cardiac dysfunction as the terminal event in the shock spiral because of Lactate, free radicals, and other humoral factors released by ischemic cells all act as negative inotropes and, in a decompensated patient
cardiac dysfunction as the terminal event in the shock spiral because of Lactate, free radicals, and other humoral factors released by ischemic cells all act as negative inotropes and, in a decompensated patient
A patient with cardiac disease or cardiac trauma(fixed stroke volume)…….
Shock in the elderly may therefore be rapidly progressive and may not respond predictably to fluid administration.
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Accumulation of immune complex and cellular factors in pulmonary capillaries leads to neutrophils and platelet aggregation, increased capillary permeability, destruction of lung architecture, and respiratory distress syndrome.
This is evidence that traumatic shock is more than just a hemodynamic disorder.
