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Anemia*
Thrombocytopenia, a sharp increase in gamma globulins*
Leukocytosis
Polycythemia
Thrombocytosis
2828. 3 The absolute indications for surgical treatment of infective endocarditis
Expressed heart failure*
Resistance to antibiotic therapy within 3 weeks*
Abscess heart*
Rhythm disturbance
Conduction disturbances
Myocardial infarction
2829. Name prognostically unfavorable signs of infective endocarditis
No streptococcus etiology of endocarditis*
Expressed heart failure*
Involvement of the aortic valve*
Young age
female
male
2830. The most characteristic clinical symptoms of infective endocarditis are
Fever, chills*
Increase the spleen*
Symptom Lukin – Liebman*
Increase in liver
Petechial on the skin, bleeding in the mucosa of the lower eyelids
epigastric pain
2831. List the diagnostic criteria for immunoinflammatory phase BE
Development of heart disease*
Fever, after declining corticosteroids*
Increase the spleen*
Normalization of temperature after antibiotics
Geperalbuminemiya
Hyperproteinemia
2832. 3 classic symptoms of acute ES
heart murmur*
fever*
anemia*
erythrocytosis
leukopenia
hyperproteinemia
2833.3 classic symptoms of acute IE
Noise in the heart*
Fever*
Anemia*
Erythrocytosis
Leukopenia
Giperproteinemiya
2834.List 3 forms of cardiomyopathy:
dilated*
cardiogenic
restrictive*
eosinophilic
obstructive
hypertrophic*
2835.3 Names of the forms of flow dilatation cardiomyopathy:
Progressive*
Lightning
Relapsing*
Stable*
non Stable
Episodic
2836.List 3 morphological features of hypertrophic cardiomyopathy:
hardening of the mitral valve
of vegetation on the valves of the heart
ventricular septal hypertrophy*
hypertrophy of the left ventricular wall*
decreasing the left ventricular cavity*
dilation of the left ventricular cavity
2837.What are 3 main symptoms of hypertrophic cardiomyopathy:
expansion of the apical impulse*
expansion of the boundaries of the heart to the left*
pulse deficit
systolic murmur at the apex*
the weakening of the apical impulse
expanding the boundaries of the right heart
2838.Name 3 most commonly used drug in the treatment of hypertrophic cardiomyopathy:
Verapamil*
Papaverine
Kordaron*
Eufillin
Strophanthin
Ritmodan*
2839.List three diseases combined concept restrictive cardiomyopathy:
fibrosclerosis
endomyocardial fibrosis*
restrictive pericarditis
eosinophilic myocarditis fibroplastic Leffler*
fibroelastosis*
idiopathic myocarditis Fiedler
2840.For dilatation cardiomyopathy characterized by 3:
Shortness of breath*
Swelling*
Pain in the Heart
Arrhythmias*
Hypotension
Fainting
2841. The main 3 causes of death in patients with dilated cardiomyopathy is:
Sudden death from ventricular fibrillation*
Congestive circulatory failure*
pulmonary Embolism*
Rupture of the interventricular septum
Separation of the papillary muscles
Acute levomitsetina failure
2842. 3 Specific risk factors for sudden cardiac death in hypertrophic cardiomyopathy are
cardiac history*
right ventricular hypertrophy
stable ventricular tachycardia*
dyspnea
pronounced left ventricular hypertrophy*
ventricular bradicardia
2843.Give 3 reasons of restrictive cardiomyopathy
Eosinophilic endomyocardial disease*
Sarcoidosis*
Idiopathic myocarditis Fiedler
Scleroderma*
Restrictive pericarditis
Fibrosclerosis
2844. What are the main symptoms of hypertrophic cardiomyopathy:
expansion of the apical impulse*
expanding the boundaries of the right heart
pulse deficit
expansion of the boundaries of the heart to the left*
systolic murmur at the apex*
the weakening of the apical impulse
2845.List the four main groups of drugs used in the treatment of dilatation cardiomyopathy:
B vitamins
anabolic steroids
cardiac glycosides*
potassiumsparing diuretics*
peripheral vasodilators*
calcium antagonists of verapamil series
2846.List 3 major complaints of hypertrophic cardiomyopathy:
epigastric pain
hemoptysis
shortness of breath*
precordialgia*
palpitations*
cough
2847.List the 3 pathological criterias for hypertrophic cardiomyopathy:
asymmetric hypertrophy of the interventricular septum*
increasing the thickness of the top of the left (right) ventricle*
unchanged or reduce the size of the cavity of the left (right) ventricle*
absence of specific macroscopic signs of other diseases ,
always symmetric hypertrophy of the interventricular septum
increased thickness apex of the left (right) atrial
2848.The most characteristic 3 histologic features of hypertrophic cardiomyopathy include:
chaotic arrangement of cardiomyocytes*
cardiomyocyte hypertrophy*
myocardial fibrosis*
ordinal location of cardiomyocytes
thinning cardiomyocytes
calcification infarction
2849.The main 3 echocardiographic features of hypertrophic cardiomyopathy include:
left ventricular hypertrophy*
decreasing the left ventricular cavity*
outflow tract obstruction of the left ventricle*
dilatation of the left ventricle
aortic regurgitation
reduced ejection fraction
2850. 3 Drugs used in the treatment of heart failure in patients dilatation cardiomyopathy
Dopamine*
Captopril*
Enalapril*
allapinin
kordaron
validol
2851. The use of calcium antagonists in hypertrophic cardiomyopathy contribute to (3):
the improvement of diastolic compliance of the myocardium*
have an antiplatelet effect
decrease the systolic pressure gradient
improve perfusion of the coronary arteries*
decrease the end diastolic pressure and increase diastolic volume of the left atrium
exert anti-arrhythmic effect*
2852.Diagnostics of chronic heart failure (3)
ECG*
Ultrasound of the kidneys
Holter monitoring*
Spirography
Echocardiography*
angiography
2853. For the development of heart failure due to overloading of the heart disease leads 3
Myocarditis
Myocardiodystrophy
Heart defects*
Hypertension*
Symptomatic hypertension*
Hepatitis
2854.I step on the stage of circulatory failure Strazhesko - Vasilenko is characterized by 3 symptoms
Appearance of shortness of breath, fatigue only under load*
Heart only when the load*
Exists myocardial hypertrophy*
Stagnation in the pulmonary circulation
Zastoynye phenomenon in the systemic circulation
Increase (congestive) liver
2855.Stage IIB stage for circulatory failure Strazhesko - Vasilenko is characterized by 3 symptoms
Congestion in small and large circulation*
Increase (congestive) the liver and peripheral edema*
Signs of circulatory failure are reduced in the treatment of*
The appearance of shortness of breath, fatigue only under load
serdtsebieniya only with exertion
There is myocardial hypertrophy
2856. III stage by stage circulatory failure Strazhesko - Vasilenko is characterized by 3 symptoms
Are determined by liver cirrhosis*
Denominated peripheral edema, ascites, hydrothorax, cachexia*
Symptoms are persistent in nature and difficult to treat*
The appearance of shortness of breath, fatigue only under load
Heart only when the load
There is myocardial hypertrophy
2857. 3 contraindications to quinidine
Atrioventricular block*
High blood pressure
Sinus tachycardia
Severe circulatory failure*
Liver disease*
Anemia
2858.3 diseases leading to diastolic heart failure
IHD*
Alcoholism
Hypotension
Gipertrofik kardiomiopati*
Hypertensive heart disease*
Gastritis
2859.3 basic drug treatment of chronic heart Failures
ACEinhibitors*
Diuretics*
Cardiac glycosides*
Nedigidropiridinovye Channel blockers slow canals
Nitrates
Sympathomimetic
2860.In the treatment of chronic heart failure should avoid the drug 3
NSAIDs*
Class 1 antiarrhythmic drugs*
ACE inhibitors
Tricyclic antidepressants*
Diuretics
Serdechnye Glycosides
2861.What are the causes of cardiac heart failure progression
Atrial Fibrillation*
The progression of mitral insufficiency*
Myocardial infarction*
Acceptance of cardiac glycosides
Diuretics
Smoking
2862.3 glycoside intoxication symptoms
Heart rhythm disorders*
Conduction disorders*
Visually impaired*
Oliguria
Hearing loss
Violation of speech
2863.For the development of heart failure due to overloading of the heart diseases cause 3
Myocarditis
Myocardiodystrophy
Heart defects*
Hypertension*
Symptomatic hypertension*
Alcoholism
2864. 3 basic drug treatment of chronic heart Failures
ACEinhibitors*
Diuretics*
Cardiac glycosides*
Nedigidropiridinovye Channel blockers slow kanaltseevyh
Nitrates
Sympathomimetics
2865.Stage IIB circulatory failure of Strazhesko Vasilenko is characterized by 3 symptoms
Congestion in small and large circulation*
Increase (congestive) the liver and peripheral edema*
Signs of circulatory failure are reduced in the treatment of*
The appearance of shortness of breath, fatigue only under load
Heart only when the load
There myocardial hypertrophy
2866. What are the drugs for the treatment of diastolic heart failure
Diuretics*
Blockers*
Nitrates
Sympathomimetics
Calcium antagonists*
Antianaemia drugs
2867. By late clinical signs of circulatory failure are
Fixed crackles in the low back of the lungs
Appearance of peripheral edema, ascites, anasarca
Increase the liver
Dry rales in the lower lung
Headache
Dizziness
2868. 3 drugs used for relief of pulmonary edema
Nitroglycerin*
Obzidan
Furosemide*
Eufillin
Morphine*
Omeprazole
2869.Treatment of chronic heart failure (level of evidence A)
Peripheral vasodilators
ACEinhibitors*
Antagonists angiotensin II receptor
Diuretics*
Cardioselective betablockers*
Calcium channel blockers slow
2870. 3 causes the progression of chronic heart failure
Alcohol*
Infection*
No treatment*
Compliance table diet №10
The use of vegetarian food
Smoking cessation
2871."Triple therapy" severe heart failure
Diuretics*
Betablockers
Angiotensin converting enzyme inhibitors*
Xanthines
Cardiac glycosides*
Holinomimetiki
2872.Describe the 3 signs of chronic gout:
Presence of tophi*
Hypertension*
Urolithiasis /ICD/*
Absence of tophi
Chronic pyelonephritis
Hypotension
2873.Describe 3symptoms of intermittent gout:
Bouts of arthritis by 1 to 3 times year, for up to 6 to 10 days; in remission periods the health state of the patient is satisfactory*
Intermittent hyperuricemia*
low rate of internal organs lesions*
Bouts of arthritis 1 to 2 times per year for up to 6 months
Constant hyperuricemia
Obligatory visceral lesions
2874.Name 3 causes of the joints deformation in gout:
Destruction of cartilage*
Destruction of the articular surfaces*
periarticular infiltration of urates intissue with the tophiformation*
Subluxations
Proliferative changes in periarticular tissues
Swelling of the ankle
2875.Most common complication of gout is nephropathy. It may course into the following three conditions:
Urolithiasis*
Interstitial nephritis*
Nephrosclerosis*
Acute glomerulonephritis
Chronic glomerulonephritis
Acute pyelonephritis
2876.Specify the 3 basic radiographic changes typical for the gout:
Joint space narrowing*
"Punch" sign*
Erosingarticular surfaces*
Expansion of joint space
"Niche"symptom
Syndesmophytes
2877.Describe the 3 radiographic stages of gouty arthritis:
I large cysts (tophi) in the subchondral bone, sometimes sealing of soft tissue*
II large cysts, small erosion, compaction ofperiarticular soft tissues, sometimes calcifications occur*
III large erosion, osteolysis of the pineal gland, seal with lime deposits*
III large cysts (tophi) in the subchondral bone, sometimes sealing of soft tissue
I large cysts, small erosion, compaction of periarticular soft tissues, sometimes calcifications occur
II large erosion, osteolysis of the pineal gland, seal with lime deposits
2878.What are the three basic diagnostic criteria for gout:
Elevated levels of uric acid in the blood*
Tophi*
Acute attacks of arthritis, occurs suddenly, with complete clinical remission within 1 to 2 weeks*
Morning stiffness for more than 30 minutes
Heberdene’snodules
Start nature of pain
2879.What are the three changes, characteristic for the analysis of synovial fluid in acute gouty arthritis:
Low viscosity*
High cytosis*
The presence of crystals of sodium urate*
High viscosity
Low cytosis
The presence of oxalates
2880.List 3 signs of the mild gout:
Frequency of attacks of 1 to 2 times a year*
No signs of destruction on chest radiography*
No kidney damage*
Frequency of attacks 3 to 4 times a year
The presence of destructive changes on the radiography
The presence of pyelonephritis
2881.List 3 character of the average severity of gout:
Frequency of attacks 3 to 5 times a year*
Moderate articular destruction on chest radiography*
The presence of nephropathy*
Frequency of attacks 3 to 4 times a year
Involvement of not more than one joint
Pyelonephritis
2882.List 3 characteristic signs of severe gout:
Frequency of attacks of 5 or more per year*
Severe bone destruction on chest radiograph*
Severe nephropathy*
Frequency of attacks up to 4times a year
The absence of signs of destruction on chest radiograph
Glomerulonephritis
2883.What are the 3 changes in laboratory parameters characteristic of an acute attack of the gout:
Leukocytosis*
Increase in Creactive protein*
Increased ESR*
Leukopenia
Anemia
Thrombocytopenia
2884.What 3 factors are important in the development of gout?
Heredity*
Diet*
Smoking
Age*
None of the above
Mind
2885.What 3 factors are important pathogenesis of gout?
Violation of purine metabolism*
Anemiya
The excess uric acid synthesis*
Lack of urates excretion by the kidneys*
Liver damage
None of the above
2886.Mark the foods containing the highest amounts of uric acid and purine
bean*
potatoes
sprats*
sardines*
milk and milk products
carrot
pumpkin
2887.Gout is a disease associated with metabolic disorders of purine and is characterized by the following 3features:
The increase of serum uric acid*
Uratedeposits in the joints, kidneys, and blood vesselstissues*
Acute arthritis*
Increase in the content of uric acid in urine
Increase of serum urea
Redness of the skin
2889.What are the three primary pathogenic forms of gout:
Metabolic (increase of uric acid synthesis in the body)*
Kidney (impaired excretion of uric acid in the body)*
Mixed*
Metabolic (increased excretion of uric acid in the kidneys)
Kidney (increase urate deposition in the parenchyma)
Hepatic (liver delayed release of urate)
2890.Mark 3 diseases , which must be differentiated with gout:
Reactive arthritis*
Rheumatoid arthritis*
Osteoarthritis with synovitis*
Rheumatic fever
Systemic lupus erythematosus
Systemic scleroderma
2891.Name 3 "malicious sisters", i.e. the metabolic blood disorders, which are most often associated with gout:
Hyperuricemia*
Hypercholesterolemia*
Hyperglycemia*
Uremia
Hyperenzymemia
Proteinuria
2892.What are the three reasons that can provoke gouty arthritis:
Consumption of foods rich in purines (meat, beans)*
Alcohol*
Nervous and physical stress*
Use of cultured milk foods
Consumption of food rich in purines (rice, cheese, onions)
Intake of allopurinol
2893.Describe the 3 signs of articular syndrome in gout:
Acute onset, the duration of an attack from few hours to 10 days or more*
Acute pain, rapid development of crimson, then cyanotic soft tissue tumors, often in the 1st metatarsophalangeal joint of the foot*
Increase in body temperature up to 38-40C*
Gradual onset, lasting from few weeks to few months
Increased pain in the daytime
Acute pain with bleeding sores on his feet
2894.What are the three diseases that most often cause secondary gout:
"Blue" congenital heart disease*
Obese*
Diabetes*
Chronic hepatic insufficiency
Chronic hepatitis
Diabetes insipidus
2895.Specify the most frequent localization of tophi:
Ears*
Elbow*
Fingers (around joints)*
Palpebra
Achilles tendon
Spine
2896.What diseases are often associated with gout:
CHD*
Obesity*
Hypertension*
Peptic ulcer
Chronic hepatitis
Chronic pancreatitis
2897.Uricosuricremedies include:
Anturane*
Benzbromarone (dezurik)*
Sulfinpirasone*
Milurite
Allopurinol
Furosemide
2898.What foods contain the greatest amount of uric acid and purines:
Legumes*
Beef, pork, veal*
Sprats, sardines*
Chicken eggs
Potatoes
Milk and milk products
2899.What are 3 symptoms characteristic of acute gouty arthritis?
Affects single joint*
Inflammation of the joints begins suddenly*
Body temperature rises to 39-40°C*
Increased pain in the daytime
Acute pain with bleeding sores on his feet
All of the above is untrue
2900.Tophi are the deposition of urates under the skin to form dense, quite clearly demarcated and rising arthritic knots above the surface of the skin. What is the most characteristic3 features of the tophi:
Developsin 6 years (rarely over 2 to 3 years) after the first attack*
Localized in the ears, elbow, knee, feet and hands*
Content of tophi white curdled mass (urate crystals)*
Develop in the first few months of illness
Painful to the touch
Frequently infected
2901.What are the 3 reasons that can provoke gouty arthritis:
Nervous and physical stress*
Cold, hypothermia*
Exacerbation of underlying chronic diseases*
Use of cultured milk foods
Consumption of food rich in purines (rice, cheese, onions)
Administration of allopurinol
2902. Group of seronegative spondiloarthrites includes
ankylosing spondylitis (Bechterew's disease)*
Psoriatic arthritis*
pyrophosphate arthropathia
reactive arthritis*
rheumatoid arthritis
osteoarthritis
2903.Ankylosing spondylitis (Bechterew's disease) is characterized by
night pain in the lumbar region*
double sided sacroileitis*
antigen HLA B27*
nodules of Heberden
nodules of Bouchard
chorea
2904.Characteristic radiographic signs of psoriatic arthritis
narrowing of joint spaces*
regional erosion of the articular surfaces*
osteolysis*
osteophytes
symptom of "punch"
usuras
2905.Clinical signs of reactive arthritis
asymmetric arthritis of the lower limbs*
eye involvement*
urethritis or diarrhea*
symmetric arthritis of hands
rheumatoid nodules
heart lesion
2906.Select the 3 directions of cure for first degree the activity of ankylosing spondylitis
antibiotics
prednisolone
plasmapheresis
nonsteroid antiinflammatory drugs*
exercise therapy*
massage*
2907.List the 3 diseases that cause arrhythmias
ischemic heart disease*
myocarditis*
myocardiodystrophy*
chronic gastritis
chronic bronchitis
arterial hypotension
2908.List 3 signs of sinus tachycardia
the increase in heart rate over 90 per minute*
increased I tone at the apex*
RR interval shortening of on the ECG*
increased heart rate over 80 per minute
weakening of the I tone at the apex
shortening of PQ interval
2909.List 3 ECG signs of sinus bradycardia
extension of PQ interval*
decrease heart rate up to 4050 per minute*
increase in the RR interval*
shortening of PQ interval
decrease heart rate to 60 per minute
shortening of RR interval
2910.Define 3 ECG signs of ventricular extrasystole
the emergence of extraordinary QRS complex*
absence of Pwave on the ECG before the ventricular extrasystole*
deformed and belled QRS complex*
ST segment and T discordant to the main wave of QRS complex
emergence of extraordinary PQRST complex
presence of rwave on the ECG before the ventricular extrasystole
2911.Name the 3 types of extrasystoles, lifethreatening
Early*
Paired*
Polytopic*
not frequent
not complete
cardiac
2912. List 3 types of extrasystoles (localization)
Atrial*
Atrioventricular*
Ventricular*
liver function
intestinal
cardiac
2913.Name 3 diseases, most often causing paroxysmal tachycardia
myocardial infarction*
myocarditis*
thyrotoxicosis*
chronic gastritis
chronic colitis
acute bronchitis
2914.List 3 main forms of atrial fibrillation
tachysystolic (90 in 1 minute)*
normosystolic (HR = 6080 in 1 minute)*
bradysystolic (heart rate less than 60 in 1 minute)*
supraventricular
sinoauricular
ventricular
2915. What are 3 signs of atrial fibrillation
the absence of P wave*
the existence of chaotic f waves (often in leads V1V2)*
irregular ventricular complexes – varied RR intervals*
negative P wave
presence of P wave after ventricular complex
regularity of ventricular complexes – RR intervals are equal
2916.List 3 ECG characteristic AV blockade of II degree (Mobitz I)
gradual extension of PQ interval*
periodic shedding of QRS*
HR less than 60 in 1 minute*
equally elongated PQ interval
drop of two consecutive QRST complexes
increased heart rate
2917.Name 3 ECG signs of AV blockade of II degree (Mobitz II)
interval PQ stable, normal or extended*
periodic shedding of QRS*
expansion and deformation of QRS*
gradual extension of PQ interval
drop of 2 QRS complexes in a row
rapid heart rate
2918.List 3 ECG signs of AV blockade of II degree (Mobitz III)
expressed bradycardia*
drop every second of the QRS complex or more than two in a row*
QRS complexes when distal blockade can be expanded and twisted, tachycardia*
intermittent loss of QRST complex
negative P wave
QRS complex is unchanged
2919.List 3 diseases that cause arrhythmias
Ischemic Heart Disease*
Cardiomyopathy*
neurocirculatory dystonia*
chronic gastritis
chronic bronchitis
arterial hypotension
2920.Define 3 ECG signs of supraventricular extrasystole
the emergence of extraordinary P wave and QRS complex*
P wave on ECG changed in form, can be negative, twohumped, twophase, merge with T wave of previous contraction*
QRS complex is not changed*
full compensatory pause (that is equal to the sum of two normal RR intervals)
absence of P wave
deformation of the QRS complex
2921.List the 3 diseases that cause arrhythmias
heart disease*
myocarditis*
myocardiodystrophy*
chronic colitis
bronchial asthma
arterial hypotension
2922.What are the 3 main signs of paroxysmal tachycardia
Dizziness*
sudden onset of attack*
flapping I tone*
gradual commencement
weakening of I tone
diastolic murmur
2923.Name the 3 types of lifethreatening extrasystoles
Frequent*
Group*
Paired*
rare
not complete
central
2924.Name 3 ECG signs of AV blockade of I degree
extension of PQ interval more than 020 sec*
expansion of wave Р*
deformed wave Р*
extension of PQ interval more than 040 sec
negative P wave
loss of ventricular complex
2925. Name 3 diseases most often causing paroxysmal tachycardia*
Hypertension*
Thyrotoxicosis*
Neurasthenia*
chronic gastritis
chronic colitis
acute bronchitis
2926.List 3 ECG signs characteristic of WPW syndrome
shortening of the PQ interval*
Delta wave in the QRS complex*
extension and deformation of QRS*
irregularity of QRS complexes
incomplete compensatory pause
usually not belled QRS complex
2927.What refers to the ECGfeatured hypokalemia
merged T and U waves*
violation of repolarization*
offset of the ST segment lower than baseline*
usually not extended QRS complex
violation of depolarization processes
negative P wave
2928.List 3 ECG signs characteristic of the paroxysmal forms of supraventricular tachycardia
positive reflex tests*
ventricular ECG complexes are not changed*
use of finoptin is indicated*
use of lidocaine is indicated
emergence of extraordinary P wave and QRS complex
intermittent loss of QRST complex
2929."Quail" rhythm in mitral stenosis is
reinforced I tone*
reinforced II tone *
tone of opening of the mitral valve*
I tone depressed
II tone unchanged
pathologic III tone in diastole
2930. Complaints of patients with severe mitral regurgitation
shortness of breath with slight physical exertion*
attacks of night paroxysmal dyspnea*
hemoptysis*
pain in the liver
swelling of the feet
joint pain
2931. Complaints of the patients with aortic stenosis
chest pain*
loss of consciousness (syncope)*
shortness of breath*
enlargement of the liver
ascites
swelling of the feet
2932. Etiology of the acute aortic valve regurgitation
infective endocarditis*
dissecting aortic aneurysm*
injury of the heart*
atherosclerosis
NCD
liver cirrhosis
2933. Etiology of the aortic valve regurgitation
aortic aneurysm*
injury*
syphilis*
jaundice
anemia
encephalopathy
2934. Name the anatomical types of acquired heart disease
valves insufficiency*
stenosis of the atrioventricular openings or mouths of the great vessels*
сombined defect*
IVS window
ventricular septal defect
patent foramen ovale
2935.What are the causes of death in heart diseases
chronic heart failure decompensation8
thromboembolism*
paralysis of the hypertrophied heart*
encephalopathy
anemia
hypothyroidism
2936.Weakening of the 1st tone can be observed in the following heart diseases
mitral regurgitation*
tricuspid valve*
aortic stenosis*
stenosis of the atrioventricular openings
stenosis of the arteries
aortic aneurysm
2937.What sign is typical for aortic regurgitation
pulsation of the pupils*
pulsus celer et altus*
high systolic blood pressure*
decrease in pulse pressure
anemia
nausea
2938. List the main microscopic changes in the valve for acquired heart defects
valve sclerosis with its deformation
Hyalinosis*
Petrification*
vascularization valve*
trauma
rash
tumor
2939.List diseases, leading to the development of acquired heart disease
Rheumatism*
Atherosclerosis*
bacterial endocarditis*
stomach ulcer
colitis
hepatitis
2940.3 forms of stenosis of the ostium of aorta
Valvular*
Supravalvular*
Subvalvular*
membranous
pseudomembranous
aortic arch’s
2941.What are the 3 main reasons for occurrence of stenosis of the ostium of aorta
Rheumatism*
congenital malformations hearts*
atherosclerosis*
infective endocarditis
syphilis
arterial hypertension
2942.List 3 "vascular" features, characteristic for stenosis of the ostium of the aorta
pallor of skin covers*
reduced systolic AP*
small and slow pulse*
facial hyperemia
increase in pulse pressure
fast and celer heartbeat
2943.List the basic 3 methods of examination needed for establishing the diagnosis of stenosis of the ostium of the aorta
ECG*
EchoCS*
XRay examination*
scintigraphy of the heart
pneumotachometry
XRay kymography of the heart
2944.List three specific signs of AP change in aortic insufficiency
high systolic pressure*
low diastolic blood pressure*
high pulse pressure*
low systolic blood pressure
low pulse pressure
different pressure on both upper limbs
2945.Moderate hyperkaliemia in chronic renal failure can be moderated by introducing:
calcium salts*
sodium hydrogencarbonate solution*
concentrated solution of glucose with insulin*
isotonic solution
hypertonic solution
vitamins
2946.In a healthy person
Albumin does not penetrate into the primary urine
Primary urine contains about 0.1% from the amount of albumin in serum*
Approx. 84%-97% of protein is being reabsorbed from primary urine*
Daily proteinuria is 30-150 mg*
Daily proteinuria is 1.0-2.0 g
Daily proteinuria is 2.0-3.0 g
2947.Clinical manifestations of facies nefritica are:
Facial swelling*
Acrocyanosis*
Haemorrhagic rash on face
Pallor of the skin*
Swelling of the jugular veins
Mitral butterfly rash
2948.Diagnostic criteria of nephrotic syndrome are:
proteinuria more than 3.5 g/day*
hypoalbuminemia*
hypergammaglobulinemia
hypercholesterolemia*
Hyper α2-globulinemia
hypolipidemia
2949.Nephrotic syndrome is a result of
chronic glomerulonephritis*
amyloidosis*
pulmonary tuberculosis
thrombosis of the renal veins*
gastritis
hepatitis
2950.What systemic diseases are complicated by nephrotic syndrome:
systemic lupus erythematosus*
rheumatoid arthritis*
dermatomyositis
periarteritis nodosa*
scleroderma
acute rheumatic fever
2951.What is the cause of hypoproteinemia in nephrotic syndrome:
protein loss in the urine*
enhanced synthesis of βglobulin
moving proteins from plasma to the extracellular fluid*
protein loss through the oedematous intestinal mucosa*
intensified catabolised globulins.
decay of bilirubin
2952.Pathogenetic treatments of nephrotic syndrome include:
diuretics
glucocorticoids*
protein preparations
cytostatics*
anticoagulants*
betablockers
2953.Cytostatics for treatment of nephrotic syndrome when you select the correct answers:
membranous nephritis*
nodular periarteriitis*
purulent diseases
nephritis in systemic diseases*
worsening latent infection
medicinal poisoning
2954.What complications typical for nephrotic forms of chronic glomerulonephritis? Select the correct answers:
pyonephrosis
necrosis of the renal papillae
hypovolemic shock*
acute thrombosis of the renal veins*
acute heart failure*
hepatitis
2955.What are the signs of information about the functional abilities of the kidneys? Select the correct answers:
content of glucose in the urine
isostenuria*
reducing glomerular filtering*
increase in urea*
increased plasma creatinine
increase plasma enzymes
2956.Acute glomerulonephritis is characterized by the following clinical symptoms
edema*
oliguria*
bacteriuria
pyuria
hematuria*
glucosuria
2957.Urinary sediment in acute nephrotic syndrome:
hematuria*
cylindruria*
proteinuria*
leukocyturia
increasing epithelial cells count
glycosuria
2958.What diseases can cause nephrotic syndrome?
glomerulonephritis*
diabetic glomerulosclerosis*
lupus nephritis*
acute pyelonephritis
thrombosis of the renal veins
chronic pyelonephritis
2959.Nephrotic syndrome can occur in:
hypotension
systemic lupus erythematosus*
diabetic nephropathy*
hemorrhagic vasculitis by ShonleinHenoh*
hepatitis
cholecystitis
2960.Prognostically unfavorable clinical manifestations of nephropathy are:
frequent relapses of the nephrotic syndrome*
combination of nephrotic hypertensive syndromes*
noncompliance of proteinuria with hematuria and swelling
overlaying tubulointersticial lesions*
combination with hepatitis
combination with anemia
2961.Moderate hyperkaliemia in chronic renal failure can be moderated by introducing:
calcium salts*
sodium hydrogencarbonate solution*
concentrated solution of glucose with insulin*
isotonic solution
hypertonic solution
vitamins
2962.When composing diet the patient with chronic renal insufficiency one should provide:
not to limit proteins
adequate calorie intake*
exception potassiumbased products*
administration of ketosterins
not to limit salt
limiting the vitamins
2963.Most accurately reflects the degree of chronic renal failure in saving serum levels:
urea*
residual nitrogen*
creatinine
uric acid*
bilirubin
enzymes
2964.Causes of generalized edema are
heart failure
hypoproteinemia*
hypoalbuminemia*
decrease in diuresis
water and Na+ retention*
increase in enzymes
2965.Nephrotic syndrome corresponds to:
edema*
proteinuria 3g/day*
dysproteinemia*
hypolipidemia
hyperbilirubinemia
elevation of enzymes
2966.Nephrotic syndrome corresponds to:
hyperbilirubinemia
edema*
1 g proteinuria/day
dysproteinemia*
hyperlipidemia*
elevation of enzymes
2967.Blood analysis in patients with nephrotic syndrome identifies
hypoproteinemia*
hypoalbuminemia*
hyperalbuminemia
hypolipidemia
hyperlipidemia*
hypobilirubinemia
2968.Signs of nephrotic syndrome include
arterial hypertension
hypobilirubinemia
massive swelling*
hypoalbuminemia*
proteinuria > 3.5 grams per day*
proteinuria < 5.0 grams per day
2969.What kind of pneumonia are there by aetiology
Bacterial*
Viral*
Fungal*
Primary
Secondary
Lobular
2970.What are the pathological conditions precede focal pneumonia
Acute respiratory viral infection, influenza*
Acute or worsening of chronic bronchitis*
Stagnation of blood in the pulmonary circulation*
Chronic Gastritis
Neurologic Disorders
Thrombosis of Veins of the lower extremities
2971.What are the main morphological features of bronchopneumonia
Obligatory presence of inflammatory changes in the bronchial tubes (bronchioles)*
Focal character inflammation of lung tissue*
Localization Posteroventral departments*
Required Presence of inflammatory changes in the pleura (pleural effusion)
Effect Share or the entire lung
Localisation Upper anterior regions
2972.What are the main microscopic features of mycotic bronchopneumonia
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