infections
* All of the above
What drug is used as etiotropic therapy in adenoviral infection?
Paracetamol
Aspirin
Ceftriaxone
* Dezoxirybonukleaz
All of the above
What indicates possible complications in flu?
Duration of hyperthermia over 5 days
Leukocytosis
Netrophilosis
Accelerated ESR
* All of the above
What is color in fluorescent microscope preparation in case of influenza?
Red
* Green
Blue
Yellow
Violet
What is the entrance gate at infectious mononucleosis?
Mucosa of colon
Mucosa of digestive tract
Epithelial cells of skin
Peyer‘s plate and follicles
*Mucosa of nazo-pharig
What is preparation of specific therapy for viral neuro infection?
* Acyclovir
Cefataxime
Ceftriaxone
Gentamycin
Furazolidon
What is preparation for specific therapy of viral neiroinfection?
Lazix
Cefotaksim
Ceftriakson
* Acyclovir
Prednisolon
What is drug for specific therapy of widespread form of diphtheria of nasopharynx.
Macrolids per os
Penicillin i/m
Cortycosteroid
* Antidiphterial serum i/v
Antitoxic therapy
What is the first dose of antidiphterial serum for a 6 years child with a diphtherial widespread croup:
* 40 AО
15 AО
20 AО
80 AО
60 AО
What is the first dose of antitoxic antidiphtherial serum for a patient with diphtheria of pharynx?
120 thousand of AО
80 thousand of AО
* 30 thousand of AО
50 thousand of AО
150 thousand of AО
At the end of treatment of patients with tonsillitis it is recommended to enter:
500 000 of Bicyllin-3 intramuscular
* 1 500 000 of Bicyllin-5 intramuscular
1 000 000 of Bicyllin-5 intramuscular
1 500 000 of Bicyllin-3 intramuscular
500 000 of Bicyllin-3 intramuscular
Before revaccination from diphtheria of adult persons, are recommended:
* To explore an immune type
To use antibiotics
To use antihistamines
5 years after last revaccination
10 years after last revaccination
Before revaccination from diphtheria of adult persons, are recommended:
* To explore an immune type
To use antibiotics
To use antihistamines
5 years after last revaccination
10 years after last revaccination
What is the exciter of tonsillitis (angina).
* Streptococcus of group A
Streptococcus of group B
Streptococcus of group C
Streptococcus of group D
Streptococcus of group E
Choose, what changes are characteristic for diphtheria of tonsils.
Tonsils enlarged, edematous, on-the-spot of tonsills are some heaved up subephithelial abscesses yellow-white color
In lacunes of tonsils are a pus as yellow-white coat
Tonsils are hyperemic, hypertrophied, on both are necrotizing areas dark grey color, after removing layer by layer of which the deep defect of mucus shell appeared with an uneven bottom
One tonsils hyperemic and filling out, on its surface there is a grey-white coat, under it coat – bleeding ulcer with a smooth bottom
* One tonsil is enlarged, on him dense grey-white color coat, which becomes separated from hardness, mucus bleeds under him
Choose, what changes are characteristic for a follicle tonsillitis (angina).
* Tonsils enlarged, edematous, on-the-spot of tonsils are some heaved up subephithelial abscesses yellow-white color
In lacunes of tonsils are a pus as yellow-white coat
Tonsils are hyperemic, hypertrophied, on both are necrotizing areas dark grey color, after removing layer by layer of which the deep defect of mucus shell appeared with an uneven bottom
One tonsils hyperemic and filling out, on its surface there is a grey-white coat, under it coat – bleeding ulcer with a smooth bottom
One tonsil is enlarged, on him dense grey-white color coat, which becomes separated from hardness, mucus bleeds under him
Choose, what changes are characteristic for a lacunars tonsillitis (angina).
Tonsils enlarged, edematous, on-the-spot of tonsils are some heaved up subephithelial abscesses yellow-white color
* In lacunes of tonsils are a pus as yellow-white coat
Tonsils are hyperemic, hypertrophied, on both are necrotizing areas dark grey color, after removing layer by layer of which the deep defect of mucus shell appeared with an uneven bottom
One tonsils hyperemic and filling out, on its surface there is a grey-white coat, under it coat – bleeding ulcer with a smooth bottom
One tonsil is enlarged, on him dense grey-white color coat, which becomes separated from hardness, mucus bleeds under him
Choose, what changes are characteristic for a ulcers-necrotic tonsillitis (angina).
Tonsils enlarged, edematous, on-the-spot of tonsils are some heaved up subephithelial abscesses yellow-white color
In lacunes of tonsils are a pus as yellow-white coat
* Tonsils are hyperemic, hypertrophied, on both are necrotizing areas dark grey color, after removing layer by layer of which the deep defect of mucus shell appeared with an uneven bottom
One tonsils hyperemic and filling out, on its surface there is a grey-white coat, under it coat – bleeding ulcer with a smooth bottom
One tonsil is enlarged, on him dense grey-white color coat, which becomes separated from hardness, mucus bleeds under him
Choose, what changes are characteristic for a Vensan-Plaut‘s tonsillitis.
Tonsils enlarged, edematous, on-the-spot of tonsils are some heaved up subephithelial abscesses yellow-white color
In lacunes of tonsils are a pus as yellow-white coat
Tonsils are hyperemic, hypertrophied, on both are necrotizing areas dark grey color, after removing layer by layer of which the deep defect of mucus shell appeared with an uneven bottom
* One tonsils hyperemic and filling out, on its surface there is a grey-white coat, under it coat – bleeding ulcer with a smooth bottom
One tonsil is enlarged, on him dense grey-white color coat, which becomes separated from hardness, mucus bleeds under him
Complication of diphtheria of larynx is:
Myocarditis
Paresis of auditory nerve
Nephrosonephritis
* Cereals
Poliomyelitis
Complications of 4-5th week of diphtheria are:
Encephalitis
Bulbar disorders, pancreatitis, hepatitis
* Poliomyelitis, myocarditis
Nephrosonephritis
Stenotic laryngotracheitis
Complications which often develop on the first week of diphtheria of otopharynx are:
Poliomyelitis
Asphyxia
Insufficiency of glandulars
hepatospleenomegaly
* Paresis of soft palate
Diphtheria planned vaccination begin in:
In first days after birth of child
* In 3 month age
In 6-month age
In 1 year
In 6 years
Early complications of diphtheria of otopharynx is:
* Paresis of soft palate
Pneumonia
Asphyxia
Croup
Poliomyelitis
Especially high titre of ant diptheria antitoxic antibodies testifies in:
Recovering
Acute period of diphtheria
* Bacteriocarriering
Forming of immunity to diphtheria
About nothing does not testify
Etiology agent of meningitis is:
* Neisseria meningitides
Entamoeba histolytica
Vibro cholerae
Clostridium botulinum
Campylobacter pylori
For corynebacterium diphtheria is typical:
Contain endotoxin only
* Exotoxin production
Exotoxin does not product
Enterotoxin production
Myelotoxin production
For the treatment of acidosis in meningococcal meningitis is better to use.
10-20 % glucose solution
10 % chloride solution
* 4 % sodium bicarbonate solution
Albumen
Concentrated dry plasma
What disease is typical changes in blood (presence of atypical mononucleares)?
Flu
* Infectious mononucleosis
Measles
AIDS
Diphtheria
What etiotropic (antistreptococcal) facilities are the most effective :
Furazolidonum
Gentamicin
* Benzilpenicilin and Oxacillinum
Benzilpenicilin and Furazolidonum
Doksiciklin and Gentamicin
How is it possible to specify the diagnosis of meningococcal meningitis.
Meningitis is primary
Presence of a lot of cells in the CSF
Presence of gram-negative diplococcus in CSF
Meningococes from the throat
* All the above
How long is the incubation period of a tonsillitis (angina)?
From a few hours to 5 days
From a few hours to 4 days
From a few hours to 3 days
* From a few hours to 2 days
From a few hours to 1 days
How long is the period of fever in patients with a tonsillitis (angina)?
1-2 days
2-3 days
* 3-5 days
5-7 days
..More than week
Name the exciter of acute tonsillitis:
α -hemolytic streptococcus of group A
γ -hemolytic streptococcus of group A
α -hemolytic streptococcus of group C
*β -hemolytic streptococcus of group C
β -hemolytic streptococcus of group A
The source of exciter of tonsillitis is:
patient with angina
patient with erysipelas
patient with the scarlet fever
healthy transmitter of hemolytic streptococcus
*all listed above
What is the basic mechanism of transmission of tonsillitis?
*air-droplet
alimentary
contact
transmisive
vertical
How long last the incubation period in tonsillitis?
from a few hours to 5 days
from a few hours to 4 days
from a few hours to 3 days
*from a few hours to 2 days
from a few hours to 1 days
Angina begins sharply, from headache, increase of temperature of body, dull ache in joints and chill. What other characteristic syndrome of appears simultaneously (rarer in the end of 1st days)?
nausea
vomiting
*pain in the throat
pain in the stomach
tachycardia
A frequent and early symptom of tonsillitis is an enlargement of lymph nodes, their pain. Which group of lymph nodes enlarge first of all?
posterior-neck
occypital
supraclavicular
*submandibular
anterior-neck
What is the average duration of fever period in tonsillitis?
days
2-3 days
*3-5 days
5-7 days
More one week
What kinds of angina can be distinguish according to the changes in a tonsils?
Catarrhal, follicle and lacunar
*Catarrhal, follicle, lacunar and necrotic-ulcerous
Catarrhal, follicle, lacunar, pellicle and necrotic-ulcerous
Follicle, lacunar and necrotic-ulcerous
Follicle, lacunar, pellicle and necrotic-ulcerous
What is the medicine for specific therapy of widespread form of nasopharynx diphtheria:
Macrolids per os
Penicillin i/m
Cortycosteroid
*Antidiphterial serum i/v
Antitoxic therapy
Specify the correct method of serum introduction after the Bezredko method:
1,0 ml of divorced 1:100 hypodermic – through 30 min. 0,1 ml of undivorced hypodermic – through 30 min. all dose of intramuscle
0,1 ml of divorced 1:1 000 endermic – through 30 min. 0,1 ml of divorced 1:10 hypodermic – through 30 min. all dose of intramuscle
0,1 ml of undivorced endermic – through 30 min. 0,1 ml hypodermic – through 30 min. all dose of intramuscle
*0,1 ml of divorced 1:100 endermic – through 30 min. 0,1 ml of undivorced hypodermic – through 30 min. all dose of intramuscle
1,0 ml of divorced 1:10 hypodermic – through 30 min. 0,1 ml of undivorced hypodermic – through 30 min. all dose of intramuscle
What is characteristic signs of raid at diphtheria?
One-sided, grey-white, on-the-spot crateriform ulcers
*Grey-white, dense with clear edges and brilliant surface
Yellow-white, fragile, perilacunar is located
One-sided, yellow-white, in lacunas
White, fragile, is easily taken off by a spatula
What is the exciter of diphtheria:
Virus of Epshtein-Barr
*Leffler Bacillus
Corynebacteria ulcerans
Fusiform stick
Corynebacteria xerosis
Etiology agent of meningitis are accept:
Staphylococci
Neisseria meningitides
Mycobacterium tuberculosis
Viruses
*Entamoeba histolytica
Wich of these symptoms are often present in patients with meningitis?
Profuse watery diarrhea, vomiting, dehydratation, muscular cramps
What laboratory methods should be taken to discharge meningitis?
*Lumbar puncture
Serologic detection
Urine examination
Coprograma
Biopsy of tissues
Source of meningitis is:
Animals
Birds
Fish
Pediculus humanus
*People
How is it possible to specify the diagnosis of meningococcal meningitis.
Meningitis is primary
Presence of a lot of cells in the CSF
Presence of gram-negative diplococcus in CSF
Meningococes from the throat
*All the above
What are the rules at taking of smear material on the discovery of meningococal infection?
The taken away material at drawing out must not touch only mucus shell of cheeks and tongue
The taken away material at drawing out must not touch only teeth and tongue
The taken away material at drawing out must not touch only teeth, mucus shell of cheeks
*The taken away material at drawing out must not touch teeth, mucus shell of cheeks and tongue
The taken away material at drawing out can touch teeth, mucus shell of cheeks and tongue
What temperature terms is it needed for cultivation of meningococcal on artificial mediums?
23-40 °C
35-43 °C
*35-37 °C
23-35 °C
37-39 °C
When does the laboratory give the results of bacteriological examination of smear from throat?
On 2th days
On 3th days
*On 4th days
On 5th days
On 6th days
What is taken for serum research for confirmation of meningococcal infection?
*Blood
Mucus
Urine
CSF
Saliva
What antibiotics preparations of choice of etiotropic therapy at a meningococcal infection.
* Benzylpenicillin and it derivatives
Gentamycin
Cefazolin
Sulfolamide
Ciprofloxacin
In what dose should| benzyl penicillin be administered at meningococcal meningitis?
From a calculation 100-300 thousands unit on 1 kg of mass of body on days
*From a calculation 200-500 thousands unit on 1 kg of mass of body on days
From a calculation 500-700 thousands unit on 1 kg of mass of body on days
From a calculation 700-900 thousands unit on 1 kg of mass of body on days
Regardless of mass of body
In what daily interval should the dose of benzylpenicillin at meningococcal meningitis administered.
2 hrs
*4 hrs
6 hrs
5 hrs
8 hrs
Which preparation has a bacteriostatic action, and is more expedient to begin etiotropic therapy in the case of infectious toxic shock.
From benzylpenicillin and its derivatives
From ciprofloxacin
From gentamycin
From ciprofloxacin
*From levomycitin of succinate
A patient is sick with meningococcal meningitis. He take a massive dose of penicillin. 4 days temperature of body 36,6-36,8 °C. Meningeal signs are negative. When is it possible to stop the antibiotic therapy.
*At a cytosis in a CSF 100 and less, lymphocytes prevail
After 10 days from the beginning antibiotic therapy
After 7 days from the beginning antibiotic therapy
At a cytosis 100 and less, neutrophil prevail
From 6 days from the beginning antibiotic
A patient with meningococcal meningitis gets penicillin during 7 days. The temperature of body is normal 4 days. Meningeal signs are negative. When is it possible to stop the antibiotic therapy.
In default of leucocytosis displacement in blood
*. At a cytosis in a neurolymph 100 and less, lymphocyte prevail
At a cytosis in a neurolymph 100 and less, neutrophil prevail
At a cytosis in a neurolymph 150, lymphocyte prevail
At once immediately
For the treatment of acidosis at meningococcal meningitis is better to use.
10-20 % glucose solution
10 % chloride solution
*4 % sodium bicarbonate solution
Albumin
Concentrated dry plasma
Meningococemia and ID-syndrome require above all things.
Administration of diuretics
Administration of analgesic
*Administration of heparin
Administration of vitamins
Administration of antihistaminic preparations
What is used as specific prophylaxis in the period of epidemic spreading of meningococcal infection.
Immun globulin
Serum
*Vaccine
Anatoxin
Nothing
What measures are conducted in the place of meningococcal infection?
Supervision during 2 weeks
Phagoprophylaxis
Immunization
*Bacteriological inspection of contact
Chemoprophylaxis
What complication has developed in patient with diphtheria of mouth pellicle severe form was diagnosed. On the 6th day of disease when pain in the heart region, palpitation were appeared. Pulse – 120 per 1 min, systolic noise on apex of heart. On ECG is incomplete blockade of left leg of Giss bunch?
*Early infectious-toxic myocarditis
Myocardial dystrophy
Heart attack of myocardium
Acute cardio-vessel insufficiency
Stenosis of mitral valve
What group of infectious diseases diphtheria belong to?
Sapronosis
Zoonosis
*Anthroponosis
Zooanthroponosis
A group is not certain
What is the properties of сorynebacterium diphtheria:
Contain endotoxin only
*Exotoxin products
Exotoxin does not product
An enterotoxin products
Myelotoxin products
The source of infection at diphtheria is:
*Sick people and carriers
Sick agricultural animals
Rodents
Mosquitoes
Aerosol of saliva and epipharyngeal mucous of patients
What is mechanism of transmission of Corynebacterium diphtheria?
Vertical
Transmissive
*Air-drop
Contact
Parenteral
Especially high titre of ant diptherial antitoxic antibodies testifies to:
Incubation
Acute period of diphtheria
*Bacteriocarriering
Forming of immunity to diphtheria
About nothing does not testify
What group of infectious diseases by L. Gromashevsky classification diphtheria belong to?
External covers
Blood
Intestinal
*Respiratory
Transmissive
What is transmissive factors in diphtheria?
Blood
Water
*Saliva
Urine
Exrements
What is seasonal character of diphtheria?
Spring-summer
Summer-autumn
*Autumn-winter
Winter-spring
Spring-autumn
Before revaccination from diphtheria of adult persons, they are recommended:
*To explore an immune type
To use antibiotics
To use antihistamines
5 years after last revaccination
10 years after last revaccination
Diphtheria planned vaccination begin in:
In first days after birth of child
*In 3 month age
In 6-month age
In 1 year
In 6 years
In preschool is case of disease on diphtheriA. What prophylactic measures must be conducted above all things?
Urgent hospitalization
Urgent vaccination
*Quarantines measures
Urgent by chemical prophylactic antibiotics
Introduction of antidiphterial whey
At a child 6 years with a diphtherial widespread croup the first dose of antidiphterial serum makes:
*40 AО
15 AО
20 AО
80 AО
60 AО
What material it’s necessary to take for bacteriologic examination in suspicion on diphtheria?
Excrement
Blood
Urine
*Mucous
Neurolymph
What is main complication of diphtheria of larynx:
Myocarditis
Paresis of auditory nerve
Nephrosonephritis
*Croup
Poliomyelitis
What complications more often develops during the first week of diphtheria of otopharynx:
Poliomyelitis
Asphyxia
Paratonsillitis
Hepatospleenomegaly
*Paresis of soft palate
What is early complications of diphtheria of otopharynx:
*Paresis of soft palate
Pneumonia
Asphyxia
Croup
Poliomyelitis
What complications more often develops during 4-5th week of diphtheria:
Encephalitis
Bulbar disorders, pancreatitis, hepatitis
*Poliomyelitis, myocarditis
Nephrosonephritis
Stenotic laryngotracheitis
What are the rules of hospitalization of patients with infectious mononucleosis?
Patients are not hospitalized
In a chamber for the infections of respiratory tracts
*In a separate chamber
In a chamber for the infections of external covers
In a chamber for intestinal infections
What additional inspections must be conducted to the patient with infectious mononucleosis?
*IFA on HIV-infection, bacteriology inspection on diphtheria
IFA on HIV-infection, bacteriology inspection on a rabbit-fever
Bacteriology inspection on diphtheria and typhoid
Reaction of Burne and Rihth-Heddlson
Reaction of Paul-Bunnel and punction of lymphatic knot
What from the following symptoms are not characteristic of infectious mononucleosis?
Fever
*Defeat of kidneys
Lymphadenopathy
Tonsillitis
Increasing of liver and spleen
For what disease characterize changes in a blood (presence of lymphomonocytes and atypical mononuclears)?
Flu
*Infectious mononucleosis
Measels
AIDS
Diphtheria
What additional test should hold for the patient with infectious mononucleosis?
Burne and Wright-Hadlson‘s reactions
ELISA-test, bacteriological test for tularemia
Bacteriological test for diphtheria and typhoid fever
*ELISA-test, bacteriological test for diphtheria
Paul-Burne reaction and lymph node puncture
What the most possible complication occurs during infectious mononucleosis?
Meningitis
Autoimmune alopecia
Encephalitis
*Splenic rupture
Obstruction of respiratory tract
The source of infection at infectious mononucleosis is:
*Sick people and carriers
Sick agricultural animals
Rodents
Mosquitoes
Aerosol of saliva and epipharyngeal mucous of patients
What is seasonal character of infectious mononucleosis?
Spring-summer
Summer-autumn
Autumn-winter
*Winter-spring
Spring-autumn
After the disease which was accompanied by the fever and pharyngalgias, there were an odynophagia, dysarthria, weakness and violation of motions in hands and feet, hyporeflexia, violation of sensitiveness in extremities to the polyneurotic type. What disease does it follow to think about above all things?
Neuropathy of hypoglossus
*Diphtherial polyneuropathy
Neuropathy of glossopharyngeus nerve
Trunk encephalitis
Pseudobulbar syndrome
What is immediately investigation in suspicious of diphtheria:
Strokes with tonsills, nose or other areas for the exposure of diphtherial stick
IFA
*Microscopy (painting by Neiser)
Haemoculture
RDHA with a diphtherial diagnosticum
Etiology agent of meningitis is:
*Neisseria meningitides
Entamoeba histolytica
Vibrio cholerae
Clostridium botulinum
Campylobacter pylori
Wich of these symptoms are often present in patients with meningitis?
Algor, high temperature, headache
What group of infectious diseases meningococcal infection belong to:
Intestinal
Blood
*Respiratory
Transmissive
External covers
What is the mechanism of transmission of meningococcal infection?
Fecal-oral
Contact
Transmissive
*Air-drop
Vertical
What is seasonal character of meningococcal infection?
Summer-autumn
Autumn-winter
*Winter-spring
Winter
Summer
What clinical form of meningococcal infection more often may happened?
Meningococcemia
Meningitis
Meningoencephalitis
*Nasopharengitis
Pneumonia
What syndrome may appear in severe meningococcemia?
Paul-Bunnel
Plaut-Vincent
Jarish-Gersgeimer
Gien-Barre
*Waterhause-Friedrichsen
What laboratory methods should be taken to discharge meningitis?
*Lumbar puncture
Serologic detection
Urine examination
Coprograma
Biopsy of tissues
Source of meningitis is:
Animals
Birds
Fish
Pediculus humanus
*People
How is it possible to specify the diagnosis of meningococcal meningitis.
Meningitis is primary
Presence of a lot of cells in the CSF
Presence of gram-negative diplococcus in CSF
Meningococes from the throat
*All the above
What are the rules at taking of smear material on the discovery of meningococal infection?
The taken away material at drawing out must not touch only mucus shell of cheeks and tongue
The taken away material at drawing out must not touch only teeth and tongue
The taken away material at drawing out must not touch only teeth, mucus shell of cheeks
*The taken away material at drawing out must not touch|| teeth, mucus shell of cheeks and tongue
The taken away material|| at drawing out can touch|| teeth, mucus shell of cheeks and tongue
When does the laboratory give the results of bacteriological examination of smear from throat?
On 2th days
On 3th days
*On 4th days
On 5th days
On 6th days
What is taken for serum research for confirmation of meningococcal infection?
*Blood
Mucus
Urine
CSF
Saliva
What antibiotics preparations of choice of etiotropic therapy at a meningococcal infection.
*Benzylpenicillin and it derivatives
Gentamycin
Cefazolin
Sulfolamide
Ciprofloxacin
In what dose should| benzyl penicillin be administered at meningococcal meningitis?
From a calculation 100-300 thousands unit on 1 kg of mass of body on days
*From a calculation 200-500 thousands unit on 1 kg of mass of body on days
From a calculation 500-700 thousands unit on 1 kg of mass of body on days
From a calculation 700-900 thousands unit on 1 kg of mass of body on days
Regardless of mass of body
In what daily interval should the dose of benzylpenicillin at meningococcal meningitis administered.
2 hrs
*4 hrs
6 hrs
5 hrs
8 hrs
Which preparation has a bacteriostatic action, and is more expedient to begin etiotropic therapy in the case of infectious toxic shock.
From benzylpenicillin and its derivatives
From ciprofloxacin
From gentamycin
From ciprofloxacin
*From laevomycitin succinate
For the treatment of acidosis at meningococcal meningitis is better to use.
10-20 % glucose solution
10 % chloride solution
*4 % sodium bicarbonate solution
Albumen
Concentrated dry plasma
Meningococсemia and DIC-syndrome require above all things.
Administration of diuretics
Administration of analgesic
*Administration of heparin
Administration of vitamins
Administration of antihistaminic preparations
What is used as specific prophylaxis in the period of epidemic spreading of meningococcal infection.
Immune globulin
Serum
*Vaccine
Anatoxin
Nothing
A patient with meningococcal meningitis gets penicillin during 7 days. Last 4 days temperature of body is normal. Meningeal signs are absent. When is it possible to abolish an antibiotic?
*At cytosis in liquor 100 and less, lymphocytes prevails
At absence of leukocytosis and stab-nucleus shift in a blood
At cytosis in liquor 100 and more less, neutrophils prevails
At cytosis in liquor 150, lymphocytes prevails
At once
What measures are conducted in the place of meningococcal infection?
Supervision during 2 weeks
Phagoprophylaxis
Immunization
*Bacteriological inspection of contact
Chemoprophylaxis
Violations of electrolyte balance show up at leptospirosis:
*Metabolic acidosis
By a metabolic alkalosis
Respirator acidosis
By a respiratory alkalosis
All above enumerated
The decline of arteriotony at a leptospirosis is not caused:
By expansion of vessels under the action of toxin
Hypovolemia
By myocarditis
*Hypercalcgesty
By adrenal insufficiency
All are the clinical signs of measles except:
Acute beginning of high fever
*Icterus
Maculo-papular rash
Sequential appearance of rash
Scaling
For how long a patient with complicated form of measles should be isolated:
For 4 days from the beginning of rash
For 7 days from the beginning of rash
*For 10 days from the beginning of rash
For 17 days from the beginning
For 20 days from the beginning of illness
Term of contagious period of patient diagnosed with uncomplicated form of measles
Until clinical recovery
After rash starts disappearing
Before appearance of rash
*4 days from the beginning of rash
10 days from the beginning of illness
What is the duration of quarantine in child's establishment in case of rubella?
5 days after the isolation of the last patient
11 days
21 day
10 days
No need for quarantine
*5 days after isolation of the last child
What is duration of contagious period for a patient with epidemic parotitis?
21 days
First week of illness
First 10 days from the beginning of disease
Whole period of clinical symptoms
*First 9 days of disease.
What measures should be taken in regards to persons, who were in contact with a patient diagnosed with epidemic parotitis?
Observation after contact people during a maximal length of incubation period
Quorantin in child's establishment
Isolation of people who were in contact with ill from 11th to the 21t day of illness
Isolation of children up to 10 years old, who were not ill with epidemic parotitis, for 21 day from a moment of contact
*All above enumerated
What is the duration of contagious period for a patient diagnosed with scarlet fever?
10 days from the beginning of illness
Until patient is discharged from the hospital
Until rash is present
*Till the 22d day from the beginning of illness
Not contagious
What is duration period of supervision after ill with scarlet fever?
*7 days from time of contact
21 day
Till patient’s rash is present
Till patient is discharged from permanent establishment
Not conducted
Methods of specific prophylaxis of scarlet fever:
Isolation of ill
Vaccination
Use of antibiotics
Disinfection
*Does not exist
What are the anti epidemic measures in regards to people who were in contact with chicken-pox patient:
*Separation and limit of contacts with others
Vaccination
Use of antibiotics
Disinfection
Does not exist
Measures of urgent prophylaxis for unvaccinated children who have never been ill with measles in case of exposure to an ill with measles
Separation from the source
*Vaccination
Administration of antibiotics
Disinfection
Does not exist
Measures of urgent prophylaxis of measles for contacts which have never been ill, but were vaccinated against measles
Separation from the source
Vaccination
Use of antibiotics
Use of immunoglobulin
*No need to conduct
Measures of urgent prophylaxis of measles for people who had been ill with measles, but never have been vaccinated
Separation from the ill
Vaccination
Use of immunoglobulin
*Use of antibiotics
No need to conduct
Possible side effects at application of antibiotics are all, except.
Stomach-ache
Nausea, vomit
Diarrhea
*Fever
Skin rash
Contra-indication for the application of antibiotics are all, except.
An increased sensitiveness to preparation
Severy disorders of liver
Severy disorders of kidneys
Period of pregnancy and lactation
*Prolonged fever
Principles of etiotropic therapy of sepsis.
Administration of antibiotics quick as possible
Administration of antibiotics in maximal therapeutic doses
In accordance to credible microbiological diagnosis
An account of possible of therapeutic concentration of antibiotic in field of infection
*All the above
Immune modulator therapy of sepsis are all, except.
Ronkoleykin
Interferon
Inductors of interferon
*Vaccine
Normal human immune globuline
Basic principles of antibiotics therapy.
A selection of antibiotic after the sensitiveness of the selected exciter
selection of antibiotic according to the diagnosis of certain infectious disease clinically
Choose the most active preparation
Choose the least toxic preparation
*All the above
Basic principles of antibiotics therapy of sepsis.
A selection of preparation from data of bacteriostatic
Determination of dose, method and multiple of introduction the preparation
Timeliness and definite duration of introduction input of antibiotic
D Combining antibiotics between itself for enhancement of antibacterial effect
*E. All the above
Agglutinines at a leptospirosis arrive at a maximal titre:
On the third day of illness
*On the third week of illness and later
On the fourth week of illness
On the second month of illness
To the second week of illness
At a leptospirosis the exposure of antibodies is considered reliable in a titre:
1:70 and anymore
1:80 and anymore
1:60 and anymore
*1:100 and anymore
1:40 and anymore
At treatment of patients with leptospirosis antibiotics consider most effective:
*Penicillin
Macrolids
Cefalosporins
Ftorhinolons
Sul'fanilamids
Etiotropic therapy of leptospirosis includes:
Oxyhinolons
Sulfanilamids
Nitrofurans
Antitocidns
*Antibiotics
What from antibiotics more expedient to use for treatment of leptospirosis?
Macrolids
Tetracyclins
Aminoglicosids
*Penicillins
Metrogil
A patient has a severy icteric form of leptospirosis. What from antibiotics is better to appoint as etiotropic therapy?
Yunidoks
Tetracyclinum
Azitromicin
Rovamicin
*Penicillin
Treatment of leptospirosis:
Desintoxication, dehydratation, antibiotics, glukokorticosteroids
Daily allowance doses of penicillin at treatment of leptospirosis:
2-3 million units
*3-12 million units
20 million units
40 million units
Over 40 million units
With the purpose of immunotherapy it is better to apply at treatment of leptospirosis:
Immunodepressants
Antihistaminics
Horse whey
*Antileptospirosis human immunoprotein
Antileptospirosis neat immunoprotein
What from antibiotics are more effective in treatment of icteric form of leptospirosis:
*Penicillins
Aminoglicozids
Tetracyclins
Macrolids
Metrogil
For the prophylaxis of leptospirosis use:
*Active vaccine
Anavaccine
Toxoid
All
Antibiotics
Vaccinations against leptosprosis perform for:
All
Only villagers
Only to the habitants of endemic districts
*Only to the persons busy in the stock-raising
It is not conducted
Which serotypes of leptospirosis caused the disease more frequent:
L. interogans
L. grippotyphosa
L. canicola
*L. icterohaemorrhagia
L. Pomona
All of these have an epidemic dangerous exept:
Farm animals
Wide rodents
Domestic animals
Foxes
*Human
How long lasts the leptospirosis incubation period:
2 month
1-7 days
*7-14 days
14-21 days
2-3 days
The main of antibiotics which is used in treatment of leptospirosis:
Tetracyclin
Tetraolean
Erythromycin
Streptomycin
*Penicillin
When there can be such specific complication of typhoid fever, like to intestinal bleeding?
On the 1st week of illness
On the 2nd week of illness
*On the 3rd week of illness
On the 4th week of illness
On the any week of illness
When there can be such specific complication of typhoid fever, like to perforation of bowel?
On the 1st week of illness
On the 2nd week of illness
*On the 3rd week of illness
On the 4th week of illness
On the any week of illness
Who is the source of typhoid fever?
*Sick people
Sick agricultural animals
Sick rodents
Soil
Defecating of patients
Salmonella typhi contains:
Only O-antigen and Н-antigen
Only O-antigen and Vi-antigen
Only H-antigen and Vi-antigen
*O-antigen, H-antigen and Vi-antigen
O-antigen, H-antigen, Vi- antigen and HBsAg
When is it possible to abolish etiotropic preparations in a patient with typhoid fever?
At once after normalization of temperature of body
After normalization of sizes of liver and spleen
After disappearance of roseollas
In 10 days after disappearance of roseollas
*After the 10th day of normal temperature of body
By which method is it possible to find out bacterial carrier in case of typhoid fever?
Coproculture
Reaction of agglutination of Vidall
Indirect hemaglutination test with О-аntigen
Indirect hemaglutination test with a Н-antigen
*Indirect hemaglutination test with a Vi-antigen
Typhoid bacilli are usually cultured from:
*Blood, stool, urine
Blood, urine, sputum
Stool, liquor, urine
Blood, stool, sputum
Stool, liquor, sputum
A suspected case of typhoid fever of 1st week is admitted in the hospital. What examination (laboratory diagnosis) do you suggest for this patient?
Coproculture
Reaction of agglutination of Vidall
Indirect hemaglutination test with О-, Н-, Vi-аntigens
Urinoculture
*Hemoculture
A suspected case of typhoid fever of 3nd week is admitted in the hospital. What examination (laboratory diagnosis) do you suggest for this patient?
Coproculture
Reaction of agglutination of Vidall
Urinoculture
Hemoculture
*All about it
What from the transferred signs is not characteristic for a typhoid rash?
*Papular, disappears together with normalization of temperature of body
Appears on a 7-10th day, rosella-type
Located mainly on a abdomen and lateral surfaces of trunk, observed at the half of patients
The amount of elements is limited, pours in addition
rosella-type, sometimes saved longer than fever
What ever symptom is not characteristic for typhoid on the second week of illness?
Constipation
Headache
Fever
Relative bradycardia
*Cramps
What changes in general analysis of blood are characteristic for typhoid?
*Leykopenia, aneosiniphilia, lymph-, monocytosis, enhanceable RSE
What does the diagnostic titre of reaction of Vі-haemaglutination testify to?
*About typhoid bacterial-carrier
About the period of height of the epidemic typhus
About meningococcaemia
About a malaria
About the latent period of brucellosis
What term of looking after the hearth of typhoid?
14 days
*21 days
7 days
30 days
Does not look after
Reconvalescente of typhoid fever may go out from clinic after:
Non-permanent negative bacteriologic examination of defecating
*21th day of normal temperature of body and 3-multiple negative bacteriologic examination of excrement and urine
of 14th day of normal temperature of body and 2-multiple negative bacteriologic examination of excrement and urine
Clinical convalescence and normalization of rectal manoscopic picture
Normalisations of rectal manoscopic picture and in default of title of antibodies in RNGA
In epydfocus of typhoid fever doing, except for:
Daily thermometery
Coprologic culture
Reaction of Vidal
*Haemoculture
Urine culture
For establishment of transmitter of Salmonella typhi utilize:
Test of Cuverkalov
RA with O- and Н-antigen
PCR
Bacteriologic examination and reaction of Vidal
*Bacteriologic examination and RNGA with a Vi-antigen
What from the adopted ways of transmission is characteristic for typhoid?
*Alimentary
Contact
Transmission
Air-drop
Vertical
What environments do typhoid sticks grow on well?
Chicken embryos
water-whey nourishing environment
*Bilious clear soup
Meat-peptone gelose + cistin
To the Bismute-sulfate gelose
Name of the basic factors of pathogen of typhoid stick?
*Vi-antigen and endotoxin
Exotoxin
Vi-antigen
Enzymes of pathogenicity
Endotoxin
Duration of latent period at typhoid?
3-7 days
*9-21 day
From a few hovers to 2-3 days
From 12 to 100 days
From a few hovers to 17 days
The part of reproduction of typhoid bacterium in the organism of man is:
Stomach
*Lymphatic formations of colon
Blood
Bilious ways
Mucous membrane of colon
What from the adopted phases of pathogenesis is not characteristic for typhoid?
*Swelling, edema of mucous membrane of overhead respiratory tracts
Stage of penetration
Stage of lymphodefence reactions
Stage of bacteriaemia
Stage of intoxication
What from the indicated pathology anatomic phases is not characteristic for typhoid?
*Catarrhal inflammation of amygdales
Cerebral-type of swelling
Necrosis
Ulcers
Clean ulcers
What is entrance gates of typhoid fever agent?
Mucous membrane of amygdales
Mucous membrane of nasopharynx
Epithelial cells of skin
Mucous membrane of colon
*Mucous membrane of digestive tract
Who is the source of epidemic typhus?
Patients with epidemic typhus
Patients with disease Brill-Zinsser
*Patients with epidemic typhus and disease Brill-Zinsser
Patients with Brill-Zinsser disease and Sachs disease
Patients with epidemic typhus and abdominal typhoid
During what time the lice can transfer the epidemic typhus?
Up to 10 days
Up to 15 days
Up to 20 days
*Up to 30 days
Up to 40 days
What is Brill's disease?
Vertiacal borne
*Remote relapse typhus
Early relapse typhus
Re-infection rickettsia
Self nozological unit
Often, in patient with epidemic typhus is tongue‘s tremor when protrusion that sticked on the lower teeth. What term did it call?
Symptom of Heller
Conjunctivitis
*Govorov-Godele symptom
Zorohovich-Chiari symptom
Enantema Rosenberg‘s
What is not typical for epidemic typhus exanthema?
*Arise on 7-10-day of illness
Has rosy-petehia nature
Localized mainly on the lateral surface of the torso and limbs flexion surfaces
It can grab his hands and feet, but never on the face
Abundant
What is not typical for epidemic typhus exanthema?
Disappear with the drop in temperature
Never appeared another rash
Roseola saved up to 6 days, petehii – 12
Leave a little pigmentation and poor peeling
*Single elements
What is not typical of blood in the severe epidemic typhus?
Neutrofil leukocytosis
Academy of Sciences or hypleozinofil
Limphopeniya
*Anemia
The increase in ESR
What is not typical for the analysis of urine in the severe epidemic typhus?
Proteinuria
Single-cylinder hyaline
*Multiple granular cylinders
A small number of erythrocytes
A small number of leukocytes
When is serological diagnosis possible in patients with epidemic typhus?
From the 1st day of illness
On the 2nd day illness
From the 3-4th day illness
From 4-5th day of illness
*From the 5 to 7th day of illness
What diagnostic titer response agglutination test with typhus rickettsia?
1:40 and above
1:80 and above
*1:160 or higher
1:320 or higher
1:640 or higher
In with titre will be positive reaction of agglutination in patient with epidemic typhus in droplets test when Mosing?
*1:40 and above
1:80 and above
1:160 or higher
1:320 or higher
1:640 or higher
For contact persons in the centre of the epidemic typhus establish surveillance over:
21 days
25 days
*51 days
72 days
3 months
During the contact person with the Brill‘s disease establish surveillance over:
21 days
*25 days
51 days
72 days
3 months
In the case of head lice carry out sanitation: hair cut, followed by incineration, processing hair. What kind of insecticide products for this use?
0,5 % solution of water emulsions karbofosa
0,5 % metilatsetofos
10 % solution of liquid neutral metilatsetofosa soap
0,5 % water emulsion dikrezilu
*3 % soap RHTSG
When patients after epidemic typhus may go out from clinic?
*After clinical recovery, but not before the 12-day normal temperature
After a full clinical recovery
After clinical recovery, but not before the 12-day period following the lifting of antibiotics
At the 12-day normal body temperature
After clinical recovery, but not earlier than the 9-day normal body temperature
Whit antibiotics are less effective from the transferred at the epidemic fever?
Tetracyclin
Metacyclin
*Levomicetyn
Vibramycinum
Doxyciclin
Who is the source of epidemic typhus?
Patients with epidemic typhus
Patients with disease Brill-Zinsser
*Patients with epidemic typhus and disease Brill-Zinsser
Patients with Brill-Zinsser disease and Sachs disease
Patients with epidemic typhus and abdominal typhoid
When sick people gets epidemic typhus infection, which period affects more?
Over the past 2 days, the incubation period and 2-3 days after lowering temperature
All hectic period and 2-3 days after lowering temperature
2-3 days after lowering temperature
*Over the past 2 days, the incubation period, all febrile period and 2-3 days after lowering temperature
Over the past 2 days, the incubation period and the hectic period
On which period the maximal growth of infection occurs during epidemic typhus disease?
At the incubation period
*At the 1th week of illness
At the 2nd week of illness
At the 3rd week of illness
At the time of recovery
During what time the lice can transfer the epidemic typhus?
Up to 10 days
Up to 15 days
Up to 20 days
*Up to 30 days
Up to 40 days
What is Brill's disease?
Vertiacal borne
*Remote relapse typhus
Early relapse typhus
Re-infection rickettsia
Self nozological unit
Often, in patient with epidemic typhus arise transition petehies in the conjunctivA. What term did it call?
Symptom of Heller
Conjunctivitis
Symptom of Govorova-Godele
*Symptom of Zorohovich-Chiari-Avtsyna
Enantema Rosenberg‘s
Often, in patient with epidemic typhus arises petehies on mucosal soft palate. What term did it call?
Symptom of Heller
Conjunctivitis
Symptom of Govorova-Godele
Symptom of Zorohovich-Kiari
*Enantema Rosenberg‘s
Often, in patient with epidemic typhus is tongue‘s tremor when protrusion that sticked on the lower teeth. What term did it call?
Symptom of Heller
Conjunctivitis
*Symptom of Govorova-Godele
Symptom of Zorohovich-Chiari
Enantema Rosenberg‘s
What is not typical for epidemic typhus exanthema?
*Arise on 7-10-day of illness
Has rosy-petehia nature
Localized mainly on the lateral surface of the torso and limbs flexion surfaces
It can grab his hands and feet, but never on the face
Abundant
What is not typical for epidemic typhus exanthema?
Disappear with the drop in temperature
Never appeared another rash
Roseola saved up to 6 days, petehii – 12
Leave a little pigmentation and poor peeling
* Arise on 7-10-day of illness
What is not typical of blood in the severe epidemic typhus?
Neutrofil leukocytosis
Academy of Sciences or hypleozinofil
Limphopeniya
*Anemia
The increase in ESR
What is not typical for the analysis of urine in the severe epidemic typhus?
Proteinuria
Single-cylinder hyaline
*Multiple granular cylinders
A small number of erythrocytes
A small number of leukocytes
When is serological diagnosis possible in patients with epidemic typhus?
From the 1st day of illness
On the 2nd day illness
From the 3-4th day illness
From 4-5th day of illness
*From the 5 to 7th day of illness
What diagnostic titer response agglutination test with typhus rickettsia?
1:40 and above
1:80 and above
*1:160 or higher
1:320 or higher
1:640 or higher
In with titre will be positive reaction of agglutination in patient with epidemic typhus in droplets test when Mosing?
*1:40 and above
1:80 and above
1:160 or higher
1:320 or higher
1:640 or higher
In the family of the patient with epidemic typhus, were lice in the children. With the help of any of these events could prevent the subsequent spread of the disease?
*Monitoring and complete sanitation of contact in the centre
The use of chemoprophylaxis
The use of antibiotics
Isolation contact
Check-up
When you can stopped etiotropic medications treatment of the patient with epidemic typhus?
Immediately after the normalization of body temperature
After the normalization of the liver and spleen
*After a 2-day normal body temperature
After the disappearance of roseola
Within 10 days after the disappearance of roseola
For contact persons in the centre of the epidemic typhus establish surveillance over:
21 days
25 days
*51 days
72 days
3 months
During the contact person with the Brill‘s disease establish surveillance over:
21 days
*25 days
51 days
72 days
3 months
In the case of head lice carry out sanitation: hair cut, followed by incineration, processing hair. What kind of insecticide products for this use?
0,5 % solution of water emulsions karbofosa
0,5 % metilatsetofos
10 % solution of liquid neutral metilatsetofosa soap
0,5 % water emulsion dikrezilu
*3 % soap RHTSG
When patients after epidemic typhus may go out from clinic?
*After clinical recovery, but not before the 12-day normal temperature
After a full clinical recovery
After clinical recovery, but not before the 12-day period following the lifting of antibiotics
At the 12-day normal body temperature
After clinical recovery, but not earlier than the 9-day normal body temperature
Phage symptom in case of yellow fever is:
Pain in right iliac area
Enanthema on a soft palate
*Replacement of tachicardia on expressed bradicardia
Hemorrhages in a conjunctiva
Yellow hands
Hemograme in the second period of yellow fever:
Leukocytosis
Normal global analysis of blood
*Leukopenia, neutropenia
Leukopenia, neutrophilosis
Leukocytosis, lymphomonocytosis
Whatever complication meets at the yellow fever:
*Liver insufficiency
Kidney insufficiency
Infectious-toxic shock
Myocarditis
Edema of lungs
Unlike leptospirosis in case of yellow fever is absent:
Hemorrhagic syndrome
Kidney insufficiency
Іntoxication syndrome
Міalglic syndrome
*Hepatic insufficiency
For confirmation of yellow fever diagnosis use:
Bacteriological analysis of blood
Bacteriological examination of urine
*Virological hemanalysis
Biochemical blood test
Global analysis of blood
In the initial period of hemorrhagic fever with a kidney syndrome a characteristic sign is:
High temperatures
Pains in gastrocnemius muscles and positive Pasternatsky symptom
*Pains in joints and positive Pasternatsky symptom
Hemorragic syndrome
Dyspepsia phenomena
An initial period at the hemorrhagic fever with a kidneys syndrome lasts:
Few hours
Day
*To three days
Week
Two weeks
Whether there is violation of diuresis at patients with hemorrhagic fever with a kidneys syndrome:
In an initial period
It is not
It is in all periods of disease
*It is in climax period
It is in the period of recovering
General view of patient with the hemorrhagic fever with a kidneys syndrome:
Skinning covers
*Pallor of nasolabial triangle, hyperemia of neck and overhead half of trunk
Hyperemia of person, scleritis, conjunctivitis
Grayish color of person
Icteric color of skin
In the biochemical blood test at patients with the hemorrhagic fever with a kidneys syndrome not characteristically:
High level of urea
Decline of potassium level
*Bilirubinemia
Increasing of kreatinine
Increasing of nitrogen
For confirmation of diagnosis of hemorragic fever with a kidney syndrome use:
Bacteriological method
Virological method
*Reaction of immunofluorescence
Reaction of braking of hemagglutination
Research of blood drop under a microscope
For treatment of patients with the hemorrhagic fever with a kidney syndrome does not use:
Glucocorticoids
Anabolic steroid
Disintoxication facilities
*Dihydration facilities
Antihistaminics
For the initial period of the Congo hemorrhagic fever not characteristically:
Fever
Pains in joints and muscles
Severe pain of head
*Oliguria
Dizziness
At an objective review for the Congo hemorrhagic fever characteristically:
*Mucosal hyperemia of person
Pallor of person
Puffiness of person
Ochrodermia of person
Exanthema on face
The most characteristic symptom in the climax period of the Congo hemorrhagic fever is:
*Hemorrhagic syndrome
Hepatic insufficiency
Dyspepsia phenomena
Sharp kidney insufficiency
Мeningeal syndrome
In the general blood analysis of in case of Congo hemorrhagic fever is not typically:
Leukocytosis
*Leukopenia
Neutropenia
Thrombocytopenia
Increasing of ESR
What rashes in case of haemorrhagic fevers with kidneys syndrome?
Roseola
Maculo-papular
Punctuate
*Petechial
Rashes is not characteristic
What rashes present in case of Congo hemorrhagic fever?
Roseola
Maculo-papular
Punctulate
*Petechial
Rashes not is characteristic
What rashes present in case of Crimea hemorrhagic fever?
Roseola
Maculo-papular
Punctulate
*Petechial
Rashes not is characteristic
How long the rash is present in case of haemorrhagic fever with kidneys syndrome?
*During all feverish period
Before the reconvalescense
Before development of clinical features of kidneys insufficiency
During whole disease
Appears yet in a latent period and disappears in the period of early reconvalescense
A kidney syndrome at haemorrhagic fever with kidneys syndrome shows up usually:
Only laboratory changes
Only on BRIDLES
*By pain in lumbar area, positive Pasternatsky symptom, development of oliguria
By fever, polyuria, dyspepsia
By paradoxical ischuria
What changes in biochemical blood test inherent for haemorrhagic fever with kidneys syndrome?
Increase level of urea and bilirubin
The level of urea and kreatinine falls
The level of kreatinine grows and urea falls
The level of urea grows and kreatinine falls
*The level of urea and kreatinine grows
What changes in haemogram inherent for haemorrhagic fever with kidneys syndrome?
Normochromic anaemia, leucocytosis with atypical mononucleosis, thrombocytopenia enhanceable ESR
erythrocytosis, lymphocytosis,ESR is enhanceable
Normochromic anaemia, leucopenia with neutrophylosis, thrombocytopenia enhanceable ESR
*Hypochromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia enhanceable ESR
Hyperchromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia mionectic ESR
The period of polyuria at haemorrhagic fever with kidneys syndrome is a sign of:
*Recovering
Chronic process
Unfavorable flow of illness
Development of complications
Complete convalescence
In most patients with Congo hemorrhagic fever temperature curve is:
Wunderlich type
Botkin type
Undulating
Intermittent
*Two-humped
With appearance of hemorrhagic syndrome at Congo fever temperature of body always:
Normalize
Grows critically
*Goes down
Does not change
Grows gradually
What changes in haemogram inherent Congo hemorrhagic fever?
Normochomic anaemia, leucocytosis mononuclear
Erythrocytosis, lymphocytosis
*Hypochromic anemia, erythrofilosis
Hypochromic anemia, neutrofilosis
Hyperchromic anemia, neutrofilosis
What is typical for the Lassa hemorrhagic fever:
Effect of cardiovascular system
Development of acute hepatic insufficiency
Hundred-per-cent lethality
*Defeat of breathing organs
Development of paresis and paralysis
Confirm diagnosis of haemorrhagic fever with kidneys syndrome by a way of:
Only virological methods
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
*Virologic and serum methods
Confirm the diagnosis of Lassa hemorrhagic fever by a way of:
Only virological methods
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
*Virologic and serum methods
Confirm the diagnosis of Congo hemorrhagic fever by a way of:
Only virological methods
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
*Virologic and serum methods
Confirm the diagnosis of Ebola fever by a way of:
Growth of viruses on chicken embryons
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
*Selection of virus on the Vero culture
Confirm the diagnosis of Omsk fever by a way of:
Growth of virus on chicken embryons
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
*Selection of virus on the Vero culture
Confirm the diagnosis of Marburg fever by a way of:
Growth on chicken embryos
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiologys information
*Selection of virus on the Vero culture
What etiothropic means use at treatment of haemorrhagic fever with kidneys syndrome:
Benzylpenicillin
Dopamine
*Virolex
Dexamethazone
Etamsylatum
What etiothropic means use at treatment of patients with Lassa fever:
Benzylpenicillin
Dopamine
*Ribavirin
Dexamethazole
Etamsylatum
What etiothropic means use at treatment of patients with Omsk fever:
Benzylpenicillin
Dopamine
*Ribavirin
Dexamethazone
Etamsylatum
What etiothropic means use at treatment of patients with Marburg fever:
Benzypenicillin
Dopamine
*Ribavirin
Dexamethazone
Etamsylatum
What etiothropic means use at treatment of patients with Congo fever:
Benzylpenicillin
Dopamine
*Ribavirin
Dexamethazone
Etamsylatum
What etiothropic means use at treatment of patients with Ebola fever:
Benzylpenicillin
Dopamine
*Virolex
Dexamethazone
Etamsylatum
What etiothropic means use at treatment of patients with Crimea fever:
Benzylpenicillin
Dopamine
*Ribavirin
Dexamethazone
Etamsylatum
Specific prevention of hemorrhagic fevers:
The live vaccine
Killed vaccine
The specific immunoglobulin
*Do not developed
Polivalent vaccine
Who is the source of the causal agent in the Crimean-Congo haemorrhagic fever?
Rodents, cattle, birds
The source of infection of Omsk‘s hemorrhagic fever are muskrat, water rats and other rodents. Who are the carriers?
Bee and flea
*Pliers and flea
Mosquitoes
Fly
Pliers and mosquitoes
Specific prevention of Crimean-Congo haemorrhagic fever are:
*Vaccine and human immunoglobulin
Serum
Serum and human immunoglobulin
Do not developed
Antibacterial drugs
Those who have been in contact with sick haemorrhagic fevers, as well as those who had bite by the ticks in endemic areas are introducing:
Specific vaccine
The specific immunoglobulin in doses of 10-15 ml vaccine
The specific immunoglobulin in doses of 10-15 ml
*The specific immunoglobulin in doses 5-7,5 ml
Nothing
Hemograme in the second period of yellow fever:
Leukocytosis
Normal global analysis of blood
*Leukopenia, neutropenia
Leukopenia, neutrophilosis
Leukocytosis, lymphomonocytosis
What ever complication meets at the yellow fever:
*Liver insufficiency
Kidney insufficiency
Infectious-toxic shock
Myocarditis
Edema of lungs
In the initial period of hemorrhagic fever with a kidney syndrome a characteristic sign is:
High temperatures
Pains in gastrocnemius muscles and positive Pasternatsky symptom
*Pains in the joints and positive Pasternatsky symptom
Hemorragic syndrome
Dyspepsia phenomena
For treatment of patients with the hemorrhagic fever with a kidney syndrome do not use:
Glucocorticoids
Anabolic steroid
Disintoxication facilities
*Dihydration facilities
Antihistamins
For the initial period of the Congo hemorrhagic fever not characteristically:
Fever
Pains in joints and muscles
Severe pain of head
*Oliguria
Dizziness
The most characteristic symptom in the climax period of the Congo hemorrhagic fever is:
*Hemorrhagic syndrome
Hepatic insufficiency
Dyspepsia phenomena
Sharp kidney insufficiency
Мeningeal syndrome
In the global analysis of blood in case of Congo hemorrhagic fever not characteristically:
Leukocytosis
*Leukopenia
Neutropenia
Thrombocytopenia
Increasing of ESR
What rashes in case of haemorrhagic fevers with kidneys syndrome?
Roseola
Maculo-papular
Punctuate
*Petechial
Rashes is not characteristic
What does change in biochemical blood test in the patient with haemorrhagic fever with kidneys syndrome?
Increase level of urea and bilirubin
The level of urea and kreatinine falls
The level of kreatinine grows and urea falls
The level of urea grows and kreatinine falls
*The level of urea and kreatinine grows
What does change in haemogram in the patient with haemorrhagic fever with kidneys syndrome?
Normochromic anaemia, leucocytosis with atypical mononucleosis, thrombocytopenia enhanceable ESR
erythrocytosis, lymphocytosis,ESR is enhanceable
Normochromic anaemia, leucopenia with neutrophylosis, thrombocytopenia enhanceable ESR
*Hypochromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia enhanceable ESR
Hyperchromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia mionectic ESR
The temperature curve in most patients with Congo hemorrhagic fever is:
Wunderlich type
Botkin type
Undulating
Intermittent
*Two-humped
What is typical for the Lassa hemorrhagic fever:
Effect of cardiovascular system
Development of acute hepatic insufficiency
Hundred-per-cent lethality
*Defeat of breathing organs
Development of paresis and paralysis
What etiothropic means use at treatment of patients with Lassa fever:
Benzylpenicillin
Dopamine
*Ribavirin
Dexamethazole
Etamsylatum
What etiothropic means use at treatment of haemorrhagic fever with kidneys syndrome:
Benzylpenicillin
Dopamine
*Virolex
Dexamethazone
Etamsylatum
Specific prevention of hemorrhagic fevers:
The live vaccine
Killed vaccine
The specific immunoglobulin
*Do not developed
Polivalent vaccine
Who is the source of the causal agent in the Crimean-Congo haemorrhagic fever?
Rodents, cattle, birds
Phage symptom in case of yellow fever is:
Pain in right iliac area
Enanthema on a soft palate
*Replacement of tachicardia on expressed bradicardia
Hemorrhages in a conjunctiva
Yellow hands
General view of patient with the hemorrhagic fever with a kidneys syndrome:
Skinning covers
*Pallor of nasolabial triangle, hyperemia of neck and overhead half of trunk
Hyperemia of person, scleritis, conjunctivitis
Grayish color of person
Icteric color of skin
For confirmation of diagnosis of hemorrhagic fever with a kidney syndrome use:
Bacteriological method
Virological method
*Reaction of immunofluorescence
Reaction of braking of hemagglutination
Research of blood drop under a microscope
For treatment of patients with the hemorrhagic fever with a kidney syndrome does not use:
Corticosteroids
Anabolic steroids
Disintoxication facilities
*Dehydration facilities
Antihistaminics
For the initial period of the Congo hemorrhagic fever not characteristic:
Fever
Pains in joints and muscles
Severe pain of head
*Oliguria
Dizziness
At an objective examination for the Congo hemorrhagic fever character:
*Mucosal hyperemia of person
Pallor of person
Puffiness of person
Ochrodermia of person
Exanthema on face
The most characteristic symptom in the climax period of the Congo hemorrhagic fever is:
*Hemorrhagic syndrome
Hepatic insufficiency
Dyspepsia phenomena
Sharp kidney insufficiency
Мeningeal syndrome
In the global analysis of blood in case of Congo hemorrhagic fever not characteristic:
Leukocytosis
*Leukopenia
Neutropenia
Thrombocytopenia
Increasing of ESR
What rashes in case of haemorrhagic fevers with kidneys syndrome?
Roseola
Maculo-papular
Punctuate
*Petechial
Rashes is not characteristic
What rashes present in case of Congo hemorrhagic fever?
Roseola
Maculo-papular
Punctulate
*Petechial
Rashes not is characteristic
What rashes present in case of Crimea hemorrhagic fever?
Roseola
Maculo-papular
Punctulate
*Petechial
Rashes not is characteristic
How long the rash is present in case of hemorrhagic fever with kidneys syndrome?
*During all feverish period
Before the convalescence
Before development of clinical features of kidneys insufficiency
During whole disease
Appears yet in a latent period and disappears in the period of early reconvalescense
What changes in biochemical blood test inherent for hemorrhagic fever with kidneys syndrome?
Increase level of urea and bilirubin
The level of urea and kreatinine falls
The level of kreatinine grows and urea falls
The level of urea grows and kreatinine falls
*The level of urea and kreatinine increase
What changes in blood analysis inherent for hemorrhagic fever with kidneys syndrome?
Normochromic anaemia, leucocytosis with atypical mononucleosis, thrombocytopenia increased ESR
erythrocytosis, lymphocytosis,ESR is increased
Normochromic anaemia, leucopenia with neutrophylosis, thrombocytopenia increased ESR
*Hypochromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia increased ESR
Hyperchromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia mionectic ESR
The period of polyuria at haemorrhagic fever with kidneys syndrome is a sign of:
*Recovering
Chronic process
Unfavorable flow of illness
Development of complications
Complete convalescence
With appearance of hemorrhagic syndrome at Congo fever temperature of body always:
Normal
Grows critically
*Goes down
Does not change
Grows gradually
What changes in blood analysis inherent at Congo hemorrhagic fever?
Normochromic anaemia, leucocytosis mononuclear
Erythrocytosis, lymphocytosis
*Hypochromic anemia, erythrophilosis
Hypochromic anemia, neutrophilosis
Hyperchromic anemia, neutrophilosis
What is typical for the Lassa hemorrhagic fever:
Effect of cardiovascular system
Development of acute hepatic insufficiency
Hundred-per cent lethality
*Defeat of breathing organs
Development of paresis and paralysis
Confirm the diagnosis of hemorrhagic fever with kidneys syndrome by a way of:
Only virological methods
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
*Virologic and serum methods
Confirm the diagnosis of Lassa hemorrhagic fever by a way of:
Only virological methods
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
*Virologic and serum methods
Confirm the diagnosis of Congo hemorrhagic fever by a way of:
Only virological methods
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
*Virologic and serum methods
Confirm the diagnosis of Ebola fever by a way of:
Growth of viruses on chicken embryos
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
*Selection of virus on the Vero culture
Confirm the diagnosis of Omsk fever by a way of:
Growth of virus on chicken embryons
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
*Selection of virus on the Vero culture
The measures of urgent prophylaxis of plague.
Administration of human immunoglobulin
Chlorochin (delagil) 0,25 g 2 times in week
*6-day’s prophylaxis with streptomycin or tetracycline
In first 5 days intake antibiotics of penicillin or tetracycline origin
Іnterferon
The rules of hospitalization of patients with plague:
To separate ward
To ward for respiratory infections
*To ward cubicle
Patient’s are not hospitalized
To ward for intestinal infections
Patient T., drives in a country unhappy on a plague. Conduct measures on a specific prophylaxis.
Human immunoglobulin
Interferon
Bacteriophage
*Dry living vaccine
Live measles vaccine
Preparations for urgent prophylaxis of plague:
Injection of human immunoglobulin
*Streptomycin or tetracycline
Human immunoglobulin
Dry living vaccine or tetracycline generations.
Interferon
Y. pestis is transmitted more frequently by:
*Flea
Water
Air
Food storage
Tick
The duration of incubation period of plague is:
3 to 8 days;
2 to 12 days;
2 to 10 days;
1 to 8 days.
*2 to 6 days;
What is the main feature of septicemic plague?
*Massive bacteriemia
Headache
Pain in the abdominal
Throat ache
Bleeding
What drug is first step of choice for the treatment of plague?
Amoxicillin
*Streptomycin
Penicillin
Biseptol
5-NOK
What is the treatment of patients with a plague:
Immediately after hospitalization
*Immediately after hospitalization, carrying out only material for research
After raising of final diagnosis
After laboratory and instrumental diagnostics
All answers are faithful
How many pandemics of plague was in history of mankind?
*Three
Four
One
Two
Five
Especially dangerous for surroundings are patients with:
Skin form of plague
*Pulmonary form of plague
Skin-bubonic form of plague
Bubonic form of plague
Septic form of plague
Risk group of plague infection the most frequent is:
Doctors
*Hunters
Alcoholic
Drug users
Prostitutes
Who is the reservoir of causative agent of plague in nature?
Birds
Insects
Fresh-water fish
*Rodents
Cattle
What is the susceptibility of human to plague?
Non susceptible
50 %
*Almost 100 %
10 %
70 %
The causative agent of plague is:
*Yersinia pestis
Yersinia enterocolitica
Yersinia pseudotuberculosis
Bac. anthracis
Pseudomonas mallei
The duration of incubation period at plague is:
2-6 h
*2-6 d
10-15 d
17-21 d
1-6 w
To the localized forms of plague belong:
Secondary-septic
Primary-septic
*Skin
Primary-pulmonary
Intestinal
To the localized forms of plague belong:
Intestinal
Primary-septic
Secondary-septic
Primary-pulmonary
*Skin-bubonic
To the localized forms of plague belong:
Intestinal
Primary-septic
Secondary-septic
Primary-pulmonary
*Bubonic
To the internal-disseminated forms of plague belong:
*Primary-septic
Bubonic
Secondary-pulmonary
Primary-pulmonary
Intestinal
Choose the specific treatment of tetanus.
Antibiotics
*Serum
Anticonvulsant medicine
Cardiac preparations
Desintoxication therapy
The exciter of tetanus is:
*Clostridia
Escherichia
Candida albicans
Neisseria
Gonococcus
For the exciter of tetanus characteristic such properties, except:
Formation of exotoxins
Ability to propagate in anaerobic conditions
Formation of spores
*Formation of gametes
The best terms of tetanus exciter cultivation:
*Anaerobic conditions
Oxygen supply
Presence of animal albumen in nutritive medium
Low temperature
1 % peptone water
Vegetative form of exciter of tetanus is destroyed in such terms, except for:
At a temperature of 100 °C
*At room temperature
Under action of carbolic acid
Under the action of oxygen
Who is the source of tetanus?
Sick person
Rodents
*Soil
Insects
Cattle
The spores of tetanus are saved:
After boiling during 1 hour
Under act of dry air at the temperature of 115 degrees C
*In soil during many years
In 1 % solution of formalin during 6 hours
Tetanus toxin consists of all units among the listed below, except:
Tetanospasmin
Tetanolysin
Exotoxin
Low-molecular fraction
*Enterotoxin
What is the receptivity of population to the tetanus?
0 %
50 %
*Almost 100 %
10 %
Causing of tetanus are:
*C. tetani
E. coli
Candida
Epstein-Barr virus
Hemolytic streptococcus group A
Duration of the latent period in case of tetanus:
1-6 hours
1-4 days
5-14 days
*1-6 weeks.
1-6 months
How long does the incubation period of tetanus last?
1-5 days
5-10 days
3-5 days
*5-14 days
15-20 days
Tetanus might appear in case of:
*Trauma
Mosquito bite
Usage of stranger clothes
Contact with the sick people
Drink the water with poor quality
Tetanus might appear in case of:
*Dog bite
Mosquito bite
Usage of stranger clothes
Contact with the sick people
Drink the water with poor quality
What is the medical tactic development of the severe tetanus after criminal abortion?
Anticonvulsant preparations
Revision of the uterus cavity
Analgesic therapy
Antibiotics
*All answers are correct
Choose dose of the specific treatment for patients with tetanus.
600 units/kg of antytetanus serum
900 units/kg of antytetanus Ig
500 units/kg of antytetanus Ig
900 units/kg of antytetanus serum
*500 units/kg of antytetanus serum
What is the first aid preparation for the patient with tetanus?
Glucocorticoids
Analgetics
*Anticonvulsant medicine
Surgical treatment of the wound
Oxygen therapy
Among the listed below choose the complication of the tetanus, which is not early:
Tracheobronchitis
*Contracture of muscles and joints
Asphyxia
Myocarditis
Pneumonia
Among the listed below choose the complication of the tetanus, which is not early:
Tracheobronchitis
*Compressive deformation of the spine
Asphyxia
Myocarditis
Pneumonia
Specify the measures of urgent prophylaxis of anthrax.
Anti-anthrax immunoglobulin
*Penicillinum or tetracyclinum during 5 days
Vaccination
Medical supervision
Biseptolum 5 days
What specific test is used for anthrax diagnostic?
Compliment fixation test
Indirect hemaglutination test
*Coetaneous test with antraxin
Hemaglutination test
RIFA with anthrax antigen
What anthrax prophylactic measures are entertained by farm workers?
Vitamin therapy
Immunization by inactivated vaccine
Formulated vaccine
*Immunization by live vaccine
Antibiotic therapy
The etiological factor of anthrax is:
Salmonella thyphi
Erysipelothrix rhysiopothiac
*Bacillus anthracis
Rickettsiosis sibirica
Toxocara canis
The source of infection of anthrax is more frequent than all:
People
Birds
*Home animals
Rodents
Fly
Mechanism of transmission of anthrax are:
Contact
Alimentary
Air-droplets’
Transmissiv
*All above it
What organ damaged more frequent than all in patients with anthrax?
*Skin
Lights
Gastrointestinal tract
Lymphatic system
Nervous system
The basic clinical display of a skin form of anthrax is:
Hyperemic of skins
Vesiculs
*Ulcer
Phlegmon
Abscess
For anthrax most characteristically:
Change of stool
Icterus of skin
Catarrhal phenomena
Meningeal phenomena
*Change of skin
For a skin form of anthrax the most characteristically:
Hyperemia
Painful carbuncle
*Not painful carbuncle
Painful noodles
Vesicles and bulls
For anthrax carbuncle the most characteristically:
Ulcer with a festering bottom, roller on periphery and insignificant area of edema
Ulcer with hyperemia on periphery without an edema
*Ulcer with a black scab, black color, second vesicles and area of edema around of ulcer
Ulcer with a festering bottom, roller on periphery, second vesicles and area of edema
Ulcer with serosis-hemorrhagic exudates, painful, with the area of edema around of ulcer
Symptom of Stefansky – it is:
Enantema on a soft palate
Enantema on a conjunctiva
Shaking of tongue at an attempt to put out a tongue
*Shaking of edema like to jelly at pattering a hammer in the area of edema
Painful of stomach in a right iliac area
For the pulmonary form of anthrax characteristically:
*Foamy sputum with blood
Glassy sputum with blood
Foamy sputum without blood
Foamy green sputum
Like to «ferruginous» sputum
With what diseases it is necessary to differentiate anthrax:
Leptospirozis
Typhoid fever
Dermatitis
*Carbuncle
Meningococcal infection
What material is necessary take for diagnosis of anthrax:
Spinal liquid
Urine
Saliva
*Content of carbuncle
Nose swab
The diagnostic reaction of anthrax is:
Rayt‘s reaction
Vidal‘s reaction
*Reaction of term precipitation of Askoly
Paul-Bunnel‘s reaction
Reaction of agglutination-lysis
The diagnostic endermic reaction of anthrax take:
*Antraksin
Dizenterin
Ornitin
Malein
Brucellin
For treatment of anthrax is:
Sulfanilamids
Nitrofurans
Hormones
Antiviral facilities
*Antibiotics
It is necessary to appoint for successful treatment of anthrax:
*Antyanthrax immunoglobulin and penicillin
Antyanthrax immunoglobulin and prednizolon
Antyanthrax immunoglobulin and vyrolex
Antyanthrax immunoglobulin and vermox
To what group of infections does the rabies belong?
*Zoonosis
Anthroponosis
Capronosis
Anthropozoonosis
Caprozoonosis
The basic reservoir of rhabdovirus is:
Pisces
Reptiles
Birds
Weed-eaters
*Carnivores
Rhabdovirus from an organism of the patient or animal is revealed to the flow:
Last 20 days of latent period and during all the illness
*Last 7-10 days of latent period and during all the illness
Last 7-10 days of latent period
Last 7-10 days of latent period and at the beginning of illness
During all the illness
You may be infected a rhabdovirus in case of:
*Bite +salivation to the skin by an animal
Infected meal
Infected water
Contact with the infected air
Bite with the infected insect
What is the sensitivity to the rabies?
45 %
25 %
85 %
*100 %
10 %
What is the mechanism of transmission of rabies?
Transmissive
Fecally-oral
Air drop
*Wound
Domestic contact
What is the main mechanism of transmission of rabies?
*Airborne
Alimentary
Contact
Transmisiv
Vertical
Mechanism of transmission of rabies are often:
*Air
Contact
Transmissiv
Fecal-oral
Transplacental
What is the entrance for the rabies?
*Damaged skin and mucous tissues
Respiratory tracts
Family ways
Gastrointestinal tract
Blood
Causing of rabies are:
C. tetani
E. coli
Candida
Epstein-Barr virus
*Rabdovirus
Duration of the latent period in case of tetanus:
1-6 hours
7-14 days
*7 days – 1 ear
1-6 weeks.
1-6 months
What periods of rabies do you know?
Incubation, depressions, excitation
*Incubation, depressions, excitation, paralytic
Depression, excitation, paralytic
Incubation, excitation, paralytic
Incubation, depressions, paralytic
For rabies the source of infection can be a dog bite in all the cases, except for:
With rabies
Suspicion on rabies
Vagrant
*Month prior to the disease
Last 10 days before the disease
In rabies the source of the virus can be:
Wild animals
Home animals
Bats
Rodents
*All the answers are correct
What is the duration of the prodromal period for the rabies?
*1-3 days
Up to 1 day
4-7 days
3-4 days
3-5 days
The first symptom of prodromal period of rabies is:
Cough
Nausea
Vomiting
Diarrhea
*Slight swelling and erethema of the scar
The first symptom of prodromal period of rabies is:
Cough
Nausea
Vomiting
*Neurological pains in motion nervous barrels, the nearest to the place of bite
Diarrhea
The first symptom of prodromal period of rabies is:
Cough
Nausea
Vomiting
*Apathy and depression
Diarrhea
Most characteristic symptoms of the rabies are:
*Paroxysm of hydrophobia
Apathy and depression
Neuralgic pains on motion nervous barrels, the nearest to the place of bite
Dyspepsia disorders
Catarrhal phenomen
Most characteristic symptoms of the rabies are:
*Paroxysm of hydrophobia
Paroxysm of aerophobia
Paroxysm of fotophobia
Paroxysm of akuzophobia
Paroxysm of soilphobia
What is the duration of excitation period of rabies?
7-10 days
24 hours
*2-3 days, sometimes to 6 days
Not more than 2 days
Up to 6 hours
What temperature of the body is typical for the paralytic period?
*Hyperpyrexia
Hypothermia
High
Normal
Subfebril
Who of the listed below persons must take the conditional course of inoculations against rabies?
*A teenager bitten by a dog which is on a leash, not instilled
Man bitten by a fox which perished
A child, scratched by a squirrel which disappeared in-field
A woman, bitten by a cat ill with rabies
Man, who had a meal of undercooked of animal with rabies
When from the beginning of vaccination an antibodies to the rhabdovirus appear?
In a week
*In 2 weeks
In a month
After half of year
Don’t produced
For what infectious pathology is characterized Babesh-Negri‘ bodies?
Poisoning mushrooms
Meningo-encefalit
Poliomyelitis
*Rabies
Tetanus
Dog bite man on foot. What kind of specific prophylaxis should be conducted for this patient?
Human rabies immunoglobulin 6 doses of antirabies vaccine
12 doses of antirabies vaccine
Human rabies immunoglobulin and 6 doses of antirabies vaccine
Human rabies immunoglobulin and 21 dose of antirabies vaccine
*6 doses of antirabies vaccine
What kind of specific prophylaxis should be conducted for patient with bitten foot?
Human rabies immunoglobulin
*Vaccine antirabies
Vaccine antirabies and rabies immunoglobulin
Human rabies immunoglobulin and serum
Human rabies serum
How are the little bodies named (in patients with rabies)?
Lorin-Epshteyn
Blyumberg
Murson
Rozenberg
*Babesh-Negri
Where are the little bodies of Babesh-Negri?
In lungs
In liver
In a spinal cord
*In neurons
In blood
Rabies might appear in case of:
*Dog bite
Mosquito bite
Usage of stranger clothes
Contact with the sick people
Drink the water with poor quality
Select the correct rabies vaccination scheme:
0, 3, 6, 14, 20, 90 days
0, 4, 7, 14, 50, 90 days
0, 5, 8, 14, 30, 100 days
*0, 3, 7, 14, 30, 90 days
0, 1, 5, 13, 60, 90 days
The exciter of tetanus is:
*Clostridia
Escherichia
Candida albicans
Neisseria
Gonococcus
For the exciter of tetanus characteristic such properties, except:
Formation of exotoxins
Ability to propagate in anaerobic conditions
Formation of spores
*Formation of gametes
Gram positive
The best terms of tetanus exciter cultivation:
*Anaerobic conditions
Oxygen supply
Presence of animal albumen in nutritive medium
Low temperature
1 % peptone water
Vegetative form of exciter of tetanus is destroyed in such terms, except for:
At a temperature of 100 °C
*At room temperature
Under action of carbolic acid
Under the action of oxygen
Under action of antibiotics
Who is the source of tetanus?
Sick person
Rodents
*Soil
Bacteriocarrier
Sick person and bacteriocarrier
The spores of tetanus are saved:
After boiling during 1 hour
Under act of dry air at the temperature of 115 degrees C
*In soil during many years
In 1 % solution of formalin during 6 hours
All answers are correct
Tetanus toxin consists of all units among the listed below, except:
Tetanospasmin
Tetanolysin
Exotoxin
Low-molecular fraction
*Enterotoxin
Mechanism of transmission in case of tetanus are:
Intra muscular conduction
*Contact
Insect conduction
Faecally-oral
Vertical conduction
What is the receptivity of population to the tetanus?
0 %
50 %
*Almost 100 %
10 %
70 %
Causing of tetanus are:
*C. tetani
E. coli
Candida
Epstein-Barr virus
Hemolytic streptococcus group A
Duration of the latent period in case of tetanus:
1-6 hours
1-4 days
*5-14 days
1-6 weeks.
1-6 months
How long does the incubation period of tetanus last?
1-5 days
5-10 days
3-5 days
*5-14 days
15-20 days
Tetanus might appear in case of:
*Trauma
Mosquito bite
Usage of stranger clothes
Contact with the sick people
Drink the water with poor quality
Tetanus might appear in case of:
*Dog bite
Mosquito bite
Usage of stranger clothes
Contact with the sick people
Drink the water with poor quality
What is the medical tactic development of the severe tetanus after criminal abortion?
Anticonvulsant preparations
Revision of the uterus cavity
Analgesic therapy
Antibiotics
*All answers are correct
What measures should be taken in relation to contact persons in case of tetanus?
Vaccination
Isolation of contacts
Chemoprophylaxis
Laboratory inspection
*They need no measures
Among the listed below what preparations are not etiological for tetanus?
AC-anatoxin
Medical horse serum
Human immunoprotein
*Anticonvulsant preparations
Penicillin
Choose dose of the specific treatment for patients with tetanus.
500 international units of antytetanus Ig
500 international units of antytetanus serum
*900 international units of antytetanus Ig
900 international units of antytetanus serum
900 units/kg of antytetanus serum
Which early complications occurs in tetanus?
Tracheobronchitis
Asphyxia
Myocarditis
Pneumonia
*All the above
Which late complications occurs in tetanus?
Contracture of muscles and joints
Compressive deformation of the spine
Asthenic syndrome
Chronic heterospecific diseases of lungs
*All the above
What is the duration of outpatient supervision for patients, recovered of tetanus?
*2 years
3 months
1 month
For the decreed groups of population for life time
There is no such supervision at all
Urgent immuno prophylactic of tetanus in the case of trauma should be conducted in such period:
25 days from the moment of trauma
30 days from the moment of trauma
In the first 10 days from the moment of trauma
*At once after the trauma
Not mentioned
Among the listed below people who should receive an immediate prophylactic of the tetanus in form of AC-anatoxin and AC IP injections after trauma?
Man of 40 years, in anamnesis with 1 inoculation one year ago
Pregnant woman of 30 years, in the second half of pregnancy
Child, 7 months, instilled according to a calendar
*Retire man of 57 years, who is not instilled
Child of 6 years, instilled according to a calendar
In case of tetanus the epidemiological measures are directed on:
Elimination of the source of tetanus
Treatment of the source of tetanus
*Specific prophylaxis
Medicines prophylactics
Nothing should be performed
At what infectious disease does conduct the spasm almost always commences in the muscles of the neck and jaw. causing closure of the jaws?
Poisoning mushrooms
Meningoencefalitis
Poliomyelitis
Rabies
*Tetanus
For what disease is characterized this symptom (the generalized spasm of soft muscles, flexion of the arms and extension of the legs)?
Poliomyelitis
Brucellosis
Pseudo tuberculosis
*Tetanus
Hydrophobia
For what disease is characterized opistotonus?
Poliomyelitis
Brucellosis
Pseudo tuberculosis
*Tetanus
Hydrophobia
For what disease is characterized rizos sardonicus?
Poliomyelitis
Brucellosis
Pseudo tuberculosis
*Tetanus
Hydrophobia
For what disease is characterized lockjaw?
Poliomyelitis
Brucellosis
Pseudo tuberculosis
*Tetanus
Hydrophobia
what is name of symptom characterized to the patient with tetanus?
*Lorin-Epshteyn
Blyumberg
Murson
Rozenberg
Name the HIV infection high risk groups :
Homo- and bisexual, prostitutes and other persons who conduct disorderly sexual life
Drug addicts who enter drugs parenterally
Only recipeint of blood, its preparations, sperm and organs
Only patients with venereal diseases and parenteral viral hepatitis and from the HIV infected mothers
*All the above
What is the most effective methods of HIV prevention:
Vaccination and immunoprotein
Chemoprophylactic
solation of patients
*Safe sex and prevention of drug addiction
Disinfection
How many types of HIV are known?
One
*Two
Three
Four
Five
When HIV/AIDS agent was discovered?
1981
1982
*1983
2002
2003
Name the main specific method of HIV diagnosis?
RPGA
PLR
*IFA and ELISA
Bioassey
RIA
Name the most dangerous parenteral way of infection of HIV/AIDS?
*Infusion of donor blood and its preparations
Transplantation of organs
Injections of medications
Diagnostic manipulations
Cosmetic manipulations
Name the source of exciter of HIV infection/AIDS?
*Human
Animals
Poultries
Amphibious
Fish
What is the basic way of transmission of HIV infection:
Air-born
Alimentary
*Parententeral
Transmissive
Water
What humans cell of body is a target of HIV?
Erythrocytes
Neutrophyl leucocytes
Monocytes
T-killer-lymphocytes
*T-cell helpers
What cellular receptors of human attract HIV?
*CD4
CD8
CD95
CD40
CD3
What clinical features of sarcoma Kaposhi in patients with AIDS?
Strike the persons of young and middle age
Primary elements appear on a head and trunk
Elements with necrosis and ulceration
Metastasis in internal organs and high lethality
*All the above
What family of viruses the exciter of HIV/AIDS belong to?
Orto- and paramyxovirus
Rabdovirus
*Retrovirus
Herpesvirus
Reovirus
What group of infectious diseases an exciter of HIV infection/AIDS belong to according to L. Gromashevsky classification?
Intestinal infection
Infections of respiratory tract
Blood infection
*Infection of external covers
Transmissive
What group of infectious diseases the exciter of HIV/AIDS belong to?
*Antroponozis
Zoonosis
Sapronosis
Saprozoonosis
Zooantroponosis
What sexual contact are the most dangerous in relation to an infection with HIV?
Vaginal
*Anal
Oral
Lesbian
Artificial impregnation
Intravenous introduction of drugs, transfusion of blood or blood products, because of the risk of transmitting BICH should be conducted, except for:
For health reasons
By decision of the consilium
Agreement of the patient or his relatives
A careful selection of donors
* There are no restrictions
What dose of antiretroviral drugs for HIV prevention after contact of person with blood and body fluids?
600-800 mg / day
700-800 mg / day
* 800-1000 mg / day
. 1000-1100 mg / day
. 1100-1200 mg / day
How long held antiretroviral prophylaxis regimen after contact with blood and other body fluids?
1 Week
B. 2 weeks
* 1 month
3 months
6 months
HIV-infected person is dangerous for others:
Only in symptomatic period
Only in the stage of acute infection
Only in the stage of asymptomatic infection
Only in the terminal stage
. * During Lifetime
Select an indication for post-exposure prophylaxis of HIV:
* Medical accident with HIV-infected patient
HIV-infected pregnant
HIV infection
AIDS
All the above listed
Select an indication for post-exposure prophylaxis of HIV:
AIDS
HIV-infected pregnant
HIV infection
* Childbirth by HIV-infected mother
All the above listed
Select an indication for post-exposure prophylaxis of HIV:
Medical accident with HIV-infected patient
Delivery a baby by HIV-infected mother
Rape
Transfused
* All the above listed
Select an indication for post-exposure prophylaxis of HIV:
AIDS
HIV-infected pregnant
HIV infection
* Rape
All the above listed
Select an indication for post-exposure prophylaxis of HIV:
. * Medical accident with HIV-infected patients
HIV-infected pregnant
HIV infection
AIDS
All the above listed
Select an indication for post-exposure prophylaxis of HIV:
AIDS
HIV-infected pregnant
HIV infection
* Childbirth from HIV-infected mother
All the above listed
Select an HAART drugs used in the treatment of patients with AIDS except:
. * Selection of the drug according to the antibiogram
. Nucleoside reverse transcriptase inhibitors HIV
. NNRTIs
. protease Inhibitors
. integrase inhibitors
Terms of prophylaxis with antiretroviral drugs after exposure with blood and body fluids?
* No later than 72 hours
During a week
If the infection is confirmed
. Dont perform
. To seropositive persons
If the specific markers HIV identified the victim as a result of an accident during the medical examination in the first 5 days after the accident, it means:
Occupational exposure
The patient is in the incubation period of HIV infection
* The patient was HIV-positive before the accident
Seroconversion after crash
Does not mean anything
Increased risk of infection HIV, except:
Gays
Addicts
. Patients after transplantation of organs and tissues
Paramedics
* Donors of blood and organs
Increased risk of HIV infection , except:
Homosexuals
Addicts
. sex partners of HIV infected
paramedics
* Persons living in the same apartment
Increased risk of infection HIV, except:
Persons entering into promiscuity
Addicts
Hemophiliacs
Children from HIV-infected mothers
* Donors of blood and organs
Increased risk of infection HIV, except:
Homosexuals
Addicts
sex partners of HIV infected
paramedics
* Persons living in the same apartment
Increased risk of infection BICH:
Homosexuals
Addicts
Hemophiliacs
Children from HIV iinfected mothers
* all the above listed
For the diagnosis of Kaposi's sarcoma in AIDS should be:
The presence of tumor formation or brown cherry color of the skin or mucous membranes
Affects young men
Frequent ulceration, bleeding
histologic confirmation
* all the above listed
To perform highly active antiretroviral therapy, select the correct combination of drugs:
2 NRTIs 2 SP
* 2 NRTIs + 1 NNRTI
1 + 2 NRTIs, NNRTIs
1 NRTI + 1 PI 2 NNRTI
1 2 NRTI PI
To perform highly active antiretroviral therapy, select the combination of drugs:
1 2 NRTI PI
1 + 2 NRTIs, NNRTIs
* 3 NRTIs
1 NRTI + 1 PI 2 NNRTI
2 NNRTI 2 SP
For post-exposure prophylaxis of HIV infection select the correct combination of drugs:
1 2 NRTI PI
1 + 2 NRTIs, NNRTIs
* 2 NRTIs + 1 NNRTI
1 NRTI + 1 PI 2 NNRTI
2 NNRTI 2 SP
For post-exposure prophylaxis of HIV iinfection select the correct combination of drugs:
1 2 NRTI PI
1 + 2 NRTIs, NNRTIs
* 2 NRTIs + 1 NNRTI
1 NRTI + 1 PI 2 NNRTI
2 NNRTI 2 SP
What is characteristic of Kaposhi's sarcoma in AIDS except:
common malignancy
Affects young men
frequent bleeding
* Localization only on legs and feet
frequently ulcerate
What is characteristic of Kaposhi's sarcoma in AIDS except:
Affects young people
* It affects only older men
widespread localization
malignancy
frequently ulcerate
What is characteristic of Kaposhi's sarcoma in AIDS , except:
* The current benign
Affects young men
frequent bleeding
Visceral
frequently ulcerate
HIV contamination may be considered professional if specific markers of the virus identified the victim as a result of an accident during the medical examination at such times, except:
* The first 5 days after the accident
1 month
3 months
after 6 months
1 year
The most important clinical criteria of AIDS should include:
Significant weight loss (10 %) if it continues over a month
Duration of fever, if it continues over a month
Pillar, diarrhea, if it lasts more than a month
Lymphadenopathy than 3 months
*All the above
The most important clinical criteria of AIDS must include the following, except:
Significant weight loss (10 %) if it continues over a month
Duration of fever, if it continues over a month
Pillar, diarrhea, if it lasts more than a month
* Rush on the skin, if it lasts more than a month
Lymphadenopathy longer than 3 months.
. The most important clinical criteria of AIDS should include:
Significant weight loss (10 %) if it continues over a month
Duration of fever, if it continues over a month
Pillar, diarrhea, if it lasts more than a month
* Lymphadenopathy than 3 months
All the above
How to decide the child's immunizations in case of AIDS ?
Vaccination against diphtheria and tetanus
. Vaccination against polio
Vaccination against tuberculosis
Vaccination against polio and tuberculosis
* Vaccination do not conduct
How to decide on the child's immunizations with established HIV infection?
* Vaccination against diphtheria and tetanus
Vaccination against polio
Vaccination against tuberculosis
Vaccination against polio and tuberculosis
Vaccination not conduct
What antiretroviral drugs may prescribed for HIV-infected pregnant women in order to reduce the risk of infection of the fetus?
Zidovudine and lopinavir
* Zidovudine and Viramune
. Timazid and Retrovir
AZT and Retrovir
. Zidovudine, Retrovir and timazid
What antiretroviral drugs may prescribed for HIV-infected pregnant women in order to reduce the risk of infection of the fetus?
* Zidovudine and lopinavir
Retrovir and Viramune
Timazid and Retrovir
AZT and Retrovir
Zidovudine, Retrovir and timazid
What antiretroviral drugs may prescribed for HIV-infected pregnant women in order to reduce the risk of infection of the fetus?
Zidovudine and lopinavir
* Zidovudine and Nevirapine
Timazid and Retrovir
AZT and Retrovir
Zidovudine, Retrovir and timazid
What additional testing it is necessary to patients with infectious mononucleosis?
* IFA for BICH infection, bakobsledovanie of diphtheria
IFA for BICH infection, tularemia on bakobsledovanie
Bacteriological tests of diphtheria and typhoid fever
Reaction Burne and Wright-Heddlsona
The reaction of Paul Bunnelya and lymph node puncture
What immunological changes are identified in patients with HIV infection?
Is a polyclonal B-cell inhibition, increases the formation of autoantibodies and immune complexes
Is a polyclonal B-cell activation, reduced the formation of autoantibodies and immune complexes
Is a polyclonal B-cell inhibition, decreases the formation of autoantibodies and immune complexes
* There is a polyclonal B-cell activation, increases the formation of autoantibodies and immune complexes
Is a polyclonal B-cell activation, increases the formation of autoantibodies and decreases the formation of immune complexes
What laboratory findings are not typical for AIDS-related complex?
* Decreasing of the level of circulating immune complexes
The aspect ratio of 1.0 CD4/CD8
Anemia
Leukopenia
Thrombocytopenia
What laboratory findings are not typical for AIDS-related complex?
Increasing levels of circulating immune complexes
The aspect ratio of 1.0 CD4/CD8
Anemia
* Leukocytosis
Thrombocytopenia
What laboratory findings are not typical for AIDS-related complex?
* Decrease of immunoglobulins A is the G.
Decreasing CD4/CD8 ratio below 1.0
Anemiya
Leukopenia
Increase levels of circulating immune complexes
What laboratory findings are not typical for AIDS-related complex
* Decrease of immunoglobulins A is the G.
. Decreasing CD4/CD8 ratio below 1.0
Anemiya
Leukopenia
Increased levels of circulating immune complexes
What laboratory findings are not typical for AIDS-related complex?
Increased levels of circulating immune complexes
The aspect ratio of 1.0 CD4/CD8
Anemiya
* Leukocytosis
Thrombocytopenia
What laboratory findings are not typical for AIDS-related complex?
* Decrease in the level of circulating immune complexes
The aspect ratio of 1.0 CD4/CD8
Anemiya
leukopenia
thrombocytopenia
What antiretroviral drug exposure prophylaxis is carried out after contact with blood and other body fluids?
azidothymidine
nevirapine
* Azidothymidine + nevirapine
AZT + saquinavir
Іfavіrents
What term is usually defined by quantitative HIV RNA levels in blood plasma?
viral strain
the number of viral
* Viral load
viral factor
Viral RNA levels
What is the most common term of seroconversion in patients with HIV infection?
1 Week
1 month
* 3 months
1 year
indefinitely
What is the maximum duration of the incubation period in HIV infection?
1 month
3 months
1 year
* 5-6 years
indefinitely
What is the minimum duration of the incubation period in HIV infection?
* 1 week
1 month
3 months
1 year
indefinitely
What stage of the life cycle of the virus suppressed antiretroviral drugs, called protease inhibitors (PI)?
Entry of HIV into cells
reverse Transcription
integration
Transcription
* Build and branch
Clinical manifestations of acute retroviral syndrome, except:
Fever
various rashes
Influenzalike syndrome
Diarrhea
. Bleeding
Clinical manifestations of acute retroviral syndrome, except:
meningeal syndrome
various rashes
Influenzalike syndrome
diarrhea
* Jaundice
When AIDS can be diagnose?
Only when the number of CD4-lymphocytes less than 500 in 1 ml of blood
Only when the number of CD4-l lymphocytes less than 400 in 1 ml of blood
Only when the number of CD4-lymphocytes less than 300 in 1 ml of blood
* Only when the number of CD4- lymphocytes less than 200 in 1 ml of blood
Only when the number of CD4- lymphocytes less than 100 in 1 ml of blood
When prescribed antiretrovirals to a HIV-infected pregnant women in need of ART for their own health in order to reduce the risk of infection of the fetus?
* Regardless of the duration of pregnancy and childbirth
Only during childbirth
C 20 weeks of pregnancy and during labor
C 14 weeks
C 24 weeks of pregnancy and during labor
When prescribed antiretrovirals HIV-infected pregnant women that do not need ART for their own health in order to reduce the risk of infection of the fetus?
throughout pregnancy and during labor
Only during childbirth
20 weeks of pregnancy and during labor
14 weeks
* C 24 weeks of pregnancy and during labor
When prescribed antiretrovirals HIV-infected pregnant women in need of ART for their own health in order to reduce the risk of infection of the fetus?
. * Regardless of the duration of pregnancy and childbirth
Only during childbirth
C 20 weeks of pregnancy and during labor
C 14 weeks
C 24 weeks of pregnancy and during labor
When administered antiretroviral drugs to HIV-infected pregnant women to reduce the risk of infection of the fetus?
Throughout pregnancy and during labor
Only during childbirth
From 25 weeks of pregnancy and during labor
With 14 weeks of pregnancy
* From 28 weeks of pregnancy and during labor
When antiviral therapy for children born from HIV-infected women should starting?
* In the first hour after birth, 12.8
On the second day
Do not spend
After a month
After diagnosis SPIDA.
When the planned caesarean section for HIV-infected pregnant women reduce the risk of infection of the fetus?
In 36 weeks
In 37 weeks
* In 38 weeks
In 39 weeks
In 40 weeks
What cells is the main target for HIV?
T-suppressor
T-killers
* T-helper cells
D-cells
0-cells
What stage of HIV life cycle nucleoside reverse transcriptase inhibitors perform action (NRTIs)?
HIV penetration into cells
reverse Transcription
integration
transcription
* Replacement of the nucleoside
What stage of the life cycle is HIV zidovudine may destroy?
HIV penetration into cells
Reverse transcription
integration
transcription
* Replacement of the nucleoside
What stage of the life cycle is HIV non-nucleoside reverse transcriptase inhibitors (NNRTIs) may destroy?
HIV penetration into cells
* Reverse Transcription
Integration
Transcription
Broadcasting
What stage of the life cycle is HIV zidovudine nucleoside reverse transcriptase inhibitors (NRTIs) may destroy?
HIV penetration into cells
Reverse Transcription
Integration
Transcription
* Replacement of the nucleoside
What is acute retroviral syndrome clinically:
Fever
Lymphadenopathy
Enlargement of the liver
Meningeal syndrome
* All the above
What cells of the immune system, which contain CD4 molecules on their surfaces may infected by HIV.
Monocytes, macrophages
Macrophages, T-lymphocytes
T-lymphocytes, macrophages, cells of the central nervous system
* T-lymphocytes, cells of the CNS, monocytes, macrophages
Monocytes, macrophages, T-lymphocytes
What cells of the immune system, which contain CD4 molecules on their surfaces may infected by HIV.
Monocytes
Macrophages
T-lymphocyte
Microglia
* all the above
What cells of the immune system, which contain CD4 molecules on their surfaces may infected by HIV.
Monocytes
Macrophages
T-lymphocyte
Microglia
* all the above
HIV-positive, practical healthy baby of first year was allowed of such preventive vaccines, except:
Diphtheria Toxoid
Tetany toxoid
* oral polio
inactivated polio
pertussis
HIV-positive, practical healthy baby of first year was allowed of such preventive vaccines, except:
Diphtheria Toxoid
Tetany toxoid
* tuberculosis
inactivated polio
pertussis
HIV-positive, practical healthy baby of first year was allowed of such preventive vaccines, except:
Diphtheria Toxoid
Tetany toxoid
* Triple vaccine against measles, mumps, rubella
Inactivated polio
pertussis
HIV-positive, practical healthy baby of first year was allowed of such preventive vaccines, except:
Diphtheria Toxoid
Tetany toxoid
* measles
inactivated polio
pertussis
HIV-positive, practical healthy baby of first year was allowed of such preventive vaccines, except:
Diphtheria Toxoid
Tetany toxoid
* rubella
inactivated polio
pertussis
HIV-positive, practical healthy baby of first year was allowed of such preventive vaccines, except:
Diphtheria Toxoid
Tetany toxoid
* mumps
inactivated polio
pertussis
What is not typical for the IV clinical stage of AIDS, according to clinical stage classification, developed by experts of WHO(2006)?
Cachexia
PCP
Cerebral toxoplasmosis
Extrapulmonary cryptococcosis
* Cryptosporidiosis with diarrhea less than 1 month
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the IV clinical stage of AIDS?
Cachexia associated with HIV infection
PCP
Cerebral toxoplasmosis
Extrapulmonary cryptococcosis
* Diarrhea lasting less than 1 month
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the II clinical stage of AIDS?
Loss of body weight less than 10 % from the initial
Minimal damage of the skin and mucous membranes (seborrheic dermatitis, pruritus, fungal nail infections)
* Tuberculosis of the lungs, which has evolved over the year preceding the examination
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the II clinical stage of AIDS?
* Weight loss of more than 10 % from the initial
Persistent generalized lymphadenopathy
Episode of herpes zoster during last five years
Recurrent upper respiratory tract infections
Minimal mucosal lesions (recurrent ulcers of the oral mucosa, angular cheilitis)
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the II clinical stage of AIDS?
Loss of body weight less than 10 % from the initial
* Unmotivated chronic diarrhea that lasts more than 1 month
Episode of herpes zoster during last five years
Recurrent upper respiratory tract infections
Minimal mucosal lesions (recurrent ulcers of the oral mucosa, angular cheilitis)
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the IV clinical stage of AIDS?
Loss of body weight less than 10 % from the initial
* Multiple lesions of the skin and mucous membranes
Episode of herpes zoster
Recurrent upper respiratory tract infections
Minimal mucosal lesions (recurrent ulcers of the oral mucosa, angular cheilitis)
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the IV clinical stage of AIDS?
Loss of body weight less than 10 % of the initial
* Sarcoma Kaposhi
Episode of herpes zoster in the past five years
Recurrent upper respiratory tract infections
Minimal mucosal lesions (recurrent ulcers of the oral mucosa, angular cheilitis)
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the II clinical stage of AIDS?
Loss of body weight less than 10 % from the original
Minimal damage to the skin and mucous membranes (seborrheic dermatitis, pruritus, fungal nail infections)
* Tuberculosis of lungs, which has evolved over the year preceding the examination
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the II clinical stage of AIDS?
Loss of body weight less than 10 % from the initial
* Multiple lesions of the skin and mucous membranes
Episode of herpes zoster in the past five years
Recurrent upper respiratory tract infections
Minimal mucosal lesions (recurrent ulcers of the oral mucosa, angular cheilitis)
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the II clinical stage of AIDS?
* Weight loss of more than 10 % from the initial
Persistent generalized lymphadenopathy
Episode of herpes zoster in the past five years
Recurrent upper respiratory tract infections
Minimal mucosal lesions (recurrent ulcers of the oral mucosa, angular cheilitis)
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the II clinical stage of AIDS?
Loss of body weight less than 10 % from the initial
* Unmotivated chronic diarrhea that lasts more than 1 month
Episode of herpes zoster in the past five years
Recurrent upper respiratory tract infections
Minimal mucosal lesions (recurrent ulcers of the oral mucosa, angular cheilitis)
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the II clinical stage of AIDS?
Loss of body weight less than 10 % of the initial
* sarcoma Kaposhi
Episode of herpes zoster in the past five years
Recurrent upper respiratory tract infections
Minimal mucosal lesions (recurrent ulcers of the oral mucosa, angular cheilitis)
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the III clinical stage of AIDS?
Weight loss of more than 10 %
Unmotivated chronic diarrhea that lasts more than 1 month
Unexplained fever that lasts more than 1 month (permanently or temporarily)
* Episods of upper respiratory tract infection ( bacterial sinusitis)
Leukoplakia of the oral mucosa
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the III clinical stage of AIDS?
Lymphadenopathy more than 3 months
Unmotivated chronic diarrhea that lasts more than 1 month
Unexplained fever that lasts more than 1 month (permanently or temporarily)
* Cachexia
Cytomegalovirus chorioretinitis
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the IV clinical stage of AIDS?
Weight loss of more than 10 % of the initial
Unmotivated chronic diarrhea that lasts more than 1 month
Unexplained fever that lasts more than 1 month (permanently or temporarily)
* Single lymph nodes in one anatomical region, acute pain
Cytomegalovirus chorioretinitis
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the III clinical stage of AIDS?
Weight loss of more than 10 % of the initial
Unmotivated chronic diarrhea that lasts more than 1 month
Unexplained fever that lasts more than 1 month (permanently or temporarily)
* Single upper respiratory tract infection (eg, bacterial sinusitis)
Hairy leukoplakia of the oral mucos
According to clinical stage classification developed by experts of WHO (2006) what is not typical for the IV clinical stage of AIDS?
Cachexia
PCP
Cerebral toxoplasmosis
Carkoma sarcoma
* All the above
Experts of WHO believe suspicious according to AIDS lymph nodes:
3 or more nodes in more than two anatomical and topographical groups (except inguinal), measuring more than 2 cm in diameter, which extends more than 1 month.
3 or more nodes in more than two anatomical and topographical groups (except inguinal), larger than 1 cm in diameter, for more than 2 months.
* 2 or more nodes in more than two anatomical and topographical groups (except inguinal), larger than 1 cm in diameter, which lasts more than 3 months.
2 or more units in more than two anatomical topographic groups (except inguinal), larger than 2 cm in diameter, which extends over 2 months.
2 or more units in more than two anatomical topographic groups (except inguinal), larger than 1 cm in diameter, which continued for over 2 months.
Immediately after contact with blood and other biological liquids it is necessary to wash the dirty areas of skin with water and soap and to begin a postcontact prophylaxis, antiretroviral preparations not later than:
* 24-36 hrs
36-48 hrs
48-60 hrs
60-72 hrs
72-86 hrs
What antiretroviral preparation is taken as post contact prophylaxis after a contact with blood and other biological fluids?
* Azidotimidin
Nevirapin
Indinavir
Saqvinavir
Ifavirent
When do the plan caesarian section will conduct to HIV infected pregnant with the purpose for decreasing of the risk of infecting the fetus?
In 36 weeks
In 37 weeks
* In 38 weeks
In 39 weeks
In 40 weeks
After a car accident patient has been taken to hospital in critical condition, caused by shock, bleeding. Before blood transfusion the doctor should consider:
Health reasons
in order to prevent HIV transmission The conclusion of doctors consilium
Agree of the patient (or his family)
The results of a survey of emergency donor about HIV (IHA)
* All the above
Immediately after contact with blood or other body fluids person should wash exposed skin with soap and water, and contaminated mucous membranes - with clean water. When is the post-exposure prophylaxis with antiretroviral drugs should start?
*No later than 72 hours
B for a week
If the infection is confirmed
Not available
Seropositive persons
Immediately after contact with blood or other body fluids person should wash exposed skin with soap and water, and contaminated mucous membranes - with clean water. How long is conducted post-exposure prophylaxis?
During a week
*4 weeks
3 months
Until the end of the observation period
Not available
At what dose of antiretroviral drug prophylaxis is carried out after contact with blood and other body fluids?
600-800 mg
700-800 mg
* 800-1000 mg
1000-1100 mg
1100-1200 mg
How long is the antiretroviral prophylaxis after contact with blood and other body fluids perfomed?
1 Week
2 weeks
* 1 month
3 months
6 months
The epidemic outbreak rationally organize inspection of the immune system. Using the IHA to identify nonimmune individuals to diphtheria in a few hours. What is the minimum protective titer?
1:10
1:20
* 1:40
1:80
1:160
The most important clinical criteria for AIDS must include the following, except:
Significant weight loss (10%) if it continues over a month
Duration of fever, if it continues over a month
Persistent diarrhea, if it lasts more than a month
* A rash on the skin, if it continues over a month
Lymphadenopathy than 3 mesyatseB.
How to decide on the child's immunizations with unclear HIV status to HIV-infected mothers?
Vaccination against diphtheria and tetanus
Polio vaccination
Vaccination against tuberculosis
Vaccination against polio and tuberculosis
* Vaccination is not performed
What additional testing is necessary to patients with infectious mononucleosis?
*ELISA for HIV, bacteriology of diphtheria
ELISA for HIV, bacteriology for tularemia
bacteriology of diphtheria and typhoid fever
Reaction Burne and Wright-Heddlsona
The reaction of Paul Bunnelya and lymph node puncture
What immunological changes are identified in a patient with HIV infection?
Is a polyclonal B-cell inhibition, increases the formation of autoantibodies and immune complexes
Is a polyclonal B-cell activation and decreases the formation of autoantibodies and immune complexes
Is a polyclonal B-cell inhibition, decreases the formation of autoantibodies and immune complexes
* There is a polyclonal B-cell activation, increases the formation of autoantibodies and immune complexes
Is a polyclonal B-cell activation, increases the formation of autoantibodies and decreases the formation of immune complexes
What laboratory findings are not typical of Sneed-related complex?
Decreasing the amount of immunoglobulin A is the G.
* Reducing the ratio below 1.0 CD4/CD8
Anemia
Leukopenia
Increased levels of circulating immune complexes
Which antiretroviral drug prophylaxis is carried out after contact with blood and other body fluids?
Azidothymidine
Nevirapine
* Azidothymidine + nevirapine
AZT + saquinavir
Іfavіrents
What term is usually defined by quantitative HIV RNA levels in blood plasma?
Viral strain
The number of viral
* Viral load
Viral factor
Viral RNA levels
What are the characteristics of plaque in diphtheria?
Single-sided, gray-white, on the surface ulcer crater
* The gray-white, dense, with sharp edges and glossy surface
Yellow-white, brittle, located perilakunarno
Sided, yellow-white, in the gaps
White, brittle, easily removed with a spatula
What stage of the life cycle of the virus are suppressed by antiretroviral drugs, called protease inhibitors (PI)?
Entry of HIV into cells
reverse Transcription
integration
transcription
* Build and branch
When you can make a diagnosis of AIDS?
Only when the content of CD4-lymphocytes less than 500 in 1 ml of blood
Only when the content of CD4- lymphocytes less than 400 in 1 ml of blood
Only when the content of CD4- lymphocytes less than 300 in 1 ml of blood
* Only when the content of CD4- lymphocytes less than 200 in 1 ml of blood
Only when the content of CD4- lymphocytes less than 100 in 1 ml of blood
When are antiretroviral drugs administered to HIV-infected pregnant women to reduce the risk of infection of the fetus?
Throughout pregnancy and during labor
Only during childbirth
From 25 weeks of pregnancy and during labor
With 14 weeks of pregnancy
* From 28 weeks of pregnancy and during labor
When is antiviral therapy started for children born to HIV-infected women?
* In the first hour after birth, 12.8
On the second day
Do not spend
After a month
After diagnosis SPIDA.
When is the planned caesarean section for HIV-infected pregnant women to reduce the risk of infection of the fetus?
In 36 weeks
In 37 weeks
* At 38 weeks
In 39 weeks
In 40 weeks
Who is the primary target for HIV?
T-suppressor
T-killers
* T-helper cells
D-cells
0-cells
Violation of which stage of the life cycle are caused by nucleoside reverse transcriptase inhibitors (NRTIs)?
Entry of HIV into cells
reverse Transcription
integration
transcription
* Replacement of the nucleoside
Violation of which stage of the life cycle of HIV is zidovudine?
Entry of HIV into cells
reverse Transcription
integration
transcription
* Replacement of the nucleoside
Violation of which stage of the life cycle are caused by non-nucleoside reverse transcriptase inhibitors (NNRTIs)?
Entry of HIV into cells
* Reverse Transcription
integration
transcription
Translation
Complication in the 4-5th week of diphtheria:
Encephalitis
Bulbar disorders, pancreatitis, hepatitis
* Polyradiculitis, myocarditis
Nephrosonephritis
Stenosing laryngotracheitis
Complication that often develops in the first week of diphtheria oropharynx:
Polyradiculitis
Asphyxia
Failure of adrenal glands
Hepatosplenomegaly
* Paresis of the soft palate
Particularly high titre diphtheria antitoxic antibodies in moderate titer of anti-tetanus antibodies indicates:
Tetanus
Diphtheria
Carriering of Corynebacterium diphtheria
Immunity to diphtheria and to the formation of bacteria
* Nothing
Before revaccination against diphtheria in adults, they recommended:
* Identify the antibody titer
Preventive antibiotics
Proactively assign antihistamines
Five years after the last booster
10 years after vaccination
List all the cells of the immune system, which contain CD4 molecules on their surfaces that are infected with HIV.
Monocytes, macrophages
Macrophages, T-lymphocytes
T-lymphocytes, macrophages, cells of the central nervous system
* T-lymphocytes, cells of the CNS, monocytes, macrophages
How to prevent occupational HIV infection?
Local wound treatment
* Post eccident HAART
Laboratory testing for HIV
Register the fact of the accident in a special register
All the above
What preparations are necessary for prevention of occupational HIV infection?
Local wound treatment
* Antiretrovirus drugs
Antibiotics
Specific immunoglobulin
All the above
During assistance nurse accidentally pricked her finger with a contaminated needle. What is prevention of disease?
The combination of NRTIs + PI
* Combination NRTI + NNRTI
Interferons
Specific immunoglobulin
Initial debridement
During assistance nurse accidentally pricked her finger with a contaminated needle . For the recognition of occupational HIV infection?
Seroconversion after crash
HIV asymptomatic infection in accident
Occupational exposure
* Infection occurred before the accident
All the above
During assistance nurse accidentally pricked her finger with a contaminated needle . For the recognition of occupational HIV infection :
Confirmation of HIV
Accounting the fact of the accident in a special register
Negative results of laboratory examination in the first 5 days after the accident
Positive results of laboratory testing for HIV at 1, 3 or 6 months after the accident
* All the above
During assistance nurse accidentally pricked her finger with a contaminated needle . For the recognition of occupational HIV infection :
Confirmation of HIV
Statement of victim
* Negative results of laboratory examination in the first 5 days after the accident and positive 1, 3 or 6 months after the accident
Availability of health book in the affected
Full-time work regimen
A child of HIV-positive without clinical and laboratory signs of the disease. How to solve the problem of vaccination against polio?
* Conduct, as it is provided in the Calendar routine immunization
Vaccination dont perform
Hold until the results of a survey on child markers HIV
Not available as prohibit the use of live vaccines
Show all of the above is true
A child of HIV-positive without clinical and laboratory signs of the disease. How to solve the problem of vaccination against polio?
Not available as prohibit the use of live vaccines
Hold, as it is provided according the Calendar routine immunization
Postponed until the results of child markers HIV
* Replace the live vaccine to inactivated
Vaccination dont perform
A child of HIV-infected mother, was born apparently healthy. When you can confirm HIV-negative status of the child?
Immediately after birth if there are no clinical manifestations
After 3 months in the absence of HIV markers
After 6 months in the absence of the child HIV markers
* After 18 months in the absence of the child markers HIV
Never, since the HIV transmission from mother occurs in 100% of cases
A child of HIV-infected mother, was born apparently healthy. How to solve the problem of vaccination against tuberculosis?
Hold, as it is provided in the Calendar routine immunization
Performed if there is no clinical and laboratory signs of disease
Postponed until the results of a survey on child markers HIV
* Not available as prohibit the use of live vaccines
Vaccination dont perform
A child of HIV-infected mother, was born prematurely, with clinical signs of AIDS. How to solve the question of vaccination (tuberculosis, hepatitis B)?
Hold, as it is provided in the Calendar routine immunization
Performed if there is no clinical and laboratory signs of disease
Postponed until the results of a survey on child markers HIV
Not available as prohibit the use of live vaccines
* Vaccination dont perform
Immediately after contact with blood and other body fluids should be washed exposed skin with soap and water, and contaminated mucous membranes - with clean water. When is the post-exposure prophylaxis with antiretroviral drugs?
* No later than 72 hours
During the week
If the infection is confirmed
Not available
Seropositive persons
In a different places of settlement a few cases of cholera was found. Who in the focus of cholera will be send in a hospital?
Carriers
Persons contact with the patient
*Patients with cholera
Persons with dysfunction of alimentary tract
Persons with hyperthermia
Diagnosed a patient: chronic hepatitis in the stage of integration. What markers will be in patient in this stage disease?
HBeAg
Antibodies to HBeAg
DNA OF HBV
Viral DNA-polimerase
*HBsAg, anti-НBе
As etiotropic therapy of acute and chronic viral hepatitis B utillize:
Corticosteroid
Immunomodulators
Cytostatics
Antibiotics
*Antiviral
Direct bilirubin is increased , in urine there is significant increase of bilirubin and urobilin, increasing of stercobilin of excrements. What is the type of icterus?
Haemolitic
*Parenhimatous
Transport
Extraliver
Mechanical
On the average 15 to 30 % of all population of the planet suffer from some pathology of liver. Prevalence of hepatitis and cirrhosis in the European countries is about 1 % of adults. Annually in the world there are about 2 million people with acute viral hepatitis. What % of all cases will develop chronic form.
100 %
50 %
25 %
*10 %
1 %
When sick people get epidemic typhus infection, which period affects more?
Over the past 2 days, the incubation period and 2-3 days after lowering temperature
All hectic period and 2-3 days after lowering temperature
2-3 days after lowering temperature
*Over the past 2 days, the incubation period, all febrile period and 2-3 days after lowering temperature
Over the past 2 days, the incubation period and the hectic period
In which period the maximal growth of infection occurs during epidemic typhus?
At the incubation period
*At the 1th week of illness
At the 2nd week of illness
At the 3rd week of illness
At the time of recovery
Often, in patient with epidemic typhus arise transition petehies in the conjunctivA. What term is used to describe this?
Symptom of Heller
Conjunctivitis
Symptom of Govorova-Godele
*Symptom of Zorohovich-Chiari-Avtsyna
Enantema Rosenberg‘s
In the family of the patient with epidemic typhus were lice in the children. With the help of any of these measures could prevent the subsequent spread of the disease?
*Monitoring and complete sanitation of contact in the centre
The use of chemoprophylaxis
The use of antibiotics
Isolation contact
Check-up
When can you stop the etiotropic medication treatment of the patient with epidemic typhus?
Immediately after the normalization of body temperature
After the normalization of the liver and spleen
*After a 2-day normal body temperature
After the disappearance of roseola
Within 10 days after the disappearance of roseola
Often, in patient with epidemic typhus arise transition petehies in the conjunctivA. What term did it call?
Symptom of Heller
Conjunctivitis
Symptom of Govorov-Godele
*Symptom of Zorohovich-Chiari-Avtsyna
Enantema Rosenberg‘s
Often, in patient with epidemic typhus arises petehies on mucosal soft palate. What term did it call?
Symptom of Heller
Conjunctivitis
Symptom of Govorova-Godele
Symptom of Zorohovich-Chiari
*Enantema Rosenberg‘s
Often, in patient with epidemic typhus is tongue‘s tremor when protrusion that sticked on the lower teeth. What term did it call?
Symptom of Heller
Conjunctivitis
*Symptom of Govorova-Godele
Symptom of Zorohovich-Kiari
Enantema Rosenberg‘s
In the typical form of typhoid fever, the body temperature rises progressively from day to day 39-40 °C at the end of the 1st week, and during the next 10-14 days it gets back approximately to this level, and then becomes remittent and, gradually goes down to the norm. What is such temperature curve called?
*The temperature curve as Vunderlihs
The temperature curve as Botkin
Temperature curve as Kildushevsky
Temperature curve as Ellers
Intermedium temperature curve
In the typical form of typhoid fever the body temperature rises to 39-40 °C. The temperature curve looks like 2 waves (during 3-4 weeks of disease). What is such temperature curve called?
The temperature curve as Vunderlihs
*The temperature curve as Botkin
Temperature curve as Kildushevsky
Temperature curve as Ellers
Intermedium temperature curve
In the typical form of typhoid fever, the body temperature rises pregressively from day to day to 39-40 °C at the end of the 1st week, and then gradually during 2-3 week it goes down to the norm. What is such temperature curve called?
The temperature curve as Vunderlihs
The temperature curve as Botkin
*Temperature curve as Kildushevsky
Temperature curve as Ellers
Intermedium temperature curve
One methods of diagnostics of typhoid fever and paratyphoid fever is the selection of hemoculture. This is done during the fever period blood is taken from the vein on bilious bulione or Rappaport‘s media in correlation 1:10 is made. What volume of blood must be taken on the 2nd week of disease?
5 ml of blood
10 ml of blood
*15 ml of blood
20 ml of blood
25 ml of blood
One methods of diagnostics of typhoid fever and paratyphoid fever is the selection of hemoculture. This is done during the fever period blood is taken from the vein bilious bulione or Rappaport‘s media in correlation 1:10 is made. What volume of blood must be taken on the 1st week of disease?
0,5 ml of blood
*10 ml of blood
15 ml of blood
20 ml of blood
25 ml of blood
One methods of diagnostics of typhoid fever and paratyphoid fever is the selection of hemoculture. This is done during the fever period blood is taken from the vein bilious bulione or Rappaport‘s media in correlation 1:10 is made. What volume of blood must be taken on the 3nd week of disease?
5 ml of blood
10 ml of blood
15 ml of blood
*20 ml of blood
25 ml of blood
When sick people gets epidemic typhus infection, which period affects more?
Over the past 2 days, the incubation period and 2-3 days after lowering temperature
All hectic period and 2-3 days after lowering temperature
2-3 days after lowering temperature
*Over the past 2 days, the incubation period, all febrile period and 2-3 days after lowering temperature
Over the past 2 days, the incubation period and the hectic period
On which period the maximal growth of infection occurs during epidemic typhus disease?
At the incubation period
*At the 1th week of illness
At the 2nd week of illness
At the 3rd week of illness
At the time of recovery
One of methods of diagnostics of typhoid fever and paratyphoid fever is the selection of hemoculture. For that in a fever period sowing of blood from a vein on bilious bulione or Rappaport‘s media in correlation 1:10 is made. What volume of blood must be taken on the 2nd week of disease?
5 ml of blood
10 ml of blood
*15 ml of blood
20 ml of blood
25 ml of blood
One of methods of diagnostics of typhoid fever and paratyphoid fever is the selection of hemoculture. For that in a fever period sowing of blood from a vein on bilious bulione or Rappaport‘s media in correlation 1:10 is made. What volume of blood must be taken on the 1st week of disease?
0,5 ml of blood
*10 ml of blood
15 ml of blood
20 ml of blood
25 ml of blood
One of methods of diagnostics of typhoid fever and paratyphoid fever is the selection of hemoculture. For that in a fever period sowing of blood from a vein on bilious bulione or Rappaport‘s media in correlation 1:10 is made. What volume of blood must be taken on the 3nd week of disease?
5 ml of blood
10 ml of blood
15 ml of blood
*20 ml of blood
25 ml of blood
What is etiology of acute primary tonsillitis?
Influenza virus
*β-hemolitic streptococci
N. meningitidis
M. tuberculosis
K. pneumonia
What group of lymph nodes more often change in acute tonsillitis?
Anterior cervical
*Submandibular
Posterior cervical
Retroauricular
Occipital
What is most usual complication of acute tonsillitis?
Otitis
Sinusitis
*Abscess
Frontitis
Pneumonia
Differential diagnosis of acute tonsillitis should perform with?
Infectious mononucleosis
Diphtheria
Scarlet fever
Adenoviral infection
*All the above
Which group of antibiotics is most effective in treatment of tonsillitis?
*Penicillin
Tetracycline
Cephalexin
Sulfalen
Ampicillin
What is the duration of incubation period of tonsillitis?
*Few hours – two days
Few days – one week
Few weeks – one month
1 – 10 hours
1- 10 days
In tonsillitis streptococcus usually cultivated from:
Blood
Urine
Feces
*Mucous
Saliva
What group of infectious diseases diphtheria belong to according to L. Gromashevsky classification?
Intestinal infection
*Infection of respiratory tract
Blood infection
Infection of external covers
Transmissive
What is the agent of diphtheria?
Influenza virus
β-hemolitic streptococci
N. meningitidis
*B. Leffleri
K. pneumonia
Diphtheria is transmitted by:
Food
Water
*Air
Blood
Milk
What rear form of diphtheria localization do you know?
Laryng
Trachea
Bronch
*Eye
Nose
What early complication more often may occur in diphtheria?
Pneumonia
*Myocarditis
Gastritis
Otitis
Frontitis
What is the agent of infectious mononucleosis?
*Epstein-Barr virus
β-hemolitic streptococci
N. meningitidis
B. Leffleri
K. pneumonia
Which of clinical symptoms are more typical for infectious mononucleosis?
Fever, hepatomegaly, abdominal pain