Systemic scleroderma

5*

7

10

11

2064.In the treatment of chronic pancreatitis with a view to appoint replacement

1. 
   morfin
   
2. 
   But silos
   
3. 
   Panzinorm*
   
4. 
   holosas
   
5. 
   penicillin
   

2065.If the clinical examination of patients with chronic pancreatitis examined blood levels

1. 
   squirrel
   
2. 
   Glucose*
   
3. 
   fibrinogena
   
4. 
   cholesterol
   
5. 
   pepsin
   

2066. Hacells of the pancreas synthesized

1. 
   Insulin
   
2. 
   Glucagon*
   
3. 
   Gastrin
   
4. 
   Pankretichesky polypeptide
   
5. 
   Vasoactive intestinal peptide
   

2067. Pcells of the pancreas synthesized

1. 
   Somatostatin
   
2. 
   Secretin
   
3. 
   Insulin*
   
4. 
   Amylase
   
5. 
   Glucagon
   

2068.The main etiological factor of chronic pancreatitis is considered

1. 
   The abuse of fatty and spicy foods
   
2. 
   Long forced a sitting position
   
3. 
   Abuse of sweet food
   
4. 
   Kidney stones
   
5. 
   Stones in the gallbladder*
   

2069.The pathogenesis of chronic pancreatitis is

1. 
   Accelerated evacuation of secretion of the pancreas
   
2. 
   Lack of development of pancreatic secretion
   
3. 
   Infection of pancreatic secretion
   
4. 
   Intraorgan activation of pancreatic enzymes*
   
5. 
   Increasing the concentration of bicarbonate in the pancreatic secretion
   

2070.Steatorrhea is

1. 
   Elevated levels of fecal neutral fat*
   
2. 
   Elevated levels of muscle fibers
   
3. 
   Elevated fecal elastase
   
4. 
   Elevated fecal chymotrypsin
   
5. 
   Detection of fecal elements of inflammation
   

2071.Kreatoreya is

1. 
   Elevated levels of fecal neutral fat
   
2. 
   Elevated levels in the feces of the muscle fibers*
   
3. 
   Increased lipase content in the feces
   
4. 
   The remains of undigested food in the stool
   
5. 
   Aholichny cal
   

2072. Lientereya is

1. 
   Elevated levels of fecal neutral fat
   
2. 
   Elevated levels of muscle fibers
   
3. 
   Elevated levels of fecal elastase
   
4. 
   Elevated levels of fecal chymotrypsin
   
5. 
   The remains of undigested food in the stool*
   

2073.Development of flatulence in chronic pancreatitis is caused by

1. 
   Diarrhea
   
2. 
   Repeated vomiting
   
3. 
   Hyperglycemia
   
4. 
   To compression of the duodenum edematous head of the pancreas
   
5. 
   Enzymatic pancreatic insufficiency*
   

2074."Pancreatogenic" gastroduodenal ulcers and erosion are the result of

1. 
   Reduction in insulin secretion
   
2. 
   Decrease glucagon secretion
   
3. 
   Reduction in the level of bicarbonate secretion of pancreas*
   
4. 
   Reducing the level of lipase in the pancreatic secretion
   
5. 
   Decreased secretion of amylase secretion of the pancreas
   

2075.Clinical and laboratory evidence of exocrine pancreatic insufficiency in chronic pancreatitis is considered

1. 
   Pain in the epigastric region
   
2. 
   Kreatoreya, steatorrhea, weight loss*
   
3. 
   Hypoglycemia
   
4. 
   Jaundice
   
5. 
   An increase in liver
   

2076.Clinical and laboratory evidence of endocrine pancreatic insufficiency in chronic pancreatitis is considered

1. 
   'Wraparound' pain in the upper abdomen
   
2. 
   Weight loss
   
3. 
   Unformed stool
   
4. 
   Frequent uncontrollable vomiting
   
5. 
   Hyperglycemia*
   

2077.Zone Chauffard is

1. 
   Skin hypersensitivity in the area of innervation of the left thoracic segment VIII
   
2. 
   Epigastric region
   
3. 
   Mesogastric area
   
4. 
   Pyloroduodenal area
   
5. 
   Holedohopankreatoduodenalnaya area*
   

2078.The main clinical syndrome of chronic pancreatitis is considered

1. 
   Hypertension
   
2. 
   Edematous ascitic
   
3. 
   Pain*
   
4. 
   Hepatorenal
   
5. 
   Cardiac
   

2079.The most common laboratory test is the definition of the diagnosis of pancreatitis in the blood

1. 
   Lipase
   
2. 
   Amylase*
   
3. 
   Elastazy
   
4. 
   Trypsin
   
5. 
   Carboxypeptidase
   

2080.Methods of assessment of exocrine pancreatic function

1. 
   Determination of the level of Cpeptide in the blood
   
2. 
   Secretinpankreoziminovy test*
   
3. 
   Definition of radioimmunoassay of insulin in the blood
   
4. 
   Endoscopic retrograde cholangiopancreatography
   
5. 
   Angiography of the pancreas
   

2081.The main clinical and laboratory syndrome, chronic pancreatitis

1. 
   Cytolytic
   
2. 
   Inflammatory
   
3. 
   Inflammatory and destructive*
   
4. 
   Hypoxic
   
5. 
   Hepatocellular insufficiency
   

2082.The cause of pain in chronic pancreatitis is considered

1. 
   Infection of pancreatic secretion
   
2. 
   Dyskinesia pancreatic duct
   
3. 
   Accelerated evacuation of secretion of the pancreas
   
4. 
   Stretching pancreatic ducts*
   
5. 
   Reducing the concentration of bicarbonate secretion of the pancreas
   

2083.The clinical symptom of chronic pancreatitis

1. 
   Symptom St GeorgeMusso
   
2. 
   Symptom Kera
   
3. 
   Symptom Pasternatskogo
   
4. 
   Symptom GrekovOrtner
   
5. 
   Symptom MayoRobson*
   

2084.Replacement therapy in exocrine pancreatic insufficiency include the appointment of

1. 
   Insulin
   
2. 
   Octreotide
   
3. 
   Metoclopramide
   
4. 
   Enzyme inhibitors
   
5. 
   Enzymes*
   

2085.Indications for enzyme inhibitors in chronic pancreatitis are

1. 
   Progressive malnutrition
   
2. 
   Severe hyperglycemia
   
3. 
   Severe hyperenzymemia in the absence of the effect of treatment by other means*
   
4. 
   Obstruktsiya Pancreatic duct
   
5. 
   Persistent diarrhea
   

2086.Which of the following dietary measures is of particular importance in the treatment of chronic pancreatitis

1. 
   Increase The consumption of vitamins
   
2. 
   Increased consumption of protein
   
3. 
   Increasing the proportion of carbohydrates in the diet
   
4. 
   Avoiding alcohol*
   
5. 
   Increase consumption of vegetables
   

2087.Which of the following medication may improve symptoms of chronic pancreatitis

1. 
   Vasodilators
   
2. 
   Vitamins
   
3. 
   Vicarious pancreatic enzymes*
   
4. 
   Bile
   
5. 
   Propranolol
   

2088.Which of the following is the main indication for surgery in chronic pancreatitis

1. 
   Weight Loss
   
2. 
   Portal hypertension
   
3. 
   Pseudocyst
   
4. 
   Pain Syndrome*
   
5. 
   Calcinates in the pancreas
   

2089.If no vasculitis affects mainly largecaliber vessels

1. 
   Diseases Burger
   
2. 
   nodosus periartritis
   
3. 
   arteritis Takoyasu*
   
4. 
   hemorragic vasculitis
   
5. 
   gepatitis
   

2090. What vasculitis occur mainly in the elderly

1. 
   gemorragichesky vasculitis
   
2. 
   temporal arteritis*
   
3. 
   Diseases Takayasu
   
4. 
   nodus arteritis
   
5. 
   Diseases Kawasaki
   

2091. Note the most common manifestation of periarteritis nodosa except

1. 
   fever
   
2. 
   General joint pain
   
3. 
   Discover HBs Ag in the serum
   
4. 
   The plural Mononeuritis
   
5. 
   high titers of rheumatoid factor*
   

2092. The most characteristic pathologic feature of polyarteritis nodosa is

1. 
   arterii mixed type caliber
   
2. 
   arterii muscular type of small and mediumsized
   
3. 
   arterii muscular caliber
   
4. 
   artery mixed type of medium caliber*
   
5. 
   arterii and veins
   

2093. Who often suffers nodular periarthritis

1. 
   older men*
   
2. 
   molodye girl
   
3. 
   were male middle age
   
4. 
   pozhilye women
   
5. 
   molodye men
   

2094. The most common clinical sign is a periarteritis nodosa

1. 
   porazheniya joints
   
2. 
   kidney damage*
   
3. 
   porazheniya nervous system
   
4. 
   Lesionsheart
   
5. 
   Lesionsskin
   

2095. For the kidney damage if not typical nodular periarteritis

1. 
   gematuriya
   
2. 
   infarkt kidney
   
3. 
   glomerulonefrit
   
4. 
   polycystic*
   
5. 
   razryvy artery aneurysm
   

2096. The most characteristic feature is a periarteritis nodosa

1. 
   Hypergammaglobulinemia*
   
2. 
   leykotsitoz
   
3. 
   giperfermentemiya
   
4. 
   Increase the CEC
   
5. 
   poycilocitosis
   

2097. Flag of the disease, in which an observer can livedo reticularis

1. 
   System Lupus erythematosus
   
2. 
   Antifosfolipidny Syndrome
   
3. 
   Tromboticheskaya Thrombocytopenic purpura
   
4. 
   Ateroskleroticheskoe Vascular lesions
   
5. 
   All listed above*
   

2098.Mark diseases in which livedo reticularis among the diagnostic criteria

1. 
   System Lupus erythematosus
   
2. 
   Obliteriruyuschy Thromboangigitis
   
3. 
   Periarteritis nodosa*
   
4. 
   System Scleroderma
   
5. 
   Antifosfolipidny Syndrome
   

2099. If any of these rheumatic diseases have the highest incidence of lymphoproliferative tumors

1. 
   System Lupus erythematosus
   
2. 
   Rheumatoid arthritis
   
3. 
   Shegren's syndrome*
   
4. 
   System Scleroderma
   
5. 
   Polimiozit
   

2100. What factors determine the increase in the incidence of malignant tumors in rheumatic diseases

1. 
   Duration Treatment of corticosteroids
   
2. 
   Duration Treatment with cytostatics
   
3. 
   genetic Predisposition
   
4. 
   General Etiological factors
   
5. 
   All listed above*
   

2101. When Shegren's syndrome often reveal

1. 
   Eozinofiliyu
   
2. 
   Anemia*
   
3. 
   Leykotsitoz
   
4. 
   Limfotsitoz
   
5. 
   Trombotsitoz
   

2102. For Shegren's disease is characterized by

1. 
   Arthritis
   
2. 
   High Titers of rheumatoid factor in the serum
   
3. 
   Leykopeniya
   
4. 
   Kseroftalmiya And dry mouth
   
5. 
   All the above mentioned*
   

2103. For Shegren's syndrome is characterized by

1. 
   Mainly Joint damage with the rapid development of deformations
   
2. 
   Defeat the whole epithelial tissue*
   
3. 
   gout
   
4. 
   Muscular Contracture
   
5. 
   All Listed above
   

2104. What vasculitis are more common in the elderly

1. 
   Visochny Arteritis
   
2. 
   Hemorrhagic vasculitis*
   
3. 
   Diseases Takayasu
   
4. 
   Uzelkovy Polyarteritis
   
5. 
   Granulematoz Wegener
   

2105. What vasculitis more common in women than in men

1. 
   Uzelkovy Nodosa
   
2. 
   Kawasaki disease*
   
3. 
   Visochny Arteritis
   
4. 
   Granulematoz Wegener
   
5. 
   All Listed above
   

2106. Mark clinical manifestations are not typical of Kawasaki disease

1. 
   Eritematoznaya Rash on the palms and soles
   
2. 
   Kozhnaya Rash on the trunk
   
3. 
   Irit*
   
4. 
   Conjunctivitis
   
5. 
   "Crimson" language
   

2107. Which diseases can develop leukocyteclastic vasculitis

1. 
   Smeshannaya Cryoglobulinemia
   
2. 
   Bacterial Endocarditis
   
3. 
   Sindrom Shegren
   
4. 
   PurpuraHenoch purpura
   
5. 
   all of the above*
   

2108. What are the implications of hemorrhagic vasculitis are not true

1. 
   Syp Upper extremities is not typical
   
2. 
   More Than in the abdomen are classic manifestation of the disease
   
3. 
   Jade is usually manifested hematuria
   
4. 
   Development Nephritis is associated with deposition of IgAcontaining immune complexes
   
5. 
   High efficiency glucocorticosteroids*
   

2109. If any of these diseases can be observed syndrome of polymyalgia rheumatica

1. 
   Pirofosfatnaya Arthropathy
   
2. 
   Gigantocellular Arteritis
   
3. 
   Zlokachestvennye Neoplasms
   
4. 
   Bacterial Endocarditis
   
5. 
   All the above mentioned*
   

2110. Mark rare manifestations of polyarteritis nodosa

1. 
   Lesions Kidney in 80% of cases
   
2. 
   Education Aneurysms in the blood vessels of medium caliber
   
3. 
   Eozinofiliya
   
4. 
   Discover HbS antigen serum
   
5. 
   Reducing the concentration of complement component C3*
   

2111. Mark signs of giant cell arteritis

1. 
   Lesions Popliteal arteries
   
2. 
   Aortic aneurysm
   
3. 
   Blindness*
   
4. 
   City Loss in young adults
   
5. 
   All Listed above
   

2112. What are the conclusions about Henoch's disease are correct

1. 
   Chasche Common in children than in adults
   
2. 
   Porazhayutsya Mostly boys
   
3. 
   Is the characteristic symptom is purple
   
4. 
   Purpura development correlates with a decrease in concentration of platelets*
   
5. 
   Is the characteristic symptoms are abdominal pain, joint damage and stomach bleeding
   

2113. The combination of fever, polyneuritis, hypertension and bronchial obstruction typical for

1. 
   Periarteritis nodosa*
   
2. 
   Dermatomiozita
   
3. 
   System Lupus erythematosus
   
4. 
   Ostrogo Rheumatic fever
   
5. 
   Bacterial Endocarditis
   

2114. The cause of hypertension in nonspecific aortoarteriit often is

1. 
   Increase The formation of catecholamines
   
2. 
   The defeat of the renal arteries*
   
3. 
   Giperproduktsiya Corticosteroids
   
4. 
   Vospalitelnye Kidney disease
   
5. 
   Reninactivity does not change
   

2115. Diagnostic criterion periarteritis nodosa is

1. 
   Myocarditis
   
2. 
   Renal arterial hypertension*
   
3. 
   Bacterial Endocarditis
   
4. 
   Availability LEcells
   
5. 
   Sklerodaktiliya
   

2116. The reason for hemorrhagic vasculitis are more often

1. 
   Stafilokokk Gold
   
2. 
   βhemolytic streptococci
   
3. 
   Zelenyaschy Streptococcus
   
4. 
   Escherichia coli
   
5. 
   Pseudomonas aeruginosa*
   

2117. If no vasculitis affects mainly largecaliber vessels

1. 
   Diseases Burger
   
2. 
   Uzelkovy Nodosa
   
3. 
   Takayasu's arteritis*
   
4. 
   Gemorragichesky Vasculitis
   
5. 
   Uzelkovy Priarteriit
   

2118. Note the most common manifestation of periarteritis nodosa except

1. 
   Fever
   
2. 
   General Joint pain
   
3. 
   Discover HBsAg in serum
   
4. 
   The plural Mononeuritis
   
5. 
   High titers of rheumatoid factor*
   

2119. The most characteristic pathologic feature of polyarteritis nodosa is

1. 
   Arterii Mixed type of large caliber
   
2. 
   Arterii Muscular type of small and mediumsized
   
3. 
   Arterii Muscular caliber
   
4. 
   Arteries mixed type of medium caliber*
   
5. 
   Arterii And veins
   

2120. The most common clinical sign is a periarteritis nodosa

1. 
   Porazheniya Joints
   
2. 
   Kidney damage*
   
3. 
   Porazheniya Nervous system
   
4. 
   Lesions Heart
   
5. 
   Lesions Skin
   

2121. For the kidney damage if not typical nodular periarteritis

1. 
   Gematuriya
   
2. 
   Infarkt Kidney
   
3. 
   Glomerulonefrit
   
4. 
   Polycystic*
   
5. 
   Artery aneurysm
   

2122. The most characteristic feature is a periarteritis nodosa

1. 
   Hypergammaglobulinemia*
   
2. 
   Leykotsitoz
   
3. 
   Giperfermentemiya
   
4. 
   Increase The CEC
   
5. 
   the Presence of rheumatoid factor
   

2123.The reason for hemorrhagic vasculitis are more often

1. 
   stafilokokk gold
   
2. 
   βhemolytic streptococcus
   
3. 
   green streptococcus
   
4. 
   Escherichia coli
   
5. 
   Pseudomonas aeruginosa*
   

2124. Treatment of Dressler syndrome

1. 
   Prednisone*
   
2. 
   penicillin
   
3. 
   Klaforan
   
4. 
   aspirin
   
5. 
   pepsin
   

2125. The early complications of myocardial infarction include

1. 
   Pulmonary edema*
   
2. 
   Dressler's syndrome
   
3. 
   Chronic cardiac aneurysm
   
4. 
   Chronic heart failure
   
5. 
   anemia
   

2126. The most common complication of acute myocardial infarction is

1. 
   Break infarction
   
2. 
   Embolism brain
   
3. 
   Rhythm disorders*
   
4. 
   Dressler's syndrome
   
5. 
   Shock
   

2127. When a complication of acute myocardial infarction, complete atrioventricular block shows the introduction of

1. 
   Atropine
   
2. 
   Noradrenaline
   
3. 
   Izadrina
   
4. 
   Probe electrode in the right ventricle*
   
5. 
   All of the above
   

2128. Pulse pressure in cardiogenic shock

1. 
   Increases
   
2. 
   Decreases
   
3. 
   Decreases and then increases*
   
4. 
   Remains unchanged
   
5. 
   There is no right answer
   

2129. The greatest risk of fatal myocardial infarction associated with the development of

1. 
   Pulmonary edema
   
2. 
   Aneurysm
   
3. 
   Cardiogenic shock*
   
4. 
   Paroxysm of atrial fibrillation
   
5. 
   hepatitis
   

2130. Acute myocardial infarction on ECG is characterized by

1. 
   High T wave
   
2. 
   STsegment depression
   
3. 
   ST elevation arcuate slot
   
4. 
   Deep Q wave
   
5. 
   Atrial fibrillation
   

2131. What is the sign of the most characteristic of transmural myocardial infarction

1. 
   Pronounced Q wave
   
2. 
   Negative T waves
   
3. 
   Education tooth QS*
   
4. 
   Decrease in the amplitude of the R wave
   
5. 
   ST elevation arcuate slot
   

2132. Increased ESR myocardial infarction is usually marked

1. 
   In the early hours
   
2. 
   on the second day
   
3. 
   3-4 days
   
4. 
   The end of the first week*
   
5. 
   10-12 days*
   

2133. At what stage of myocardial infarction is characterized by the formation of pathological tooth Q

2134. Which biochemical parameters changed in the early hours of myocardial infarction

1. 
   ALT AST*
   
2. 
   Thymol turbidity test
   
3. 
   Decrease in fibrinogen
   
4. 
   Increase of CPK, LDH
   
5. 
   eritrocitosis
   

2135. The early complications of myocardial infarction do not apply

1. 
   pulmonary edema
   
2. 
   Cardiogenic shock
   
3. 
   Cardiac tamponade
   
4. 
   Dressler's syndrome*
   
5. 
   pericarditis
   

2136. One choice for the treatment of ventricular tachycardia in the acute phase of myocardial infarction are

1. 
   Cordarone
   
2. 
   Procainamide
   
3. 
   Betablockers
   
4. 
   Lidocaine*
   
5. 
   Verapamil
   

2137. The main cause of death in patients with myocardial infarction is

1. 
   ventricular asystole
   
2. 
   Ventricular fibrillation*
   
3. 
   Ventricular bigimenia
   
4. 
   II degree atrioventricular block
   
5. 
   Sinoauricular block II degree
   

2138. The most common cause of acute pulmonary heart is

1. 
   Pneumonia
   
2. 
   Asthmatic condition
   
3. 
   Pulmonary embolism*
   
4. 
   Spontaneous pneumothorax
   
5. 
   Pulmonary heart occurs at approximately the same frequency in all these states
   

2139. Clinical symptoms of cardiogenic shock

1. 
   fever, lymphadenopathy
   
2. 
   fever, cough with a "rusty" sputum
   
3. 
   sharp decline in blood pressure, rapid thread pulse*
   
4. 
   a sharp increase in blood pressure, intense pulse
   
5. 
   Hypertension
   

2140. An increase in temperature, leukocytosis, increased erythrocyte sedimentation rate are observed at

1. 
   hypertension
   
2. 
   myocardial infarction*
   
3. 
   cardiosclerosis
   
4. 
   angina
   
5. 
   hepatitis
   

2141. An aneurysm of the heart it

1. 
   left ventricular hypertrophy
   
2. 
   right ventricular hypertrophy
   
3. 
   a reduction of the left ventricle of the heart
   
4. 
   bulging portion*
   
5. 
   right atrial hypertrophy
   

2142. Pulmonary edema

1. 
   a form of acute disease
   
2. 
   coronary artery
   
3. 
   left ventricular*
   
4. 
   right heart
   
5. 
   vascular
   

2143. Increase some serum enzymes observed in the first 6-12 hours of myocardial infarction

1. 
   Creatine phosphokinase
   
2. 
   Lactate dehydrogenase*
   
3. 
   Aminotransferase
   
4. 
   alkaline phosphatase
   
5. 
   anemia
   

2144. For myocardial infarction characterized by the following echocardiographic features

1. 
   Diffuse hyperkinesis
   
2. 
   Diffuse hypokinesis
   
3. 
   Local hypokinesia*
   
4. 
   Local hyperkinesis
   
5. 
   total hyperkinesis
   

2145. What are the possible complications during thrombolytic therapy in acute myocardial infarction

1. 
   Hypotension
   
2. 
   anaphylactic shock
   
3. 
   Hemorrhagic shock
   
4. 
   Hematuria
   
5. 
   All of the above*
   

2146. What are the medicines used for the treatment of cardiogenic shock

1. 
   Mezaton
   
2. 
   Dopamine*
   
3. 
   penicillini
   
4. 
   cefozalini
   
5. 
   omezi
   

2147. What complication observed in atrial fibrillation

1. 
   Thromboembolism*
   
2. 
   Myocardial infarction
   
3. 
   hypertensive crisis
   
4. 
   Hepatitis
   
5. 
   Gout
   

2148. The most unfavorable prognostic sign in patients with acute myocardial infarction

1. 
   Atrial fibrillation
   
2. 
   Early ventricular arrythmia
   
3. 
   Group PVEs*
   
4. 
   Politop PVEs
   
5. 
   Supraventricular arrhythmias
   

2149. Which of the complications of myocardial infarction is the most common, the earliest and the most dangerous

1. 
   Cardiogenic shock
   
2. 
   Pulmonary edema
   
3. 
   Ventricular fibrillation*
   
4. 
   A heart attack
   
5. 
   Asystole
   

2150. Percentage of mass destruction attack in the development of cardiogenic shock

1. 
   More Than 20%
   
2. 
   Over 30%
   
3. 
   More than 40%*
   
4. 
   More Than 50%
   
5. 
   More Than 10%
   

2151. 6 hours after the onset of pain in acute myocardial infarction can be upgraded Level

1. 
   Creatine phosphokinase*
   
2. 
   Lactate dehydrogenase
   
3. 
   Aspartate
   
4. 
   Aldolase
   
5. 
   Αhydroxybutyrate
   

2152. Specify early complication of myocardial infarction

1. 
   cardiogenic shock*
   
2. 
   congestive heart failure
   
3. 
   Dressler's syndrome
   
4. 
   Chronic pulmonary heart
   
5. 
   hepatitis
   

2153. Select an atypical variant of myocardial infarction

1. 
   Abdominal*
   
2. 
   latent
   
3. 
   Nephrotic
   
4. 
   Mixed
   
5. 
   acute
   

2154.Bacterial endocarditis is more common

1. 
   Men*
   
2. 
   Women
   
3. 
   children
   
4. 
   Infants
   
5. 
   baby
   

2155.The etiology BE distinguished

1. 
   Gram + - Bacteria*
   
2. 
   Human immunodeficiency virus
   
3. 
   The herpes virus
   
4. 
   Campylobacter pylori
   
5. 
   pneumococcus
   

2156. For stage I BE characteristic feature

1. 
   Leukocytosis with a shift to the left*
   
2. 
   Slowing the ESR
   
3. 
   Leukopenia
   
4. 
   Lymphocytosis
   
5. 
   erithremia
   

2157.Pathogenetic second phase of infective endocarditis

1. 
   Dystrophic
   
2. 
   Hidden
   
3. 
   Immunoinflammatory*
   
4. 
   Infectious and toxic
   
5. 
   Sclerotic
   

2158.The most common during infectious endocarditis

1. 
   Acute
   
2. 
   Subacute*
   
3. 
   chronic
   
4. 
   Latent
   
5. 
   Progressive
   

2159.infective endocarditis most affected

1. 
   Tricuspid valve
   
2. 
   mitral valve
   
3. 
   semilunar valves of the pulmonary artery
   
4. 
   semilunar aortic valve*
   
5. 
   All equally
   

2160.Infective endocarditis can be caused

1. 
   Mushrooms
   
2. 
   Staphylococcus
   
3. 
   Brucella and rickettsia
   
4. 
   Viridans streptococcus
   
5. 
   All the mentioned microorganisms*
   

2161.How many pathogenic phases in the pathogenesis of infectious endocarditis

1. 
   One
   
2. 
   Two
   
3. 
   Three*
   
4. 
   four
   
5. 
   More than four
   

2162.The incidence of infective endocarditis in recent years

1. 
   Increased*
   
2. 
   Decreased
   
3. 
   Doubled
   
4. 
   Decreased twice
   
5. 
   Stagnant
   

2163.The most common causative agents of infectious endocarditis include

1. 
   Lactobacillus
   
2. 
   Pseudomonas aeruginosa
   
3. 
   Escherichia coli
   
4. 
   Saureus*
   
5. 
   Meningococcus
   

2164.The probability of infective endocarditis is greatest in patients with

1. 
   Atrial septal defect
   
2. 
   Ventricular septal defect*
   
3. 
   Mitral stenosis
   
4. 
   Mitral valve prolapse without regurgitation
   
5. 
   Hypertrophic cardiomyopathy
   

2165.When endocarditis, caused by the fungus, shows the assignment

1. 
   Ampicillin
   
2. 
   Tetracycline
   
3. 
   Amphotericin B*
   
4. 
   Kanamycin
   
5. 
   Carbenicillin
   

2166.The duration of treatment with antibiotics for infective endocarditis usually is

1. 
   2 weeks or less
   
2. 
   2-4 weeks
   
3. 
   4-6 weeks*
   
4. 
   8-10 weeks
   
5. 
   More than 10 weeks
   

2167.In subacute infective endocarditis anemia observed

1. 
   In most patients*
   
2. 
   rarely
   
3. 
   During recovery
   
4. 
   In combination with leukopenia
   
5. 
   At normal ESR
   

2168.The most common cause of infectious endocarditis is

1. 
   Gram - microflora
   
2. 
   Grampositive microorganisms*
   
3. 
   Mushrooms
   
4. 
   Viruses
   
5. 
   Chlamydia, Rickettsia
   

2169. Infective endocarditis most affected

1. 
   mitral valve
   
2. 
   Aortic valve*
   
3. 
   Tricuspid valve
   
4. 
   atrial
   
5. 
   venous
   

2170. The indications for early surgery for endocarditis are all except

1. 
   Severe heart failure
   
2. 
   Uncontrolled infection
   
3. 
   Loss of two or more valves*
   
4. 
   High risk of embolic complications
   
5. 
   High risk of anembolic complications
   

2171. The most common renal disease in infective endocarditis are

1. 
   pyelonephritis
   
2. 
   glomerulonephritis*
   
3. 
   amyloidosis
   
4. 
   hepatitis
   
5. 
   uremia
   

2172. The special forms of infective endocarditis does not include

1. 
   Infective endocarditis previously modified valve*
   
2. 
   Infective endocarditis in drug addicts
   
3. 
   Infective endocarditis in patients with implanted devices
   
4. 
   Infective endocarditis in patients on hemodialysis
   
5. 
   Infective endocarditis in the elderly
   

2173.Specify the most common cause of death in infective endocarditis

1. 
   thromboembolic complications
   
2. 
   heart rhythm disturbances
   
3. 
   heart failure*
   
4. 
   renal failure
   
5. 
   anemia
   

2174.Fever in infective endocarditis usually has the character

1. 
   Hectic*
   
2. 
   Subfebrile
   
3. 
   Intermittent
   
4. 
   Normal
   
5. 
   febrile
   

2175. Alleged nature of streptococcal infective endocarditis shows the assignment

1. 
   Penicillins or cephalosporins*
   
2. 
   Aminoglycosides
   
3. 
   Macrolides
   
4. 
   Fluoroquinolones
   
5. 
   omeprazole
   

2176. Patients with infective endocarditis and staphylococcal etiology intolerant of penicillins shows the assignment

1. 
   Vancomycin in combination with gentamicin*
   
2. 
   Ciprofloxacin
   
3. 
   Teicoplanin
   
4. 
   Fluoroquinolones
   
5. 
   calcium
   

2177.Identify morphological feature dilatation cardiomyopathy:

1. 
   the cavity of the heart greatly expanded*
   
2. 
   epicardium hypertrophied
   
3. 
   endocardium thinned
   
4. 
   in the heart of a Dutch shoe fluoroscopy
   
5. 
   the cavity of the left atrium dramatically expanded
   

2178.Identify morphological feature dilatation cardiomyopathy:

1. 
   cavities of the heart are narrowed sharply
   
2. 
   hypertrophied myocardium*
   
3. 
   epicardium thinned
   
4. 
   endocardium thinned
   
5. 
   in the heart of a Dutch shoe fluoroscopy
   

2179.Identify morphological feature dilatation cardiomyopathy:

1. 
   cavities of the heart are narrowed sharply
   
2. 
   the myocardium is not changed
   
3. 
   spherical shape of the heart during fluoroscopy*
   
4. 
   endocardium thinned
   
5. 
   in the heart of a Dutch shoe fluoroscopy
   

2180.List symptom in dilatation cardiomyopathy:

1. 
   Cardiomegaly*
   
2. 
   hypertension
   
3. 
   cardiomiodistrophy
   
4. 
   splenomegaly
   
5. 
   ascites
   

2181.List symptom in dilatation cardiomyopathy:

1. 
   circulatory failure*
   
2. 
   lack of coordination
   
3. 
   cardiomyodistrophy
   
4. 
   splenomegaly
   
5. 
   ascites
   

2182.List symptom in dilatation cardiomyopathy:

1. 
   the size of the heart is not changed
   
2. 
   violation of rhythm and conduction*
   
3. 
   cardiomiodistrophy
   
4. 
   splenomegaly
   
5. 
   ascites
   

2183.To determine the morphological sign of hypertrophic cardiomyopathy:

1. 
   asymmetrical left ventricular hypertrophy*
   
2. 
   endocardium thinned
   
3. 
   heart is shaped holand shoes under fluoroscopy
   
4. 
   mitral regurgitation
   
5. 
   Ushape of the mitral valve ECS
   

2184.To determine the morphological sign of hypertrophic cardiomyopathy:

1. 
   dilatation of the left atrium*
   
2. 
   endocardium thinned
   
3. 
   heart is shaped holand shoes under fluoroscopy
   
4. 
   mitral regurgitation
   
5. 
   Ushape of the mitral valve ECS
   

2185.List the symptoms of dilatation cardiomyopathy

1. 
   cardiomegaly, circulatory failure, arrhythmias and conduction*
   
2. 
   cardiomiodistrophy, hepatomegaly
   
3. 
   Ascites
   
4. 
   cerebrovascular accident
   
5. 
   splenomegaly
   

2186.Indicate signs of ECG dilatation cardiomyopathy:

1. 
   a decrease in the voltage of the teeth, the appearance of Q waves or QS*
   
2. 
   degenerative changes in the myocardium
   
3. 
   lifting ST interval
   
4. 
   the appearance of the tooth U
   
5. 
   high peaked T waves
   

2187.List EchoCG sign of dilatation cardiomyopathy :

1. 
   diffuse dilatation of the cavities of the heart*
   
2. 
   dilation of the left ventricle
   
3. 
   myocardial dyscinesia
   
4. 
   mitral regurgitation
   
5. 
   aortal regurgitation
   

2188.3 Names of form of flow dilatation cardiomyopathy:

1. 
   progressive, relapsing, stable*
   
2. 
   lightning, sub acute, chronic
   
3. 
   slowprogressive
   
4. 
   Unstable
   
5. 
   Chronic, sub acute, stable
   

2189.Systolic murmur in hypertrophic obstructive cardiomyopathy similar with noise generated

1. 
   with aortic stenosis*
   
2. 
   coarctasion of aorta
   
3. 
   tricuspid regurgitation
   
4. 
   ventricular septal defect
   
5. 
   open arterial (Botallov) duct
   

2190.For hypertrophic characteristic obstructive cardiomyopathy

1. 
   outflow tract contraction of the left ventricle
   
2. 
   mitral valve insufficiency
   
3. 
   left ventricular hypertrophy
   
4. 
   sudden death
   
5. 
   all of the above*
   

2191.Clinical manifestations of dilated (congestive) cardiomyopathy is

1. 
   shortness of breath
   
2. 
   heart
   
3. 
   paroxysmal tachycardia and atrial fibrillation
   
4. 
   violation of cardiac conduction
   
5. 
   all of the above*
   

2192.List form of cardiomiopaty:

1. 
   Hypertrophic*
   
2. 
   eosinophilic
   
3. 
   obstructive
   
4. 
   trombembolic
   
5. 
   cardiogenic
   

2193.What are the most important method of diagnosis of hypertrophic cardiomyopathy:

1. 
   Echocardiography*
   
2. 
   ECG
   
3. 
   Xrays
   
4. 
   auscultation
   
5. 
   PCG
   

2194.What characteristic feature has restrictive cardiomiopaty:

1. 
   an increase in the size of the left atrium*
   
2. 
   dilatation of the left ventricular cavity
   
3. 
   dilatation of the right ventricular cavity
   
4. 
   left ventricular hypertrophy
   
5. 
   narrowing of the aortic orifice
   

2195.For the treatment of arrhythmias hypertrophic cardiomyopathy shown most applications:

1. 
   quinidine
   
2. 
   novokainamid
   
3. 
   isoptin
   
4. 
   kordaron*
   
5. 
   inderal
   

2196.What are the daily doses of βblockers used for hypertrophic cardiomyopathy?

1. 
   40-80 mg
   
2. 
   120-240 mg
   
3. 
   320-480 mg*
   
4. 
   10-30 mg
   
5. 
   500-750 mg
   

2197.Unfavorable prognostic factors point to the possibility of sudden death in hypertrophic cardiomyopathy, it is:

1. 
   angina
   
2. 
   the development of heart failure
   
3. 
   a complete blockade of the left leg a bunch of Hiss
   
4. 
   ventricular arrhythmia*
   
5. 
   incomplete right bundle branch block
   

2198.Crucial in the differential diagnosis of coronary artery disease and has dilatation cardiomyopathy

1. 
   the age and sex of the patient
   
2. 
   High levels of plasma lipids
   
3. 
   Echocardiography
   
4. 
   coronary angiography*
   
5. 
   ECG
   

2199.In the treatment of hypertrophic cardiomyopathy preference from the group of calcium antagonists is given to:

1. 
   Verapamil*
   
2. 
   diltiazem
   
3. 
   nifedipine
   
4. 
   enalapril
   
5. 
   Corinfar
   

2200.the best antiarrhythmic drug that relieves and prevents the development of arrhythmias heart of hypertrophic cardiomyopathy, is:

1. 
   kordaron*
   
2. 
   diltiazem
   
3. 
   nifedipine
   
4. 
   enalapril
   
5. 
   Corinfar
   

2201.The main indication for amiodarone in hypertrophic cardiomyopathy, is:

1. 
   complete right bundle branch block
   
2. 
   incomplete blockade
   
3. 
   ventricular arrhythmias*
   
4. 
   supraventricular arrhythmias
   
5. 
   complete left bundle branch block
   

2202.In the treatment of diseases applies leucocitoferez:

1. 
   dilatation cardiomyopathy
   
2. 
   restrictive cardiomyopathy*
   
3. 
   hypertrophic cardiomyopathy
   
4. 
   hypertension
   
5. 
   angina
   

2203.Which type of cardiomyopathy is disturbed systolic myocardial function:

1. 
   in restrictive cardiomyopathy
   
2. 
   in hypertrophic cardiomyopathy
   
3. 
   in dilatation cardiomyopathy*
   
4. 
   angina pectoris
   
5. 
   supraventricular arrhythmias
   

2204.Which disease is first necessary to differentiate restrictive cardiomyopathy :

1. 
   constrictive pericarditis*
   
2. 
   Queen metal angina
   
3. 
   myocardial infarction
   
4. 
   PE
   
5. 
   myxoma
   

2205.Note drugs whose purpose is undesirable in hypertrophic cardiomyopathy:

1. 
   omeprazole
   
2. 
   cardiac glycosides*
   
3. 
   antiarrhythmics
   
4. 
   βblockers
   
5. 
   calcium antagonists
   

2206.Myocardial disease of unknown etiology is

1. 
   Infective endocarditis
   
2. 
   Rheumatic myocarditis
   
3. 
   Cardiomyopathy*
   
4. 
   Of alcoholic myocardiodystrophy
   
5. 
   IHD
   

2207. Hypertrophy interventricular septum and the left ventricle with decreasing cavity is observed for any form of cardiomyopathy

1. 
   Hypertrophic*
   
2. 
   Dilated
   
3. 
   Restrictive
   
4. 
   Cardiogenic
   
5. 
   Thromboembolic
   

2208.Systolic murmur in hypertrophic obstructive cardiomyopathy is similar to the noise generated

1. 
   For aortic stenosis*
   
2. 
   When koartatsii aorta
   
3. 
   When tricuspid regurgitation
   
4. 
   With ventricular septal defect
   
5. 
   With an open arterial (Botallova) duct
   

2209.Systolic murmur in the hypertrophic cardiomyopathy is reduced when the patient:

1. 
   Conducting sample Valsalvy
   
2. 
   Lies*
   
3. 
   Inhale amyl nitrite
   
4. 
   Gets
   
5. 
   Accepted cardiac glycosides
   

2210.Clinical manifestations of dilated (congestive) cardiomyopathy are:

1. 
   dyspnea
   
2. 
   Palpitations
   
3. 
   Paroxysmal tachycardia
   
4. 
   A violation of cardiac conduction
   
5. 
   Mark all listed*
   

2211.If you marked dilated cardiomyopathy

1. 
   Diffuse decrease in myocardial contractility*
   
2. 
   Local decrease myocardial contractility
   
3. 
   Increasing reduction of myocardial contractility
   
4. 
   Thickening of the interventricular septum
   
5. 
   All of the above
   

2212.When hypertrophic cardiomyopathy ECG is most characteristic:

1. 
   Axis deviation to the left and left ventricular hypertrophy*
   
2. 
   Axis deviation to the right and left ventricular hypertrophy
   
3. 
   Right ventricular hypertrophy
   
4. 
   Dome STsegment elevation
   
5. 
   P «Pulmonale»
   

2213.To EhoKG changes in hypertrophic cardiomyopathy include:

1. 
   Hypertrophy of the interventricular septum*
   
2. 
   Insufficiency of the aortic valve
   
3. 
   Ventricular septal defect
   
4. 
   Dilatation of the ventricles
   
5. 
   Atrial Hypertrophy
   

2214.The patient with dilated cardiomyopathy the most reliable method for diagnosis is:

1. 
   ECG
   
2. 
   Echocardiography*
   
3. 
   Veloergometry
   
4. 
   Myocardial scintigraphy
   
5. 
   Coronary angiography
   

2215.Specific cardiomyopathy include:

1. 
   Thyrotoxic cardiomyopathy*
   
2. 
   Arrhythmogenic right ventricular cardiomyopathy
   
3. 
   Idiopathic dilated cardiomyopathy
   
4. 
   Idiopathic hypertrophic cardiomyopathy
   
5. 
   Restrictive cardiomyopathy
   

2216.To restrictive cardiomyopathy include:

1. 
   Myxoma
   
2. 
   Endomyocardial fibrosis*
   
3. 
   Hemochromatosis Heart
   
4. 
   Alcoholic cardiomyopathy
   
5. 
   Primary amyloidosis of the heart
   

2217.When idiopathic dilated cardiomyopathy does not appear

1. 
   Hypertension*
   
2. 
   Violation of rhythm and conduction of the heart
   
3. 
   Symptoms of right heart failure
   
4. 
   Symptoms of left ventricular failure
   
5. 
   A tendency to thromboembolic complications
   

2218.For the idiopathic dilated cardiomyopathy auscultation most characteristic:

1. 
   Systolic murmur at the apex*
   
2. 
   Clapping tone I on the top
   
3. 
   Diastolic murmur at the apex
   
4. 
   Systolic murmur at the aorta
   
5. 
   Accent II tone of the aorta
   

2219.The radical treatment of dilated cardiomyopathy:

1. 
   Mitral valve replacement
   
2. 
   Prosthetic tricuspid valve
   
3. 
   Heart transplantation*
   
4. 
   The use of diuretics
   
5. 
   ACE inhibitors
   

2220.For the idiopathic dilated cardiomyopathy auscultation is characterized by:

1. 
   The rhythm of "gallop"*
   
2. 
   Clapping tone I on the top
   
3. 
   Diastolic murmur at the apex
   
4. 
   Systolic murmur at the aorta
   
5. 
   Accent II tone of the aorta
   

2221.Specify the most characteristic clinical sign of idiopathic dilated cardiomyopathy:

1. 
   Shortness of breath, attacks of cardiac asthma*
   
2. 
   Arthritis, arthralgia
   
3. 
   Fever
   
4. 
   Splenomegaly
   
5. 
   Cough with phlegm muco purulent
   

2222. Highlight auscultatory sign of the most characteristic of dilated cardiomyopathy:

1. 
   Rhythm "canter"*
   
2. 
   The rhythm of "quail"
   
3. 
   Diastolic murmur
   
4. 
   Noise Flint
   
5. 
   Accent II tone of the aorta
   

2223.The leading cause of sudden death in patients with hypertrophic cardiomyopathy is:

1. 
   pulmonary edema
   
2. 
   Cardiac arrhythmias*
   
3. 
   Myocardial infarction
   
4. 
   Acute ischemic stroke
   
5. 
   Congestive heart failure
   

2224. The most common cause of heart failure

1. 
   AН
   
2. 
   Amyloidosis heart
   
3. 
   CHD*
   
4. 
   Myocarditis
   
5. 
   Atrial fibrillation
   

2225. In the pathogenesis of chronic heart failure leading role played by the violation

1. 
   Chronotropic function of the heart
   
2. 
   Dromotropic heart function
   
3. 
   Inotropic function of the heart
   
4. 
   Bathmotropic heart function
   
5. 
   Infringement of automaticity of the heart*
   

2226. Neurohormonal activation in CHF appears

1. 
   Activation SAS and RAAS*
   
2. 
   Increased levels of cortisol
   
3. 
   Activation of the RAAS
   
4. 
   Hyperthyroidism
   
5. 
   Activation of CAC
   

2227.The main pathogenetic factors of edema formation in CHF are

1. 
   Increase the delay Na and fluid*
   
2. 
   An increase in central venous pressure
   
3. 
   The deterioration of the drainage function of the lymphatic system
   
4. 
   The reduction of plasma albumin and reducing the colloidosmotic pressure
   
5. 
   Increase transcapillary pressure gradient
   

2228. The classic triad of symptoms of CHF are

1. 
   Chest pain with deep breathing, coughing, and dyspnoea
   
2. 
   A feeling of heaviness in the chest, shortness of breath and palpitations
   
3. 
   Shortness of breath, weakness and swelling of the feet*
   
4. 
   Hepatomegaly, ascites and portal hypertension
   
5. 
   Attacks of dyspnea at night, coughing, and palpitations
   

2229. Signs of heart failure stage I

1. 
   Hidden circulatory insufficiency, occurs only during exercise*
   
2. 
   At rest, there are signs of circulatory failure in a small circle
   
3. 
   At rest, there are signs of circulatory failure in a large circle
   
4. 
   Dystrophic stage with severe hemodynamic and irreversible structural changes in organs
   
5. 
   At rest, there are signs of stagnation in the small and large circulation
   

2230. Signs of heart failure stage II A

1. 
   Hidden circulatory insufficiency, occurs only during exercise
   
2. 
   At rest, there are signs of circulatory failure, the small and the large circle
   
3. 
   With a small load, there are signs of circulatory failure in a large circle
   
4. 
   B alone, there are signs of circulatory failure, or small or large circle*
   
5. 
   Dystrophic stage with severe hemodynamic and irreversible structural changes in organs
   

2231. Signs of heart failure stage II B

1. 
   Hidden circulatory insufficiency, occurs only during exercise
   
2. 
   At rest, there are signs of circulatory failure, or small or large circle
   
3. 
   With a small load, there are signs of circulatory failure in a large circle
   
4. 
   B alone, there are signs of circulatory failure, the small and the large circle*
   
5. 
   Dystrophic stage with severe hemodynamic and irreversible structural changes in organs
   

2232. Signs of heart failure stage III

1. 
   Hidden circulatory insufficiency, occurs only during exercise
   
2. 
   At rest, there are signs of circulatory failure, the small and the large circle
   
3. 
   With a small load, there are signs of circulatory failure in a large circle
   
4. 
   At rest, there are signs of circulatory failure, the small and the large circle
   
5. 
   Dystrophic stage with severe hemodynamic and irreversible structural changes in organs*
   

2233. Manifestations of CHF, corresponds to I functional class (FC)

1. 
   The appearance of fatigue, palpitations and / or shortness of breath with little exertion
   
2. 
   Edema during exercise greater than usual
   
3. 
   The appearance of fatigue, palpitations and / or shortness of breath during normal physical activity
   
4. 
   The emergence of pain in the legs with a little exertion
   
5. 
   The appearance of fatigue, palpitations and / or shortness of breath on exertion beyond what is normal for the patient*
   

2234. Manifestations of CHF, corresponding to class II

1. 
   The appearance of fatigue, palpitations and / or shortness of breath with little exertion
   
2. 
   Development of syncope during normal physical activity
   
3. 
   The appearance of fatigue, palpitations and / or shortness of breath during normal physical activity*
   
4. 
   The emergence of pain in the legs with a little exertion
   
5. 
   The appearance of fatigue, palpitations and / or shortness of breath with exertion, beyond what is normal for the patient
   

2235. Manifestations of CHF, corresponding to FC III

1. 
   The appearance of fatigue, palpitations and / or shortness of breath with little exertion*
   
2. 
   Development of syncope during normal physical activity
   
3. 
   The appearance of fatigue, palpitations and / or shortness of breath during normal physical activity
   
4. 
   The emergence of pain in the legs with a little exertion
   
5. 
   The emergence of edema with little physical effort
   

2236. Manifestations of CHF, corresponding to FC IV

1. 
   The appearance of fatigue, palpitations and / or shortness of breath with little exertion
   
2. 
   Have manifestations of CHF alone*
   
3. 
   Development of syncope with little physical effort
   
4. 
   The emergence of pain in the legs with a little exertion
   
5. 
   The emergence of edema with little physical effort
   

2237. The drugs that slow the progression of heart failure

1. 
   Diuretics and spironolactone
   
2. 
   Cardiac glycosides
   
3. 
   Antiplatelet and indirect anticoagulants
   
4. 
   Peripheral vasodilators
   
5. 
   ACEinhibitors and betta blockers*
   

2238. Indication for ACE inhibitors are

1. 
   Atrial fibrillation
   
2. 
     The presence of edema
   
3. 
   CHF any stage and etiology*
   
4. 
   Sinus tachycardia
   
5. 
   Low blood pressure
   

2239. Indication for cardiac glycosides are

1. 
   Atrial fibrillation in any CHF FC*
   
2. 
   Sinus tachycardia
   
3. 
   Low blood pressure
   
4. 
   Left ventricular diastolic dysfunction
   
5. 
   The young age of the patients
   

2240. Indications for use of diuretics are

1. 
   Atrial fibrillation at any FC CHF
   
2. 
   Left ventricular diastolic dysfunction
   
3. 
   Any stage of heart failure and the etiology
   
4. 
   CHF PA III stage in the presence of stagnation*
   
5. 
   EF <25%
   

2241. Electrophysiological treatments CHF

1. 
   Resynchronization of left and right ventricular pacemakers statement, cardioverterdefibrillator*
   
2. 
   Coronary artery bypass grafting
   
3. 
   Surgical correction of valvular
   
4. 
   Heart transplantation
   
5. 
   Ablation AV connection
   

2242. Indications for heart transplantation

1. 
   Valvular heart disease
   
2. 
   LVEF> 40%
   
3. 
   LVEF <20%*
   
4. 
   III FC
   
5. 
   Expressed edema syndrome
   

2243. The method by which is possible to determine LVEF

1. 
   ECG
   
2. 
   Echocardiography*
   
3. 
   ECG monitoring
   
4. 
   Daily monitoring of blood pressure
   
5. 
   Chest Xray
   

2244. What are the most common cause of chronic heart failure is currently

1. 
   Rheumatic heart disease
   
2. 
   CHD*
   
3. 
   Arterial gipertneziya
   
4. 
   Cardiomyopathy
   
5. 
   Myocarditis and kardiodistrofii
   

2245. If no heart disease develop heart failure is a consequence of diastolic dysfunction of the myocardium

1. 
   Myocardial infarction
   
2. 
   Hypertrophic cardiomyopathy*
   
3. 
   Dilyatatsionnayakardiomiopatiya
   
4. 
   Hypotension
   
5. 
   Anemia
   

2246. Which of the following radiological signs is the earliest sign of stagnation in heart failure

1. 
   Redistribution of blood flow in favor of the upper lobes and increase the diameter of blood vessels*
   
2. 
   Interstitial pulmonary edema to form lines Curley
   
3. 
   alveolyarny swelling in the form of blackout spreads from the roots of the lungs
   
4. 
   Often the right pleural effusion
   
5. 
   Usually the left pleural effusion
   

2247. Furosemide has the following effects

1. 
   Has venodilatiruyuschim property
   
2. 
   Increases diuresis
   
3. 
   uvelichivaet hlorurez
   
4. 
   It increases natriuresis
   
5. 
   Mark all answers are correct*
   

2248. In some cases, verapamil can be used in the treatment of heart failure

1. 
   Nonsevere heart failure, coronary artery disease type
   
2. 
   Patients with the same cardiac output and impaired diastolic function of the heart*
   
3.