Actual problems of infectious diseases and hiv infection
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Partial answer Absence of answer *All the above What is organism answer of interferon therapy. Disappearance of markers of viral replication upon completion of course of therapy Normalization of activity of ALaT during the course of therapy Relapse in next 6 months Disappearance of icterus *All the above Answer of interferon therapy are all, except. *Disappearance of markers of viral replication Normalization of ALaT activity Disappearance of icterus Normalization of patient general condition Normalization of a liver size What investigation for confirming of viral hepatitis diagnosis. Total analysis of blood Determination of bilirubin level Determination of aminotransferases activity *Determination of HV markers by IFA All the above What examination is helpful in confirming of viral hepatitis diagnosis. Complete analysis of blood Ultrasound of abdominal region Determination of activity of aminotransferase *Determination of antigen of viruses Duodenal probing What is duration of incubation period for B hepatitis : 45 days *180 days 360 days 90 days 25 days Parenteral way of transmission is present in spreding of hepatitis except: *A B C D TTV Chronic course is possible for viral hepatitis except: *A B C D B+C All the following medicines are interferons except: Intron Roferon Reaferon Leukinferon *Cycloferon All the following medicines are hepatoprotective agents except: Carsil Silibor Legalon *Lomusol Arginine What is mechanism of viral hepatitis A transmission: Contact Transmissive Vertical *Fecal-oral Air-drop What is mechanism of hepatitis B transmission: *Contact Transmissive Alimentary Fecal-oral Air-drop What is mechanism hepatitis C transmission: *Contact Transmissive Alimentary Fecal-oral Air-drop What rash is present in case of haemorrhagic fevers with kidneys syndrome? Roseolar Maculo-papular Punctuate *Petechial Rash is not present What rash is present in case of Congo hemorrhagic fever? Roseola Maculo-papular Punctulate *Petechial Rashes not is characteristic What rashes is present in case of Crimea hemorrhagic fever? Roseolar Maculo-papular Punctulate *Petechial Rashes not is characteristic How long the rash is present in case of haemorrhagic fever with kidneys syndrome? *During all feverish period Before the reconvalescense Before development of clinical features of kidneys insufficiency During whole disease Appears yet in a latent period and disappears in the period of early reconvalescense A kidney syndrome at haemorrhagic fever with kidneys syndrome shows up usually: Only laboratory changes Only on BRIDLES *By pain in lumbar area, positive Pasternatsky symptom, development of oliguria By fever, polyuria, dyspepsia By paradoxical ischuria What changes in biochemical blood test inherent for haemorrhagic fever with kidneys syndrome? Increase level of urea and bilirubin The level of urea and kreatinine falls The level of kreatinine grows and urea falls The level of urea grows and kreatinine falls *The level of urea and kreatinine grows What changes in haemogram is typical for haemorrhagic fever with kidneys syndrome? Normochromic anaemia, leucocytosis with atypical mononucleosis, thrombocytopenia enhanceable ESR Erythrocytosis, lymphocytosis,ESR is enhanceable Normochromic anaemia, leucopenia with neutrophylosis, thrombocytopenia enhanceable ESR *Hypochromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia enhanceable ESR Hyperchromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia mionectic ESR Polyuria in haemorrhagic fever with kidneys syndrome is a sign of: *Recovering . Chronic process Unfavorable flow of illness Development of complications Complete convalescence In most patients with Congo hemorrhagic what type of fever is: Wunderlich’s type Botkin’s type Undulating Intermittent *Two-humped What changes in haemogram inherent Congo hemorrhagic fever? Normochomic anaemia, leucocytosis mononuclear Erythrocytosis, lymphocytosis *Hypochromic anemia, erythrofilosis Hypochromic anemia, neutrofilosis Hyperchromic anemia, neutrofilosis What is typical for the Lassa hemorrhagic fever: Effect of cardiovascular system Development of acute hepatic insufficiency Hundred-per-cent lethality *Defeat of breathing organs Development of paresis and paralysis Confirm diagnosis of haemorrhagic fever with kidneys syndrome by a way of: Only virological methods Only bacteriological methods Bacteriological and serum methods Proper epidemiological information *Virologic and serum methods Confirm the diagnosis of Lassa hemorrhagic fever by a way of: Only virological methods Only bacteriological methods Bacteriological and serum methods Proper epidemiological information *Virologic and serum methods Confirm the diagnosis of Congo hemorrhagic fever by a way of: Only virological methods Only bacteriological methods Bacteriological and serum methods Proper epidemiological information *Virologic and serum methods Confirm the diagnosis of Ebola fever by a way of: Growth of viruses on chicken embryons Only bacteriological methods Bacteriological and serum methods Proper epidemiological information *Selection of virus on the Vero culture Confirm the diagnosis of Omsk fever by a way of: Growth of virus on chicken embryons Only bacteriological methods Bacteriological and serum methods Proper epidemiological information *Selection of virus on the Vero culture Confirm the diagnosis of Marburg fever by a way of: Growth on chicken embryos Only bacteriological methods Bacteriological and serum methods Proper epidemiologys information *Selection of virus on the Vero culture What etiothropic means use at treatment of haemorrhagic fever with kidneys syndrome: Benzylpenicillin Dopamine *Virolex Dexamethazone Etamsylatum What etiothropic means use at treatment of patients with Lassa fever: Benzylpenicillin Dopamine *Ribavirin Dexamethazole Etamsylatum What etiothropic means use at treatment of patients with Marburg fever: Benzypenicillin Dopamine *Ribavirin Dexamethazone Etamsylatum What etiothropic means use at treatment of patients with Congo fever: Benzylpenicillin Dopamine *Ribavirin Dexamethazone Etamsylatum What etiothropic means use at treatment of patients with Ebola fever: Benzylpenicillin Dopamine *Virolex Dexamethazone Etamsylatum What etiothropic means use at treatment of patients with Crimea fever: Benzylpenicillin Dopamine *Ribavirin Dexamethazone Etamsylatum Specific prevention of hemorrhagic fevers may perform by: Live vaccine Killed vaccine Specific immunoglobulin *Do not develop Polivalent vaccine Who is the source of the causal agent in the Crimean-Congo haemorrhagic fever? Rodents, cattle, birds Tikes *Rodents, cattle, birds, sick people Sick man, reconvalescent, bacteriocarrier Rodents, cattle, birds, sick people, bacteriocarries The source of infection of Omsk‘s hemorrhagic fever are muskrat, water rats and other rodents. Who are the carriers? Bee and flea *Pliers and flea Mosquitoes Fly Pliers and mosquitoes Specific prevention of Crimean-Congo haemorrhagic fever are: *Vaccine and human immunoglobulin Serum Serum and human immunoglobulin Do not developed Antibacterial drugs How to prevent malaria? Follow the rules of personal hygiene Do not drink raw water Vaccination *Chemoprophylaxis Heating of food What group of infectious diseases malaria belong to? Intestinal Respiratory External covers Transmissive *Blood What is the treatment of malaria attacks. Antibiotics Serum transfer Delagil Primaquine *Delagil + Primaquine In a survey of donor blood microhametosis are found. Assign treatment. Delagil *Primaquine Antibiotics Fluoroquinolone Sulfanilamide Malaria must be differentiated primarily with such diseases: Pyelonephritis Sepsis Viral hepatitis Leptospirosis *All of the above The diagnosis of malaria can be confirmed by: Microscopy of urine Haemoculture Bacteriology of stool Serological reactions *Parazitoscopy of blood Rules of hospitalization of patients with malaria: *In separate room In a respiratory infections department In the Meltser’s ward Patients are not hospitalized In a intestinal infections department Why early relapse in malaria develops? The immune deficiency *Due to erythrocytic forms of shizonts Releasing of tissue shizonts to the blood stream Fresh contamination Availability of a blood gamonts Why late relapses in malaria develops? Due to erythrocytic forms of shizonts *Releasing of tissue shizonts to the blood stream Fresh contamination Availability of a blood gamonts The immune deficiency By what drug is possible to prevent early recurrence of malaria: *Delagil Immunoglobulin Antibiotics Primahin Glucocorticoids By what drug is possible to prevent relapse of malaria: Delagil Immunoglobulin Antibiotics *Primahin Glucocorticoids Radical treatment of malaria include: Primaquine within 2 weeks Glucocorticoids *Delagil + primahin Serum transfer Indications for the appointment of hematoshizotrop antimalarial drugs: *Attack of malaria Preventing late relapse Prophylactic course after returning from areas difficult to malaria Antyretsidiv course for the rekonvalescents During a check-up Indications for the appointment of histoshizotrop antimalarial drugs: Attack of malaria *Prevention of late relapse Complications of malaria Malaria chemoprophylaxis for a period of stay in endemic areas During a check-up What do you need to give to the patients with malaria‘s coma? Glucocorticoids Antishok treatment *Intravenous delagil Blood transfusion Oxygen Specific complications of malaria, except: Hemoglobinuria fever Spleen destruction Malaria‘s coma *Intestinal perforation Hemolytic anaemia Reconvalences after malaria may descharged from a clinic no earlier than: After 2 weeks till complete clinical recovery After 2 weeks of a radical course of therapy *After a radical course of therapy with a negative results of parazitoscopy After 3 weeks with a negative blood culture results After 3 weeks, if the negative results of planting feces Activities on contact with import cases of malaria: Parasitoscopy of blood The direction of the contact in the detention facility for 5 days Chemoprophylaxis Vaccination *Do not hold What is malaria prevention for those who have returned from disadvantaged areas: Introduction of human immunoglobulin Interferon 6-days-prevention by streptomycin or tetracycline *Primaquine All the above What measures must be taken in relation to people who had a contact with malaria case: Microscopy of periferal blood Isolation during 5 days Chimioprophylaxis Houses rounds *Does not conduct Who would be a subject for inspection on malaria? Recovering of malaria persons Persons, returning from endemic regions of malaria Patients with fever more than 5 days Patients with spleenomegaly *All above enumerated Methods for identification of sources of malaria: Stool culture test Haemoculture *Microscopic assessment of blood Byurne test All above enumerated When should begin treatment of malaria patients? *Immediately after hospitalization After taking of material for research After of final diagnosis After laboratory and instrumental research All answers are correct Delagil appoint in case of malaria in such doses: 0,5 g 3 times a day 3 days 0,5 g during a week *In the first day 1 g, then 0,5 g every 6 h 0,5 g a day during a month 0,5 g 2 times a day 3 days There are etiotropic drugs of malaria, except: Delagilum Prymahin *Ceresyn Quinine Fansydar The radical course of treatment of malaria includes: Five-day therapy of delagilum Prymahin during 2 weeks C. Delagilum + prymahin + fansydar *Delagilum + prymahin Delagilum + fansydar What do you need for treatment of chlorochyn resistent forms of malaria? Bactrimum Dapson Fansydar Meflohin *All enumerated The most effective antibiotics at the treatment of patients with leptospirosis are: *Penicillin Macrolids Cefalosporins Ftorhinolons Sulfanilamids What daily doses of penicillin for leptospirosis treatment: 2-3 million units *3-12 million units 10-20 million units 20-40 million units Over 40 million units All of these is an epidemic dangerous to leptospirosis except: Farm animals Wide rodents Domestic animals Foxes *Humans How long lasts the incubation period of leptospirosis: 2 month 1-7 days *7-14 days 14-21 days 2-3 days Which serotypes of leptospirosis caused the disease more frequent: L. interogans L. grippotyphosa L. canicola *L. icterohaemorrhagia L. Pomona Agglutinines in leptospirosis arrive at a maximal titre: On the third day of illness *On the third week of illness and later On the fourth week of illness On the second month of illness On the second week of illness Rules of hospitalization of patients with infectious mononucleosis: Patients are not hospitalized In a ward for the infections of respiratory tracts *In a separate ward In a ward for the infections of external covers In a ward for intestinal infections What symptoms are not characteristic of infectious mononucleosis? Hepatomegaly *Oliguria Limfocytosis Tonsillitis Splenomegaly How much atypical mononucleares present in infection mononucleosis? Less than 5 cells *10 and more cells 5 and more cells Less than10 cells 50 % cells What is most possible complication occur in infectious mononucleosis? Oliguria Autoimmune process Meningitis *Spleen rupture Pneumonia What additional inspections must be conducted to the patient with infectious mononucleosis? *IFA on HIV-infection, bacteriology inspection on diphtheria IFA on HIV-infection, bacteriology inspection on a scarlet fever Bacteriology inspection on diphtheria and typhoid fever Burne and Rihth-Heddlson reactions Paul-Bunnel reaction and punction of lymphatic node What from the following symptoms are not characteristic of infectious mononucleosis? Fever *Defeat of kidneys Lymphadenopathy Tonsillitis Increasing of liver and spleen For what disease characterize changes in a blood (presence of lymphomonocytes and atypical mononuclears? Flu *Infectious mononucleosis Measels AIDS Diphtheria In a survey of donor blood found microhametosis. Assign treatment. Delagil *Primaquine Antibiotics Fluoroquinolone Sulfanilamide Diagnosed a patient: chronic hepatitis in the stage of integration. What markers will be in patient in this stage disease? HBeAg Antibodies to HBeAg DNA OF HBV Viral DNA-polimerase *HBsAg, anti-НBе As etiotropic therapy of sharp and chronic viral hepatitis B utillize: Corticosteroid Immunomodulate preparations Cytostatics Antibiotics *Antiviral preparations Give recommendation for a patient in reconvalensent period of viral hepatitis during a clinical supervision after isolation. *Medical supervision during 6 months, periodic biochemical inspections. Control bacteriological examinations Full labor investigation To continue prophylactic inoculations Supervision is not needed Etiotropic therapy of viral hepatitis is. Ribavirin Interferon Inductors of interferon Zefix *All the above Choose the remedies for etiotropic therapy of viral hepatitis. *Ribavirin Vaccine Normal human immunoprotein Hepatoprotector Glucocorticoid Choose the remedies for etiotropic therapy for viral hepatitis. Antibiotics *Interferon Probiotics Vaccine Normal human immunoprotein The criteria for application of etiotropic therapy in viral hepatitis is. Protracted motion of HBV, HVD Any form of HV Biochemical activity Presence of virus replication *All the above The criteria for application of etiotropic therapy for the patient with HCV. Clinical displays are insignificant Icterus is absent Moderate biochemical activity There is anti-HCV in blood *RNA of HCV + Factors which are indications of successful interferon therapy in HV infections are all, except. Level of ALaT not more than 2-3 norm Low titre of HCV after the treatment Absence of cholestasis 2th and 4th genotypes of HCV *Expressed fibrosis Indirect action of interferon therapy. Influenza-like syndrome Nausea Itching Para-hypnosis *All the above|| Indirect action of interferons. Flatulence Diarrhea Nausea Depression *All the above Indirect action of interferon therapy are all except. Influenza-like syndrome Nausea Depression Intensification of autoimmune diseases *Progress of fibrosis Basic principles of antiviral therapy for viral hepatitis. Individual selection of dose and rhythm of application of preparations Duration of introduction of preparations Control of amount of erytrocytes, leucocytes and thrombocytes, in blood Control of iron level in blood *All the above Contra-indications for antiviral therapy of viral hepatitis. Decompensatory cirrhosis of liver Thrombocytopenia <50000 in 1 мм3 Psychic disorders Leucocytopenia <1500 in 1 мм3 *All the above Contra-indications for antiviral therapy of viral hepatitis. Decompensatory cirrhosis of liver Autoimmune disease Alcoholism and other drug addictions Coinfection by HIV *All the above Choose the indexes of efficiency of interferon therapy. *Disappearance| of markers of viral replication Improvement of the general state Normalization of the liver size Disappearance of icterus All the above Choose the indexes of efficiency of interferon therapy. Improvement of the general state *Normalization of activity of ALaT Normalization of the liver size Disappearance of icterus All the above Types of answer for interferon therapy are. Stable remission Unsteady Yüklə 3,17 Mb. Dostları ilə paylaş:
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